Pages that link to "Q73838472"
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The following pages link to Factor XIII A subunit-deficient mice developed severe uterine bleeding events and subsequent spontaneous miscarriages (Q73838472):
Displaying 26 items.
- Catridecacog: a breakthrough in the treatment of congenital factor XIII A-subunit deficiency? (Q27026565) (← links)
- Transglutaminase regulation of cell function. (Q33707936) (← links)
- Factor XIII deficiency causes cardiac rupture, impairs wound healing, and aggravates cardiac remodeling in mice with myocardial infarction (Q33789267) (← links)
- Enhancement of thrombogenesis by plasma fibronectin cross-linked to fibrin and assembled in platelet thrombi (Q34591264) (← links)
- Genetic sequence analysis of inherited bleeding diseases (Q34663589) (← links)
- Transglutaminase 2 regulates early chondrogenesis and glycosaminoglycan synthesis. (Q35119936) (← links)
- Role of FcRgamma and factor XIIIA in coated platelet formation (Q35848141) (← links)
- Detection of thrombophilic mutations related to spontaneous abortions by a multiplex SNaPshot method (Q35888683) (← links)
- Roles of transglutaminases in cardiac and vascular diseases (Q36664166) (← links)
- New developments in the management of congenital Factor XIII deficiency (Q36906072) (← links)
- Factor XIII-A transglutaminase deficient mice show signs of metabolically healthy obesity on high fat diet (Q37349117) (← links)
- Factor Deficiencies in Pregnancy (Q37858710) (← links)
- Factor XIII: A Coagulation Factor With Multiple Plasmatic and Cellular Functions (Q37899674) (← links)
- Factor XIII is a key molecule at the intersection of coagulation and fibrinolysis as well as inflammation and infection control (Q38000164) (← links)
- Acquired FXIII inhibitors: a systematic review (Q38052068) (← links)
- Administration of factor XIII B subunit increased plasma factor XIII A subunit levels in factor XIII B subunit knock-out mice (Q39752484) (← links)
- Characterization of the transglutaminase gene family in zebrafish and in vivo analysis of transglutaminase-dependent bone mineralization (Q40685618) (← links)
- Model thrombi formed under flow reveal the role of factor XIII-mediated cross-linking in resistance to fibrinolysis (Q41904806) (← links)
- The antifibrinolytic function of factor XIII is exclusively expressed through α₂-antiplasmin cross-linking. (Q42326716) (← links)
- Transglutaminases factor XIII-A and TG2 regulate resorption, adipogenesis and plasma fibronectin homeostasis in bone and bone marrow (Q48305796) (← links)
- Fibrin-targeting immunotherapy protects against neuroinflammation and neurodegeneration (Q57464776) (← links)
- The first report of uncontrollable subchorionic and retroplacental haemorrhage inducing preterm labour in complete PAI-1 deficiency in a human (Q82591066) (← links)
- Reduced difference of α2-plasmin inhibitor levels between plasma and serum in patients with severe factor XIII deficiency, including autoimmune hemorrhaphilia due to anti-factor XIII antibodies (Q83150402) (← links)
- Report of a patient with chronic intractable autoimmune hemorrhaphilia due to anti-factor XIII/13 antibodies who died of hemorrhage after sustained clinical remission for 3 years (Q86695142) (← links)
- State of the art in factor XIII laboratory assessment (Q90823119) (← links)
- Transglutaminase 2 limits the extravasation and the resultant myocardial fibrosis associated with factor XIII-A deficiency (Q92236086) (← links)