Pages that link to "Q70753477"
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The following pages link to Crossbridge scheme and the kinetic constants of elementary steps deduced from chemically skinned papillary and trabecular muscles of the ferret (Q70753477):
Displaying 50 items.
- Regulation of human heart contractility by essential myosin light chain isoforms (Q28286778) (← links)
- Effect of temperature on elementary steps of the cross-bridge cycle in rabbit soleus slow-twitch muscle fibres (Q28349425) (← links)
- Altered cross-bridge characteristics following haemodynamic overload in rabbit hearts expressing V3 myosin (Q28361710) (← links)
- Acceleration of stretch activation in murine myocardium due to phosphorylation of myosin regulatory light chain (Q28510291) (← links)
- Troponin T modulates sarcomere length-dependent recruitment of cross-bridges in cardiac muscle (Q28581503) (← links)
- Measuring single cardiac myocyte contractile force via moving a magnetic bead (Q30842720) (← links)
- Stretch increases the force by decreasing cross-bridge weakening rate in the rat cardiac trabeculae (Q30989862) (← links)
- A metabolite-sensitive, thermodynamically constrained model of cardiac cross-bridge cycling: implications for force development during ischemia (Q33598914) (← links)
- Force generation and phosphate release steps in skinned rabbit soleus slow-twitch muscle fibers (Q33907221) (← links)
- Phosphorylation-dependent power output of transgenic flies: an integrated study (Q33907968) (← links)
- Phosphate release and force generation in cardiac myocytes investigated with caged phosphate and caged calcium (Q34017764) (← links)
- Kinetic and thermodynamic studies of the cross-bridge cycle in rabbit psoas muscle fibers. (Q34018751) (← links)
- Effects of MgATP and MgADP on the cross-bridge kinetics of rabbit soleus slow-twitch muscle fibers. (Q34040572) (← links)
- The frequency response of smooth muscle stiffness during Ca2 -activated contraction (Q34170372) (← links)
- Elementary steps of the cross-bridge cycle in bovine myocardium with and without regulatory proteins (Q34177222) (← links)
- DCM-related tropomyosin mutants E40K/E54K over-inhibit the actomyosin interaction and lead to a decrease in the number of cycling cross-bridges. (Q34450885) (← links)
- The stretch-activation response may be critical to the proper functioning of the mammalian heart (Q34490405) (← links)
- Cardiac myosin binding protein C phosphorylation affects cross-bridge cycle's elementary steps in a site-specific manner. (Q34566207) (← links)
- Enhanced active cross-bridges during diastole: molecular pathogenesis of tropomyosin's HCM mutations (Q34568089) (← links)
- Regulatory Light Chain Phosphorylation and N-Terminal Extension Increase Cross-Bridge Binding and Power Output in Drosophila at In Vivo Myofilament Lattice Spacing (Q34771937) (← links)
- The role of tropomyosin isoforms and phosphorylation in force generation in thin-filament reconstituted bovine cardiac muscle fibres (Q34952053) (← links)
- Molecular determinants of force production in human skeletal muscle fibers: effects of myosin isoform expression and cross-sectional area (Q35178601) (← links)
- Structural and functional aspects of the myosin essential light chain in cardiac muscle contraction. (Q35609653) (← links)
- An exceptionally fast actomyosin reaction powers insect flight muscle (Q35768746) (← links)
- Two-state model of acto-myosin attachment-detachment predicts C-process of sinusoidal analysis (Q35878638) (← links)
- Dynamics of cross-bridge cycling, ATP hydrolysis, force generation, and deformation in cardiac muscle (Q38630982) (← links)
- Multi-scale modelling and the IUPS physiome project (Q35991517) (← links)
- Skeletal muscle ultrastructure and function in statin-tolerant individuals (Q36355588) (← links)
- Kinetics and energetics of the crossbridge cycle (Q36369909) (← links)
- Myosin cross-bridge dynamics in patients with hypertension and concentric left ventricular remodeling. (Q36421971) (← links)
- Use of thin filament reconstituted muscle fibres to probe the mechanism of force generation. (Q36565792) (← links)
- Molecular mechanisms underlying skeletal muscle weakness in human cancer: reduced myosin-actin cross-bridge formation and kinetics (Q36787452) (← links)
- Analysis of the molecular pathogenesis of cardiomyopathy-causing cTnT mutants I79N, ΔE96, and ΔK210. (Q36824519) (← links)
- Characterizations of myosin essential light chain's N-terminal truncation mutant Δ43 in transgenic mouse papillary muscles by using tension transients in response to sinusoidal length alterations. (Q36851273) (← links)
- Force transients and minimum cross-bridge models in muscular contraction (Q37143266) (← links)
- Age-related slowing of myosin actin cross-bridge kinetics is sex specific and predicts decrements in whole skeletal muscle performance in humans (Q37235093) (← links)
- Diversity and similarity of motor function and cross-bridge kinetics in papillary muscles of transgenic mice carrying myosin regulatory light chain mutations D166V and R58Q (Q37257335) (← links)
- Alternative S2 hinge regions of the myosin rod affect myofibrillar structure and myosin kinetics. (Q37265530) (← links)
- Effects of cardiac myosin isoform variation on myofilament function and crossbridge kinetics in transgenic rabbits (Q37382700) (← links)
- Adaptive control of cardiac contraction to changes in loading: from theory of sarcomere dynamics to whole-heart function (Q37870865) (← links)
- A study of tropomyosin's role in cardiac function and disease using thin-filament reconstituted myocardium (Q38108764) (← links)
- Using baculovirus/insect cell expressed recombinant actin to study the molecular pathogenesis of HCM caused by actin mutation A331P (Q38998926) (← links)
- Distribution of myosin attachment times predicted from viscoelastic mechanics of striated muscle (Q39532707) (← links)
- Nonlinear myofilament regulatory processes affect frequency-dependent muscle fiber stiffness (Q40193465) (← links)
- Elevated rates of force development and MgATP binding in F764L and S532P myosin mutations causing dilated cardiomyopathy (Q41287417) (← links)
- Mitochondrial colocalization with Ca2 release sites is crucial to cardiac metabolism (Q41853409) (← links)
- Tropomyosin period 3 is essential for enhancement of isometric tension in thin filament-reconstituted bovine myocardium (Q42926452) (← links)
- A quantitative analysis of cardiac myocyte relaxation: a simulation study. (Q43203647) (← links)
- The mechanism of the force enhancement by MgADP under simulated ischaemic conditions in rat cardiac myocytes (Q44104129) (← links)
- The effect of tropomyosin on force and elementary steps of the cross-bridge cycle in reconstituted bovine myocardium (Q44741546) (← links)