Pages that link to "Q57872823"
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The following pages link to Serine racemase deletion abolishes light-evoked NMDA receptor currents in retinal ganglion cells (Q57872823):
Displaying 12 items.
- Pathogenic disruption of DISC1-serine racemase binding elicits schizophrenia-like behavior via D-serine depletion (Q30447828) (← links)
- The postnatal development of D-serine in the retinas of two mouse strains, including a mutant mouse with a deficiency in D-amino acid oxidase and a serine racemase knockout mouse (Q34249737) (← links)
- AMPA receptor-dependent, light-evoked D-serine release acts on retinal ganglion cell NMDA receptors (Q36178806) (← links)
- Astrocyte-derived thrombospondins mediate the development of hippocampal presynaptic plasticity in vitro (Q36331898) (← links)
- Glycine transporter 1 modulates GABA release from amacrine cells by controlling occupancy of coagonist binding site of NMDA receptors (Q37147481) (← links)
- Retinal NMDA receptor function and expression are altered in a mouse lacking D-amino acid oxidase (Q37441423) (← links)
- Serine racemase: an unconventional enzyme for an unconventional transmitter. (Q38030879) (← links)
- New functions of Müller cells (Q38084625) (← links)
- Serine racemase is expressed in islets and contributes to the regulation of glucose homeostasis (Q39158726) (← links)
- Loss-of-function mutation of serine racemase attenuates excitotoxicity by intravitreal injection of N-methyl-D-aspartate. (Q40410315) (← links)
- Serine racemase deficiency attenuates choroidal neovascularization and reduces nitric oxide and VEGF levels by retinal pigment epithelial cells (Q47689628) (← links)
- Electroretinographic Abnormalities and Sex Differences Detected with Mesopic Adaptation in a Mouse Model of Schizophrenia: A and B Wave Analysis (Q89696141) (← links)