Pages that link to "Q73552961"
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The following pages link to atm and p53 cooperate in apoptosis and suppression of tumorigenesis, but not in resistance to acute radiation toxicity (Q73552961):
Displaying 50 items.
- Chk1 suppressed cell death (Q21203552) (← links)
- Atm-dependent interactions of a mammalian Chk1 homolog with meiotic chromosomes (Q24309630) (← links)
- Functional interaction of H2AX, NBS1, and p53 in ATM-dependent DNA damage responses and tumor suppression (Q24557573) (← links)
- Functional interactions between BRCA1 and the checkpoint kinase ATR during genotoxic stress (Q24597483) (← links)
- Molecular pathology of ataxia telangiectasia (Q24672363) (← links)
- Atm heterozygous deficiency enhances development of mammary carcinomas in p53 heterozygous knockout mice (Q24796959) (← links)
- A Small Molecule Binding to the Coactivator CREB-Binding Protein Blocks Apoptosis in Cardiomyocytes (Q27667526) (← links)
- Targeted disruption of the cell-cycle checkpoint gene ATR leads to early embryonic lethality in mice (Q28143813) (← links)
- ATM: genome stability, neuronal development, and cancer cross paths (Q28201456) (← links)
- Atm and Bax cooperate in ionizing radiation-induced apoptosis in the central nervous system (Q28508555) (← links)
- Chk2 suppresses the oncogenic potential of DNA replication-associated DNA damage (Q28511458) (← links)
- No Requirement for V(D)J Recombination in p53-Deficient Thymic Lymphoma (Q28863411) (← links)
- Atm deletion with dual recombinase technology preferentially radiosensitizes tumor endothelium (Q30584877) (← links)
- A novel mouse model for ataxia-telangiectasia with a N-terminal mutation displays a behavioral defect and a low incidence of lymphoma but no increased oxidative burden (Q30806557) (← links)
- Wortmannin selectively enhances radiation-induced apoptosis in proliferative but not quiescent cells (Q31925988) (← links)
- ATR and Chk1 suppress a caspase-3-dependent apoptotic response following DNA replication stress (Q33396987) (← links)
- How does radiation kill cells? (Q33533818) (← links)
- LIN28B confers radio-resistance through the posttranscriptional control of KRAS. (Q33575786) (← links)
- Disrupting circadian homeostasis of sympathetic signaling promotes tumor development in mice (Q33601559) (← links)
- Convergent transcription through a long CAG tract destabilizes repeats and induces apoptosis (Q33637169) (← links)
- ATM in lymphoid development and tumorigenesis (Q33653657) (← links)
- Ataxia-telangiectasia, cancer and the pathobiology of the ATM gene (Q33697357) (← links)
- Atm-deficient mice exhibit increased sensitivity to dextran sulfate sodium-induced colitis characterized by elevated DNA damage and persistent immune activation (Q33699336) (← links)
- ATM: the product of the gene mutated in ataxia-telangiectasia (Q33722965) (← links)
- Breast cancer: genetic predisposition and exposure to radiation (Q33772609) (← links)
- Involvement of p53 and p21 in Cellular Defects and Tumorigenesis in Atm−/− Mice (Q33775092) (← links)
- DNA damage-induced cell cycle checkpoints and DNA strand break repair in development and tumorigenesis (Q33814324) (← links)
- Deletion of Puma protects hematopoietic stem cells and confers long-term survival in response to high-dose gamma-irradiation. (Q33839818) (← links)
- Atm is dispensable for p53 apoptosis and tumor suppression triggered by cell cycle dysfunction (Q33957801) (← links)
- Cellular senescence: mitotic clock or culture shock? (Q34019131) (← links)
- Chk2 is dispensable for p53-mediated G1 arrest but is required for a latent p53-mediated apoptotic response (Q34075834) (← links)
- DNA damage-induced apoptosis requires the DNA-dependent protein kinase, and is mediated by the latent population of p53. (Q34089341) (← links)
- Chk2 is a tumor suppressor that regulates apoptosis in both an ataxia telangiectasia mutated (ATM)-dependent and an ATM-independent manner (Q34324643) (← links)
- ATM signalling and cancer (Q34357377) (← links)
- Chemoprotection from p53-dependent apoptosis: potential clinical applications of the p53 inhibitors (Q34369172) (← links)
- ATM, a central controller of cellular responses to DNA damage. (Q34425440) (← links)
- Elevated Cu/Zn-SOD exacerbates radiation sensitivity and hematopoietic abnormalities of Atm-deficient mice (Q34584250) (← links)
- Uncoupling p53 functions in radiation-induced intestinal damage via PUMA and p21. (Q34988091) (← links)
- Identification of dAven, a Drosophila melanogaster ortholog of the cell cycle regulator Aven (Q35002331) (← links)
- Sensing of intermediates in V(D)J recombination by ATM. (Q35005088) (← links)
- The role of p53 in determining sensitivity to radiotherapy (Q35058584) (← links)
- Mouse models with modified p53 sequences to study cancer and ageing. (Q35116669) (← links)
- Critical role for Atm in suppressing V(D)J recombination-driven thymic lymphoma (Q35197081) (← links)
- Loss of transcription factor IRF-1 affects tumor susceptibility in mice carrying the Ha-ras transgene or nullizygosity for p53. (Q35197148) (← links)
- The cell cycle, chromatin and cancer: mechanism-based therapeutics come of age. (Q35208586) (← links)
- Absence of Wip1 partially rescues Atm deficiency phenotypes in mice (Q35403658) (← links)
- Tumour-suppressor function in the nervous system (Q35679775) (← links)
- Protein Kinases and Transcription Factors Activation in Response to UV-Radiation of Skin: Implications for Carcinogenesis (Q35719245) (← links)
- Loss of the ataxia-telangiectasia gene product causes oxidative damage in target organs. (Q36439520) (← links)
- Atm is a negative regulator of intestinal neoplasia (Q36494122) (← links)