Pages that link to "Q68332076"
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The following pages link to Structure and function of the neuromuscular junction in young adultmdx mice (Q68332076):
Displaying 50 items.
- Utrophin-dystrophin-deficient mice as a model for Duchenne muscular dystrophy (Q24310537) (← links)
- Alterations of cAMP-dependent signaling in dystrophic skeletal muscle (Q27003221) (← links)
- Immunolocalisation of neuronal nitric oxide synthase at the neuromuscular junction of MDX mice: a confocal microscopy study (Q28344625) (← links)
- Acetylcholine Receptors in Innervated Muscles of DystrophicmdxMice Degrade as after Denervation (Q28591318) (← links)
- Pathological impact of SMN2 mis-splicing in adult SMA mice (Q30551534) (← links)
- Functional Neuronal Differentiation of Injury-Induced Muscle-Derived Stem Cell-Like Cells with Therapeutic Implications (Q30850601) (← links)
- The polyproline site in hinge 2 influences the functional capacity of truncated dystrophins (Q33587381) (← links)
- Characteristics of skeletal muscle in mdx mutant mice (Q33644573) (← links)
- Degeneration of neuromuscular junction in age and dystrophy (Q33658756) (← links)
- Truncated dystrophins can influence neuromuscular synapse structure. (Q33676836) (← links)
- Understanding dystrophinopathies: an inventory of the structural and functional consequences of the absence of dystrophin in muscles of the mdx mouse (Q33836366) (← links)
- Dystrophin and utrophin: genetic analyses of their role in skeletal muscle (Q33840935) (← links)
- Nerve Terminal Growth Remodels Neuromuscular Synapses in Mice following Regeneration of the Postsynaptic Muscle Fiber (Q34019649) (← links)
- The alpha-syntrophin PH and PDZ domains scaffold acetylcholine receptors, utrophin, and neuronal nitric oxide synthase at the neuromuscular junction (Q34114835) (← links)
- Safety factor at the neuromuscular junction (Q34199050) (← links)
- Granulocyte colony-stimulating factor (G-CSF) positive effects on muscle fiber degeneration and gait recovery after nerve lesion in MDX mice. (Q34300608) (← links)
- Participation of myosin Va and Pka type I in the regeneration of neuromuscular junctions (Q34345318) (← links)
- Clustering of nicotinic acetylcholine receptors: from the neuromuscular junction to interneuronal synapses (Q34560210) (← links)
- Evidence for impaired neurovascular transmission in a murine model of Duchenne muscular dystrophy (Q34753699) (← links)
- Recovery of altered neuromuscular junction morphology and muscle function in mdx mice after injury (Q34807765) (← links)
- Transgenic expression of {alpha}7{beta}1 integrin maintains muscle integrity, increases regenerative capacity, promotes hypertrophy, and reduces cardiomyopathy in dystrophic mice (Q35083511) (← links)
- Muscle-derived extracellular signal-regulated kinases 1 and 2 are required for the maintenance of adult myofibers and their neuromuscular junctions (Q35165138) (← links)
- Expression of the dystrophin isoform Dp116 preserves functional muscle mass and extends lifespan without preventing dystrophy in severely dystrophic mice (Q35565691) (← links)
- PGC-1alpha regulates the neuromuscular junction program and ameliorates Duchenne muscular dystrophy. (Q35720854) (← links)
- microRNA-206 promotes skeletal muscle regeneration and delays progression of Duchenne muscular dystrophy in mice (Q36005429) (← links)
- Pre- and postsynaptic changes in the neuromuscular junction in dystrophic mice (Q36038762) (← links)
- Subtle neuromuscular defects in utrophin-deficient mice (Q36254585) (← links)
- Postsynaptic abnormalities at the neuromuscular junctions of utrophin-deficient mice (Q36254595) (← links)
- Sarcolemmal ion channels in dystrophin-deficient skeletal muscle fibres (Q36549694) (← links)
- Neuregulin1 displayed on motor axons regulates terminal Schwann cell-mediated synapse elimination at developing neuromuscular junctions (Q36551508) (← links)
- Recovery of mouse neuromuscular junctions from single and repeated injections of botulinum neurotoxin A (Q36882760) (← links)
- Biology of the striated muscle dystrophin-glycoprotein complex (Q37085050) (← links)
- Reliability of neuromuscular transmission and how it is maintained (Q37218052) (← links)
- The value of mammalian models for duchenne muscular dystrophy in developing therapeutic strategies (Q37383091) (← links)
- Changes in aging mouse neuromuscular junctions are explained by degeneration and regeneration of muscle fiber segments at the synapse (Q39034591) (← links)
- Age-related fragmentation of the motor endplate is not associated with impaired neuromuscular transmission in the mouse diaphragm (Q40371672) (← links)
- Myofiber branching rather than myofiber hyperplasia contributes to muscle hypertrophy in mdx mice (Q40372230) (← links)
- Neuromuscular junctions are pathological but not denervated in two mouse models of spinal bulbar muscular atrophy. (Q40848344) (← links)
- Dp71 can restore the dystrophin-associated glycoprotein complex in muscle but fails to prevent dystrophy. (Q41418979) (← links)
- Innervation of dystrophic muscle after muscle stem cell therapy (Q41654030) (← links)
- Maturation and maintenance of the neuromuscular synapse: genetic evidence for roles of the dystrophin--glycoprotein complex (Q42485105) (← links)
- Muscle dystroglycan organizes the postsynapse and regulates presynaptic neurotransmitter release at the Drosophila neuromuscular junction. (Q43151473) (← links)
- Functional in situ assessment of muscle contraction in wild-type and mdx mice (Q43153801) (← links)
- Lack of desmin results in abortive muscle regeneration and modifications in synaptic structure (Q43666983) (← links)
- Axonal sprouting in mdx mice and its relevance to cell and gene mediated therapies for Duchenne muscular dystrophy (Q44443033) (← links)
- Acetylcholine receptor distribution and synapse elimination at the developing neuromuscular junction of mdx mice (Q44627078) (← links)
- The Structure of Human Neuromuscular Junctions: Some Unanswered Molecular Questions (Q45066212) (← links)
- Effects of in vivo injury on the neuromuscular junction in healthy and dystrophic muscles (Q45220017) (← links)
- Dystrophin is required for appropriate retrograde control of neurotransmitter release at the Drosophila neuromuscular junction. (Q46884602) (← links)
- Complete deletion of all alpha-dystrobrevin isoforms does not reveal new neuromuscular junction phenotype. (Q46953614) (← links)