Pages that link to "Q60153647"

The following pages link to Increased Expression of Fibronectin Isoforms After Myocardial Infarction in Rats (Q60153647):

Displaying 28 items.

• Translating Koch's postulates to identify matrix metalloproteinase roles in postmyocardial infarction remodeling: cardiac metalloproteinase actions (CarMA) postulates (Q26865099) (← links)
• Extracellular matrix remodeling following myocardial infarction influences the therapeutic potential of mesenchymal stem cells (Q33746224) (← links)
• Cardiomyocyte contractile status is associated with differences in fibronectin and integrin interactions (Q33917141) (← links)
• Fibronectin increases the force production of mouse papillary muscles via α5β1 integrin (Q34482278) (← links)
• Systems analysis of gene ontology and biological pathways involved in post-myocardial infarction responses. (Q35671282) (← links)
• Myofibroblast and endothelial cell proliferation during murine myocardial infarct repair (Q35843221) (← links)
• Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction. (Q36319909) (← links)
• Matrix metalloproteinases: drug targets for myocardial infarction (Q37308645) (← links)
• The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction (Q37561922) (← links)
• Extracellular matrix roles during cardiac repair. (Q37776560) (← links)
• Mechanotransduction: the role of mechanical stress, myocyte shape, and cytoskeletal architecture on cardiac function (Q37866169) (← links)
• Cardiac Physiology of Aging: Extracellular Considerations. (Q38542448) (← links)
• The extracellular matrix in myocardial injury, repair, and remodeling. (Q39275058) (← links)
• Fibronectin-based isolation of valve interstitial cell subpopulations: relevance to valve disease. (Q39630057) (← links)
• Behavior of CMPCs in unidirectional constrained and stress-free 3D hydrogels. (Q40637593) (← links)
• Accumulation of fibronectin in the heart after myocardial infarction: a putative stimulator of adhesion and proliferation of adipose-derived stem cells (Q41826932) (← links)
• The EIIIA domain from astrocyte-derived fibronectin mediates proliferation of oligodendrocyte progenitor cells following CNS demyelination (Q42544806) (← links)
• Induction of Cardiac Angiogenesis Requires Killer Cell Lectin-Like Receptor 1 and α4β7 Integrin Expression by NK Cells (Q42679536) (← links)
• Matrix production and remodeling capacity of cardiomyocyte progenitor cells during in vitro differentiation (Q47357598) (← links)
• The VEGFA165b isoform is dysregulated in senescent endothelial cells and may be associated with prevalent and incident coronary heart disease (Q50058818) (← links)
• Inhibiting Fibronectin Attenuates Fibrosis and Improves Cardiac Function in a Model of Heart Failure. (Q52589298) (← links)
• Myocardial Infarction Remodeling that Progresses to Heart Failure: A Signaling Misunderstanding. (Q52619556) (← links)
• Ischemic injury in posttransplant endomyocardial biopsies: immunohistochemical study of fibronectin (Q73027869) (← links)
• Expression of Dishevelled-1 in wound healing after acute myocardial infarction: possible involvement in myofibroblast proliferation and migration (Q80345305) (← links)
• Interleukin-2 enhances angiogenesis and preserves cardiac function following myocardial infarction (Q82059367) (← links)
• The Non-Fibrillar Side of Fibrosis: Contribution of the Basement Membrane, Proteoglycans, and Glycoproteins to Myocardial Fibrosis (Q90233203) (← links)
• Hypoxia-induced H19/YB-1 cascade modulates cardiac remodeling after infarction (Q90533345) (← links)
• Computational Model Predicts Paracrine and Intracellular Drivers of Fibroblast Phenotype After Myocardial Infarction (Q90627860) (← links)