Pages that link to "Q59544755"
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The following pages link to Ubiquitinated, p62 immunopositive cerebellar cortical neuronal inclusions are evident across the spectrum of TDP-43 proteinopathies but are only rarely additionally immunopositive for phosphorylation-dependent TDP-43 (Q59544755):
Displaying 28 items.
- C9ORF72, the new gene on the block, causes C9FTD/ALS: new insights provided by neuropathology (Q24605612) (← links)
- Frontotemporal dementia with the C9ORF72 hexanucleotide repeat expansion: clinical, neuroanatomical and neuropathological features (Q24630410) (← links)
- Pathological mechanisms underlying TDP-43 driven neurodegeneration in FTLD-ALS spectrum disorders (Q26828357) (← links)
- Sumoylation of critical proteins in amyotrophic lateral sclerosis: emerging pathways of pathogenesis (Q26866679) (← links)
- What does imaging reveal about the pathology of amyotrophic lateral sclerosis? (Q27007017) (← links)
- Protein Homeostasis in Amyotrophic Lateral Sclerosis: Therapeutic Opportunities? (Q33621868) (← links)
- Structural explanation of poor prognosis of amyotrophic lateral sclerosis in the non-demented state (Q33701772) (← links)
- Oligoclonal bands in the cerebrospinal fluid of amyotrophic lateral sclerosis patients with disease-associated mutations. (Q34336785) (← links)
- Brain distribution of dipeptide repeat proteins in frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9ORF72 (Q34500097) (← links)
- Drosha inclusions are new components of dipeptide-repeat protein aggregates in FTLD-TDP and ALS C9orf72 expansion cases (Q35184485) (← links)
- The Spectrum of C9orf72-mediated Neurodegeneration and Amyotrophic Lateral Sclerosis (Q35501931) (← links)
- Regulation of Autophagy by Neuropathological Protein TDP-43 (Q35639841) (← links)
- Clinical and pathological features of familial frontotemporal dementia caused by C9ORF72 mutation on chromosome 9p (Q35776895) (← links)
- Distinct clinical and pathological characteristics of frontotemporal dementia associated with C9ORF72 mutations (Q35776901) (← links)
- Frontotemporal lobar degeneration with TDP-43 proteinopathy and chromosome 9p repeat expansion in C9ORF72: clinicopathologic correlation (Q36253321) (← links)
- Pattern of ubiquilin pathology in ALS and FTLD indicates presence of C9ORF72 hexanucleotide expansion (Q36464731) (← links)
- Dipeptide repeat proteins are present in the p62 positive inclusions in patients with frontotemporal lobar degeneration and motor neurone disease associated with expansions in C9ORF72. (Q37485777) (← links)
- Progression of tau pathology within cholinergic nucleus basalis neurons in chronic traumatic encephalopathy: A chronic effects of neurotrauma consortium study (Q37696939) (← links)
- Autophagy and Its Impact on Neurodegenerative Diseases: New Roles for TDP-43 and C9orf72. (Q39370811) (← links)
- Pathological assessments for the presence of hexanucleotide repeat expansions in C9ORF72 in Alzheimer's disease (Q41874608) (← links)
- Sense-encoded poly-GR dipeptide repeat proteins correlate to neurodegeneration and uniquely co-localize with TDP-43 in dendrites of repeat-expanded C9orf72 amyotrophic lateral sclerosis. (Q46799396) (← links)
- Motor neuron degeneration in spastic paraplegia 11 mimics amyotrophic lateral sclerosis lesions (Q49169440) (← links)
- p62/SQSTM1 differentially removes the toxic mutant androgen receptor via autophagy and inclusion formation in a spinal and bulbar muscular atrophy mouse model. (Q51025659) (← links)
- The Genetics of C9orf72 Expansions (Q51165295) (← links)
- Mechanistic View of hnRNPA2 Low-Complexity Domain Structure, Interactions, and Phase Separation Altered by Mutation and Arginine Methylation. (Q52390768) (← links)
- Mechanisms of selective autophagy and mitophagy: Implications for neurodegenerative diseases. (Q61718173) (← links)
- Molecular Mechanisms of TDP-43 Misfolding and Pathology in Amyotrophic Lateral Sclerosis (Q64263070) (← links)
- Selective neuronal degeneration in MATR3 S85C knock-in mouse model of early-stage ALS (Q100750119) (← links)