Pages that link to "Q72285816"
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The following pages link to Molecular organization at the glycoprotein-complex-binding site of dystrophin. Three dystrophin-associated proteins bind directly to the carboxy-terminal portion of dystrophin (Q72285816):
Displaying 50 items.
- The interaction of dystrophin with beta-dystroglycan is regulated by tyrosine phosphorylation (Q24291543) (← links)
- Nitric oxide synthase complexed with dystrophin and absent from skeletal muscle sarcolemma in Duchenne muscular dystrophy (Q24307833) (← links)
- A novel dystrophin/utrophin-associated protein is an enzymatically inactive member of the phosphoglucomutase superfamily (Q24311341) (← links)
- Syntrophin binds to an alternatively spliced exon of dystrophin (Q24312273) (← links)
- Absence of gamma-sarcoglycan (35 DAG) in autosomal recessive muscular dystrophy linked to chromosome 13q12 (Q24313751) (← links)
- Identification and characterization of the dystrophin anchoring site on beta-dystroglycan (Q24316047) (← links)
- Genomic organization of the mouse dystrobrevin gene: comparative analysis with the dystrophin gene (Q24316279) (← links)
- The three human syntrophin genes are expressed in diverse tissues, have distinct chromosomal locations, and each bind to dystrophin and its relatives (Q24318971) (← links)
- Human adhalin is alternatively spliced and the gene is located on chromosome 17q21 (Q24324815) (← links)
- Increasing complexity of the dystrophin-associated protein complex (Q24564140) (← links)
- Dystrobrevin and dystrophin: An interaction through coiled-coil motifs (Q24648657) (← links)
- ZZ domain of dystrophin and utrophin: topology and mapping of a beta-dystroglycan interaction site (Q24672189) (← links)
- Molecular organization of sarcoglycan complex in mouse myotubes in culture (Q24685886) (← links)
- Genetic Engineering of Dystroglycan in Animal Models of Muscular Dystrophy (Q26783980) (← links)
- Galectin-1 Protein Therapy Prevents Pathology and Improves Muscle Function in the mdx Mouse Model of Duchenne Muscular Dystrophy (Q27318308) (← links)
- Cloning and expression of full length mouse utrophin: the differential association of utrophin and dystrophin with AChR clusters (Q28116310) (← links)
- Altered expression of the α7β1 integrin in human and murine muscular dystrophies (Q28258806) (← links)
- Alpha1-syntrophin has distinct binding sites for actin and calmodulin (Q28265318) (← links)
- Beta-sarcoglycan (A3b) mutations cause autosomal recessive muscular dystrophy with loss of the sarcoglycan complex (Q28290790) (← links)
- Expression of agrin, dystroglycan, and utrophin in normal renal tissue and in experimental glomerulopathies (Q28344242) (← links)
- Alternative splicing of agrin regulates its binding to heparin alpha-dystroglycan, and the cell surface (Q28574863) (← links)
- Specific disruption of a schwann cell dystrophin-related protein complex in a demyelinating neuropathy (Q28587855) (← links)
- Creatine kinase, cell membrane and Duchenne muscular dystrophy (Q33547058) (← links)
- O-mannosyl glycans in mammals (Q33783762) (← links)
- Genetic background affects properties of satellite cells and mdx phenotypes (Q33816419) (← links)
- Plasma membrane cytoskeleton of muscle: a fine structural analysis (Q33840927) (← links)
- Dystrophin and utrophin: genetic analyses of their role in skeletal muscle (Q33840935) (← links)
- Our trails and trials in the subsarcolemmal cytoskeleton network and muscular dystrophy researches in the dystrophin era (Q34567946) (← links)
- Dystrobrevin increases dystrophin's binding to the dystrophin-glycoprotein complex and provides protection during cardiac stress (Q34737660) (← links)
- Therapeutics for Duchenne muscular dystrophy: current approaches and future directions. (Q35607136) (← links)
- Dynamic (re)organization of the podocyte actin cytoskeleton in the nephrotic syndrome (Q35607185) (← links)
- Sarcoglycan complex is selectively lost in dystrophic hamster muscle (Q35796224) (← links)
- Ultrastructural localization of the C-terminus of the 43-kd dystrophin-associated glycoprotein and its relation to dystrophin in normal murine skeletal myofiber. (Q35797866) (← links)
- Mammalian alpha 1- and beta 1-syntrophin bind to the alternative splice-prone region of the dystrophin COOH terminus (Q36235194) (← links)
- Forced expression of dystrophin deletion constructs reveals structure-function correlations. (Q36237129) (← links)
- A new model for the interaction of dystrophin with F-actin (Q36237662) (← links)
- Laminin-induced clustering of dystroglycan on embryonic muscle cells: comparison with agrin-induced clustering. (Q36254531) (← links)
- Subtle neuromuscular defects in utrophin-deficient mice (Q36254585) (← links)
- alpha-Dystroglycan is a laminin receptor involved in extracellular matrix assembly on myotubes and muscle cell viability. (Q36256527) (← links)
- Animal models for muscular dystrophy show different patterns of sarcolemmal disruption. (Q36273863) (← links)
- Selective loss of sarcolemmal nitric oxide synthase in Becker muscular dystrophy (Q36367189) (← links)
- Dystrophin‐associated protein A0 is a homologue of the Torpedo 87K protein (Q36686890) (← links)
- Disruption of cortical actin in skeletal muscle demonstrates an essential role of the cytoskeleton in glucose transporter 4 translocation in insulin-sensitive tissues (Q36692823) (← links)
- Biology of the striated muscle dystrophin-glycoprotein complex (Q37085050) (← links)
- Dystroglycan receptor is involved in integrin activation in intestinal epithelia (Q37335465) (← links)
- Aquaporin expression in normal and pathological skeletal muscles: a brief review with focus on AQP4 (Q37719259) (← links)
- Exercise and muscular dystrophy: implications and analysis of effects on musculoskeletal and cardiovascular systems (Q38111711) (← links)
- Direct binding of Torpedo syntrophin to dystrophin and the 87 kDa dystrophin homologue (Q38289255) (← links)
- Utrophin lacks the rod domain actin binding activity of dystrophin (Q38317877) (← links)
- Detection of O-mannosyl glycans in rabbit skeletal muscle alpha-dystroglycan (Q38330780) (← links)