Pages that link to "Q54142755"

The following pages link to James M Slauch (Q54142755):

Displaying 24 items.

• SdiA, an N-acylhomoserine lactone receptor, becomes active during the transit of Salmonella enterica through the gastrointestinal tract of turtles (Q21144271) (← links)
• How does the oxidative burst of macrophages kill bacteria? Still an open question (Q28306532) (← links)
• The role of coupled positive feedback in the expression of the SPI1 type three secretion system in Salmonella (Q33649749) (← links)
• Either periplasmic tethering or protease resistance is sufficient to allow a SodC to protect Salmonella enterica serovar Typhimurium from phagocytic superoxide. (Q35563989) (← links)
• Periplasmic superoxide dismutase SodCI of Salmonella binds peptidoglycan to remain tethered within the periplasm. (Q36271516) (← links)
• The intestinal fatty acid propionate inhibits Salmonella invasion through the post-translational control of HilD (Q36636799) (← links)
• Adaptation to the host environment: regulation of the SPI1 type III secretion system in Salmonella enterica serovar Typhimurium. (Q36701362) (← links)
• HilD, HilC and RtsA constitute a feed forward loop that controls expression of the SPI1 type three secretion system regulator hilA in Salmonella enterica serovar Typhimurium. (Q40402350) (← links)
• The putative iron transport system SitABCD encoded on SPI1 is required for full virulence of Salmonella typhimurium. (Q40625112) (← links)
• In vivo expression technology for selection of bacterial genes specifically induced in host tissues. (Q40687575) (← links)
• Controlled Activity of the Salmonella Invasion-Associated Injectisome Reveals Its Intracellular Role in the Cytosolic Population. (Q47142460) (← links)
• Cytoplasmic Copper Detoxification in Salmonella Can Contribute to SodC Metalation but Is Dispensable during Systemic Infection (Q47781903) (← links)
• Construction of targeted single copy lac fusions using lambda Red and FLP-mediated site-specific recombination in bacteria (Q48298401) (← links)
• HilE Regulates HilD by Blocking DNA Binding in Salmonella enterica serovar Typhimurium (Q49597228) (← links)
• IVET and RIVET: use of gene fusions to identify bacterial virulence factors specifically induced in host tissues. (Q50118831) (← links)
• Effect of acetylation (O-factor 5) on the polyclonal antibody response to Salmonella typhimurium O-antigen. (Q50124125) (← links)
• Mutations that affect separate functions of OmpR the phosphorylated regulator of porin transcription in Escherichia coli. (Q52516905) (← links)
• Genetic analysis of the switch that controls porin gene expression in Escherichia coli K-12. (Q54722380) (← links)
• HilD, HilC, and RtsA Form Homodimers and Heterodimers to Regulate Expression of the Salmonella Pathogenicity Island I Type III Secretion System (Q89636193) (← links)
• The Small RNA PinT Contributes to PhoP-Mediated Regulation of the Salmonella Pathogenicity Island 1 Type III Secretion System in Salmonella enterica Serovar Typhimurium (Q91526710) (← links)
• Metal-independent variants of phosphoglycerate mutase promote resistance to nutritional immunity and retention of glycolysis during infection (Q92173459) (← links)
• PhoP-Mediated Repression of the SPI1 Type 3 Secretion System in Salmonella enterica Serovar Typhimurium (Q92641059) (← links)
• Oxygen-dependent regulation of SPI1 type three secretion system by small RNAs in Salmonella enterica serovar Typhimurium (Q93366073) (← links)
• Envelope Stress and Regulation of the Salmonella Pathogenicity Island I Type III Secretion System (Q96640188) (← links)