Pages that link to "Q58365541"

The following pages link to Oxyradical damage and mitochondrial enzyme activities in the mdx mouse (Q58365541):

Displaying 24 items.

• Anti-inflammatory drugs for Duchenne muscular dystrophy: focus on skeletal muscle-releasing factors (Q28069464) (← links)
• Nutraceuticals and Their Potential to Treat Duchenne Muscular Dystrophy: Separating the Credible from the Conjecture (Q28072269) (← links)
• The role of oxidative stress in skeletal muscle injury and regeneration: focus on antioxidant enzymes (Q28076877) (← links)
• Lack of dystrophin is associated with altered integration of the mitochondria and ATPases in slow-twitch muscle cells of MDX mice (Q30665842) (← links)
• Emerging drugs for Duchenne muscular dystrophy (Q36363716) (← links)
• Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2 , Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy. (Q36422465) (← links)
• Wasting mechanisms in muscular dystrophy. (Q37138490) (← links)
• Intracellular energetic units in healthy and diseased hearts (Q37276512) (← links)
• Contribution of oxidative stress to pathology in diaphragm and limb muscles with Duchenne muscular dystrophy (Q38056065) (← links)
• Dystrophin deficiency leads to disturbance of LAMP1-vesicle-associated protein secretion (Q39205845) (← links)
• Improvement of endurance of DMD animal model using natural polyphenols. (Q41091176) (← links)
• Dystrophin mutations predict cellular susceptibility to oxidative stress (Q41734187) (← links)
• Increased catalase expression improves muscle function inmdxmice (Q41845964) (← links)
• Between channels and tears: aim at ROS to save the membrane of dystrophic fibres (Q46670207) (← links)
• N-Acetylcysteine ameliorates skeletal muscle pathophysiology in mdx mice (Q46765634) (← links)
• Duchenne and Becker Muscular Dystrophies: A Review of Animal Models, Clinical End Points, and Biomarker Quantification. (Q47704855) (← links)
• Pre-clinical evaluation of N-acetylcysteine reveals side effects in the mdx mouse model of Duchenne muscular dystrophy. (Q47830220) (← links)
• Commentary: extraocular muscle sparing in muscular dystrophy: a critical evaluation of potential protective mechanisms (Q47986383) (← links)
• H(2)O(2) detection from intact mitochondria as a measure for one-electron reduction of dioxygen requires a non-invasive assay system (Q73066770) (← links)
• Oxidative stress as a potential pathogenic mechanism in an animal model of Duchenne muscular dystrophy (Q73778888) (← links)
• Muscle cells from mdx mice have an increased susceptibility to oxidative stress (Q74495850) (← links)
• Mdx myotubes have normal excitability but show reduced contraction-relaxation dynamics (Q74529467) (← links)
• Evidence of oxidative stress in mdx mouse muscle: studies of the pre-necrotic state (Q77763590) (← links)
• Comparative proteomic analyses of Duchenne muscular dystrophy and Becker muscular dystrophy muscles: changes contributing to preserve muscle function in Becker muscular dystrophy patients (Q92996733) (← links)