Pages that link to "Q53589254"

The following pages link to Nephrin loss in experimental diabetic nephropathy is prevented by deletion of protein kinase C alpha signaling in-vivo. (Q53589254):

Displaying 43 items.

• Membrane trafficking in podocyte health and disease (Q27015885) (← links)
• Dysregulated nephrin in diabetic nephropathy of type 2 diabetes: a cross sectional study (Q28729900) (← links)
• Podocytic PKC-alpha is regulated in murine and human diabetes and mediates nephrin endocytosis (Q33564333) (← links)
• Beneficial effects of the Rho kinase inhibitor Y27632 in murine puromycin aminonucleoside nephrosis (Q33650958) (← links)
• Toward the development of podocyte-specific drugs (Q33872286) (← links)
• Urinary Podocyte-Associated mRNA profile in Various Stages of Diabetic Nephropathy (Q33926992) (← links)
• Overexpression of Mafb in podocytes protects against diabetic nephropathy (Q34427651) (← links)
• Mechanisms by which heme oxygenase rescue renal dysfunction in obesity (Q34431848) (← links)
• Nox-4 deletion reduces oxidative stress and injury by PKC-α-associated mechanisms in diabetic nephropathy (Q34631886) (← links)
• Protein Kinase C-β Gene Variants and Type 2 Diabetes–Associated Kidney Failure: What Can We Learn From Gene Association Studies in Diabetic Nephropathy? (Q34776598) (← links)
• New insight into the molecular drug target of diabetic nephropathy (Q35125585) (← links)
• Knockout of Toll-Like Receptor-2 Attenuates Both the Proinflammatory State of Diabetes and Incipient Diabetic Nephropathy (Q35143026) (← links)
• Early-onset diabetic E1-DN mice develop albuminuria and glomerular injury typical of diabetic nephropathy. (Q35591301) (← links)
• Induction of hemeoxygenase-1 reduces glomerular injury and apoptosis in diabetic spontaneously hypertensive rats. (Q35924532) (← links)
• Influence of protein kinase C (PKC) on the prognosis of diabetic nephropathy patients (Q36464709) (← links)
• Tandem inhibition of PKC in Diαβetic nephropathy: it takes two to tango? (Q36720570) (← links)
• Protein kinase C beta inhibitors: a new therapeutic target for diabetic nephropathy and vascular complications (Q37165694) (← links)
• BMP signaling and podocyte markers are decreased in human diabetic nephropathy in association with CTGF overexpression (Q37234857) (← links)
• Targeting the protein kinase C family in the diabetic kidney: lessons from analysis of mutant mice (Q37399929) (← links)
• The heme oxygenase system suppresses perirenal visceral adiposity, abates renal inflammation and ameliorates diabetic nephropathy in Zucker diabetic fatty rats (Q37534705) (← links)
• Endocytic Trafficking at the Mature Podocyte Slit Diaphragm (Q37662983) (← links)
• The role of metabolic and haemodynamic factors in podocyte injury in diabetes (Q37838893) (← links)
• Pathophysiology of the diabetic kidney. (Q38111707) (← links)
• Role of protein kinase C in podocytes and development of glomerular damage in diabetic nephropathy (Q38270180) (← links)
• Cell biology of diabetic nephropathy: Roles of endothelial cells, tubulointerstitial cells and podocytes (Q38332511) (← links)
• Protein kinase C ϵ stabilizes β-catenin and regulates its subcellular localization in podocytes (Q38702391) (← links)
• PACSIN2 accelerates nephrin trafficking and is up-regulated in diabetic kidney disease. (Q38760772) (← links)
• Dual inhibition of classical protein kinase C-α and protein kinase C-β isoforms protects against experimental murine diabetic nephropathy. (Q39349244) (← links)
• Regulation of Nephrin Phosphorylation in Diabetes and Chronic Kidney Injury (Q39390200) (← links)
• Effects of insulin and high glucose on mobilization of slo1 BKCa channels in podocytes (Q39527086) (← links)
• Ameliorating Effects of l-Carnitine on Diabetic Podocyte Injury (Q39644251) (← links)
• GLCCI1 is a novel component associated with the PI3K signaling pathway in podocyte foot processes (Q39770356) (← links)
• PKC alpha mediates beta-arrestin2-dependent nephrin endocytosis in hyperglycemia (Q42715766) (← links)
• Targeted glomerular angiopoietin-1 therapy for early diabetic kidney disease. (Q42778066) (← links)
• Proteinuria in mice expressing PKB/SGK-resistant GSK3. (Q46262360) (← links)
• Podocyte-specific Nox4 deletion affords renoprotection in a mouse model of diabetic nephropathy (Q46648044) (← links)
• Nephrin Loss Can Be Used to Predict Remission and Long-term Renal Outcome in Patients With Minimal Change Disease (Q49165882) (← links)
• Inhibition of protein kinase C in diabetic nephropathy--where do we stand? (Q51732685) (← links)
• Niclosamide ethanolamine improves diabetes and diabetic kidney disease in mice. (Q55286343) (← links)
• Characterization of a novel disease-associated mutation within NPHS1 and its effects on nephrin phosphorylation and signaling (Q58744148) (← links)
• Rosmarinic acid monotherapy is better than the combination of rosmarinic acid and telmisartan in preventing podocyte detachment and inhibiting the progression of diabetic nephropathy in rats (Q90353253) (← links)
• Loss of Roundabout Guidance Receptor 2 (Robo2) in Podocytes Protects Adult Mice from Glomerular Injury by Maintaining Podocyte Foot Process Structure (Q90708189) (← links)
• Therapeutic Application of Nutraceuticals in Diabetic Nephropathy: Current Evidence and Future Implications (Q94950021) (← links)