Pages that link to "Q63384843"
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The following pages link to Chloé Journo (Q63384843):
Displaying 30 items.
- NRP/Optineurin Cooperates with TAX1BP1 to potentiate the activation of NF-kappaB by human T-lymphotropic virus type 1 tax protein (Q33483337) (← links)
- Discovery and characterization of auxiliary proteins encoded by type 3 simian T-cell lymphotropic viruses (Q34991026) (← links)
- Combination of arsenic and interferon-α inhibits expression of KSHV latent transcripts and synergistically improves survival of mice with primary effusion lymphomas (Q35046691) (← links)
- HTLV-1 and Innate Immunity (Q35261692) (← links)
- A Transgenic Drosophila melanogaster Model To Study Human T-Lymphotropic Virus Oncoprotein Tax-1-Driven Transformation In Vivo (Q35861179) (← links)
- Dendritic cell maturation, but not type I interferon exposure, restricts infection by HTLV-1, and viral transmission to T-cells. (Q36352268) (← links)
- Cell-Free versus Cell-to-Cell Infection by Human Immunodeficiency Virus Type 1 and Human T-Lymphotropic Virus Type 1: Exploring the Link among Viral Source, Viral Trafficking, and Viral Replication. (Q37182721) (← links)
- Human T-cell leukemia virus type 2 produces a spliced antisense transcript encoding a protein that lacks a classic bZIP domain but still inhibits Tax2-mediated transcription (Q37352476) (← links)
- HTLV gene regulation: because size matters, transcription is not enough (Q37470581) (← links)
- The multifaceted oncoprotein Tax: subcellular localization, posttranslational modifications, and NF-κB activation (Q37994930) (← links)
- Alpha interferon restricts human T-lymphotropic virus type 1 and 2 de novo infection through PKR activation. (Q39088537) (← links)
- ADAR1 enhances HTLV-1 and HTLV-2 replication through inhibition of PKR activity (Q41666767) (← links)
- Human T cell leukemia virus type 2 tax-mediated NF-κB activation involves a mechanism independent of Tax conjugation to ubiquitin and SUMO. (Q41881538) (← links)
- Correction: Dendritic cell maturation, but not type I interferon exposure, restricts infection by HTLV-1, and viral transmission to T-cells (Q42333111) (← links)
- A transgenic Drosophila melanogaster model to study HTLV-I oncoprotein Tax-driven leukemogenesis in vivo (Q45826746) (← links)
- SQSTM1/p62 regulates HTLV-1 tax mediated NF-κB activation (Q46507413) (← links)
- Antisense protein of HTLV-2 (APH-2) associates with PML nuclear bodies: molecular determinants and functional implications (Q46960129) (← links)
- Low levels of HTLV-2 Tax conjugation to ubiquitin and SUMO do not impede Tax-mediated activation of NF-κB. (Q47129932) (← links)
- HTLV-1 Tax targets centrosomal Cep63 protein. (Q53835105) (← links)
- How to Control HTLV-1-Associated Diseases: Preventing de Novo Cellular Infection Using Antiviral Therapy. (Q54246489) (← links)
- [The double antiviral effect of IFITM is conserved in many species of enveloped viruses]. (Q54254820) (← links)
- Stability of HTLV-2 antisense protein is controlled by PML nuclear bodies in a SUMO-dependent manner. (Q54261796) (← links)
- [Unanchored ubiquitin chains: a new viral target for NF-κB activation]. (Q54605433) (← links)
- STLV-1 co-infection is correlated with an increased SFV proviral load in the peripheral blood of SFV/STLV-1 naturally infected non-human primates (Q57061692) (← links)
- Exclusion from the Golgi and very low levels of HTLV-2 Tax ubiquitination do not prevent IKK-gamma/NEMO relocalization and NF-κB activation (Q59255414) (← links)
- Quand le cytosquelette d’actine fait le jeu des rétrovirus (Q63707044) (← links)
- The interferon stimulated gene 20 protein (ISG20) is an innate defense antiviral factor that discriminates self versus non-self translation (Q90629837) (← links)
- SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function (Q91130338) (← links)
- TRAF3 Is Required for NF-κB Pathway Activation Mediated by HTLV Tax Proteins (Q93062480) (← links)
- Nipah virus W protein harnesses nuclear 14-3-3 to inhibit NF-κB-induced proinflammatory response (Q112746785) (← links)