Pages that link to "Q71530371"

The following pages link to Neutral endopeptidase can hydrolyze beta-amyloid(1-40) but shows no effect on beta-amyloid precursor protein metabolism (Q71530371):

Displaying 50 items.

• beta-Amyloid degradation and Alzheimer's disease (Q21284467) (← links)
• The Alzheimer's amyloid-degrading peptidase, neprilysin: can we control it? (Q21296650) (← links)
• Insulin-degrading enzyme regulates extracellular levels of amyloid beta-protein by degradation (Q22008492) (← links)
• Catabolic attacks of membrane-bound angiotensin-converting enzyme on the N-terminal part of species-specific amyloid-beta peptides (Q24314883) (← links)
• Endothelin-converting enzymes and related metalloproteases in Alzheimer's disease (Q26862901) (← links)
• Peripheral Delivery of a CNS Targeted, Metalo-Protease Reduces Aβ Toxicity in a Mouse Model of Alzheimer's Disease (Q27437619) (← links)
• Engineering Neprilysin Activity and Specificity to Create a Novel Therapeutic for Alzheimer’s Disease (Q27684924) (← links)
• Rare Variants in MME, Encoding Metalloprotease Neprilysin, Are Linked to Late-Onset Autosomal-Dominant Axonal Polyneuropathies (Q28114961) (← links)
• Identification of the major Abeta1-42-degrading catabolic pathway in brain parenchyma: suppression leads to biochemical and pathological deposition (Q28143799) (← links)
• Human brain carboxypeptidase B, which cleaves beta-amyloid peptides in vitro, is expressed in the endoplasmic reticulum of neurons (Q30669863) (← links)
• Insulysin hydrolyzes amyloid beta peptides to products that are neither neurotoxic nor deposit on amyloid plaques. (Q30974615) (← links)
• Specific cleavage of beta-amyloid peptides by a metallopeptidase from Xenopus laevis skin secretions (Q31097285) (← links)
• Exceptional amyloid beta peptide hydrolyzing activity of nonphysiological immunoglobulin variable domain scaffolds (Q33381465) (← links)
• Long-term neprilysin gene transfer is associated with reduced levels of intracellular Abeta and behavioral improvement in APP transgenic mice (Q33384861) (← links)
• Improved learning and memory in aged mice deficient in amyloid beta-degrading neutral endopeptidase (Q33412126) (← links)
• Focusing the amyloid cascade hypothesis on N-truncated Abeta peptides as drug targets against Alzheimer's disease (Q33624303) (← links)
• Iowa variant of familial Alzheimer's disease: accumulation of posttranslationally modified AbetaD23N in parenchymal and cerebrovascular amyloid deposits (Q33745216) (← links)
• The Case for Abandoning Therapeutic Chelation of Copper Ions in Alzheimer's Disease (Q33755982) (← links)
• A neuroprotective brain-penetrating endopeptidase fusion protein ameliorates Alzheimer disease pathology and restores neurogenesis (Q33793059) (← links)
• Multifaceted Roles of Metzincins in CNS Physiology and Pathology: From Synaptic Plasticity and Cognition to Neurodegenerative Disorders (Q33854483) (← links)
• Neprilysin impedes islet amyloid formation by inhibition of fibril formation rather than peptide degradation (Q33897696) (← links)
• Amyloid-beta and Alzheimer's disease: the role of neprilysin-2 in amyloid-beta clearance (Q34039695) (← links)
• Clearing the brain's amyloid cobwebs (Q34099588) (← links)
• Active site mutations change the cleavage specificity of neprilysin (Q34182580) (← links)
• Genetic association of urokinase-type plasminogen activator gene rs2227564 site polymorphism with sporadic Alzheimer's disease in the Han Chinese population (Q34720291) (← links)
• The degradation of amyloid beta as a therapeutic strategy in Alzheimer's disease and cerebrovascular amyloidoses (Q35039021) (← links)
• Partial loss-of-function mutations in insulin-degrading enzyme that induce diabetes also impair degradation of amyloid beta-protein (Q35097358) (← links)
• Mechanisms of Abeta clearance and catabolism (Q35625628) (← links)
• Structure, evolutionary conservation, and functions of angiotensin- and endothelin-converting enzymes. (Q35909190) (← links)
• Somatostatin receptor subtype-4 agonist NNC 26-9100 decreases extracellular and intracellular Aβ₁₋₄₂ trimers (Q35923336) (← links)
• Lipid rafts and Alzheimer's disease: protein-lipid interactions and perturbation of signaling (Q36052691) (← links)
• Recombinant adeno-associated viral vectors as therapeutic agents to treat neurological disorders. (Q36368045) (← links)
• The effect of LCZ696 (sacubitril/valsartan) on amyloid-β concentrations in cerebrospinal fluid in healthy subjects (Q36806654) (← links)
• Degradation of islet amyloid polypeptide by neprilysin (Q36862586) (← links)
• Human membrane metallo-endopeptidase-like protein degrades both beta-amyloid 42 and beta-amyloid 40. (Q37003261) (← links)
• Amyloid-β 1-24 C-terminal truncated fragment promotes amyloid-β 1-42 aggregate formation in the healthy brain (Q37326344) (← links)
• Neprilysin and Aβ Clearance: Impact of the APP Intracellular Domain in NEP Regulation and Implications in Alzheimer's Disease (Q37408544) (← links)
• Sustained peripheral depletion of amyloid-β with a novel form of neprilysin does not affect central levels of amyloid-β. (Q37556990) (← links)
• Utilization of APPswe/PS1dE9 Transgenic Mice in Research of Alzheimer's Disease: Focus on Gene Therapy and Cell-Based Therapy Applications (Q37960498) (← links)
• Are amyloid‐degrading enzymes viable therapeutic targets in Alzheimer’s disease? (Q37961852) (← links)
• Proteolytic degradation of amyloid β-protein (Q38016899) (← links)
• Beta-amyloidolysis and glutathione in Alzheimer's disease (Q38104717) (← links)
• Amyloid-clearing proteins and their epigenetic regulation as a therapeutic target in Alzheimer's disease (Q38256360) (← links)
• Aggregation and catabolism of disease-associated intra-Abeta mutations: reduced proteolysis of AbetaA21G by neprilysin (Q38257065) (← links)
• The antigen-binding fragment of human gamma immunoglobulin prevents amyloid β-peptide folding into β-sheet to form oligomers. (Q38806886) (← links)
• New Insights into Epigenetic and Pharmacological Regulation of Amyloid-Degrading Enzymes (Q38834930) (← links)
• Novel APP/Aβ mutation K16N produces highly toxic heteromeric Aβ oligomers (Q39362592) (← links)
• Phosphorylation of amyloid-β peptide at serine 8 attenuates its clearance via insulin-degrading and angiotensin-converting enzymes (Q39407972) (← links)
• Self-Organizing Molecular Fingerprints: A Ligand-Based View on Drug-Like Chemical Space and Off-Target Prediction (Q39991328) (← links)
• Glutamate carboxypeptidase II is not an amyloid peptide-degrading enzyme. (Q40475257) (← links)