Pages that link to "Q71189458"
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The following pages link to Eosinophil apoptosis and the resolution of airway inflammation in asthma (Q71189458):
Displaying 50 items.
- Anti-Fas mAb-induced apoptosis and cytolysis of airway tissue eosinophils aggravates rather than resolves established inflammation (Q24814265) (← links)
- The role of airway macrophages in apoptotic cell clearance following acute and chronic lung inflammation (Q26767123) (← links)
- Apoptotic cell clearance: basic biology and therapeutic potential. (Q26825935) (← links)
- Eosinophil apoptosis and clearance in asthma (Q26852263) (← links)
- Association between genetic variation in the gene for death-associated protein-3 (DAP3) and adult asthma (Q28265130) (← links)
- Molecular Basis of Allergic Diseases (Q28272471) (← links)
- beta(2)-adrenoceptor agonists inhibit release of eosinophil-activating cytokines from human airway smooth muscle cells (Q28346487) (← links)
- Non-eosinophilic asthma: importance and possible mechanisms (Q28394141) (← links)
- Induction of eosinophil apoptosis by the cyclin-dependent kinase inhibitor AT7519 promotes the resolution of eosinophil-dominant allergic inflammation (Q28477314) (← links)
- Managing asthmatic airway inflammation: what is the role of expired nitric oxide measurement? (Q30472695) (← links)
- Pharmacological manipulation of granulocyte apoptosis: potential therapeutic targets. (Q33798112) (← links)
- Molecular pathology of allergic disease: I: lower airway disease (Q33813683) (← links)
- COPD exacerbation severity and frequency is associated with impaired macrophage efferocytosis of eosinophils (Q33970269) (← links)
- Eosinophil apoptosis--pathophysiologic and therapeutic implications. (Q34059300) (← links)
- Fas ligand expression on T cells is sufficient to prevent prolonged airway inflammation in a murine model of asthma. (Q34104832) (← links)
- Molecular mechanisms of corticosteroids in allergic diseases (Q34383896) (← links)
- Innate immunity is a key factor for the resolution of inflammation in asthma (Q34465047) (← links)
- The glucocorticoid RU24858 does not distinguish between transrepression and transactivation in primary human eosinophils. (Q35016058) (← links)
- Nitric oxide: a key regulator of myeloid inflammatory cell apoptosis (Q35116659) (← links)
- The treatment of atopic dermatitis with systemic immunosuppressive agents (Q35143066) (← links)
- Fluticasone induces T cell apoptosis in the bronchial wall of mild to moderate asthmatics (Q35536914) (← links)
- The Molecular Mechanisms of Glucocorticoids-Mediated Neutrophil Survival (Q35701310) (← links)
- Inflammatory resolution: new opportunities for drug discovery (Q35770168) (← links)
- Fas-positive T cells regulate the resolution of airway inflammation in a murine model of asthma (Q36238055) (← links)
- Lyn, Jak2, and Raf-1 kinases are critical for the antiapoptotic effect of interleukin 5, whereas only Raf-1 kinase is essential for eosinophil activation and degranulation (Q36401490) (← links)
- Flavones induce neutrophil apoptosis by down-regulation of Mcl-1 via a proteasomal-dependent pathway (Q36615199) (← links)
- Phagocytosis of apoptotic cells. (Q37099932) (← links)
- Sputum analysis in diagnosis and management of obstructive airway diseases. (Q37116800) (← links)
- Oxygen levels determine the ability of glucocorticoids to influence neutrophil survival in inflammatory environments (Q37372963) (← links)
- Human circulating eosinophils secrete macrophage migration inhibitory factor (MIF). Potential role in asthma (Q37383777) (← links)
- Phagocytic clearance of apoptotic cells: role in lung disease (Q37640810) (← links)
- Eosinophil protein in airway macrophages: a novel biomarker of eosinophilic inflammation in patients with asthma (Q37711405) (← links)
- Resolution of cell-mediated airways diseases (Q37764375) (← links)
- Asthma phenotypes: the evolution from clinical to molecular approaches (Q38007831) (← links)
- Mitochondria in the center of human eosinophil apoptosis and survival (Q38194031) (← links)
- Regulation of spontaneous eosinophil apoptosis-a neglected area of importance (Q38256352) (← links)
- Glucocorticoid regulation of human eosinophil gene expression (Q38354921) (← links)
- Mapracorat, a selective glucocorticoid receptor agonist, causes apoptosis of eosinophils infiltrating the conjunctiva in late-phase experimental ocular allergy (Q40816092) (← links)
- Immunologic Basis of Chronic Allergic Diseases: Clinical Messages from the Laboratory Bench (Q40878733) (← links)
- Clinical applications of cytokines: new directions in the therapy of atopic diseases. (Q40889783) (← links)
- Asthma: A Dynamic Disease of Inflammation and Repair (Q41562664) (← links)
- Apoptosis: clinical relevance and pharmacological manipulation (Q41618904) (← links)
- Excretory-secretory product of newly excysted metacercariae of Paragonimus westermani directly induces eosinophil apoptosis (Q41994392) (← links)
- Adam8 limits the development of allergic airway inflammation in mice. (Q42150169) (← links)
- Topical corticosteroids do not revert the activated phenotype of eosinophils in eosinophilic esophagitis but decrease surface levels of CD18 resulting in diminished adherence to ICAM-1, ICAM-2, and endothelial cells (Q42213436) (← links)
- A method to study apoptosis in eosinophils by flow cytometry (Q42489182) (← links)
- Adenovirus expressing Fas ligand gene decreases airway hyper-responsiveness and eosinophilia in a murine model of asthma. (Q45876776) (← links)
- Apoptotic eosinophils express IL-2R chains alpha and beta and co-stimulatory molecules CD28 and CD86. (Q47683264) (← links)
- Lidocaine‐derivative JMF2‐1 prevents ovalbumin‐induced airway inflammation by regulating the function and survival of T cells (Q48324151) (← links)
- Effects of glucocorticoids on apoptosis of infiltrated eosinophils and neutrophils in rats (Q50867983) (← links)