Pages that link to "Q52583457"
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The following pages link to Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice. (Q52583457):
Displaying 50 items.
- Common genetic variation near the phospholamban gene is associated with cardiac repolarisation: meta-analysis of three genome-wide association studies (Q21143760) (← links)
- Regulation of cardiovascular cellular processes by S-nitrosylation (Q24300987) (← links)
- Specific role of neuronal nitric-oxide synthase when tethered to the plasma membrane calcium pump in regulating the beta-adrenergic signal in the myocardium (Q24320079) (← links)
- Uncoupled cardiac nitric oxide synthase mediates diastolic dysfunction (Q24650649) (← links)
- Acute Myocardial Response to Stretch: What We (don't) Know (Q26771506) (← links)
- Nitric oxide synthases in heart failure (Q26830146) (← links)
- Abnormal Ca(2 ) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balance (Q26852587) (← links)
- Molecular mechanisms of neuronal nitric oxide synthase in cardiac function and pathophysiology (Q27023508) (← links)
- Obligatory role of neuronal nitric oxide synthase in the heart's antioxidant adaptation with exercise (Q27302343) (← links)
- Neuronal nitric oxide synthase in hypertension - an update (Q28077204) (← links)
- Pathophysiological role of oxidative stress in systolic and diastolic heart failure and its therapeutic implications (Q28085102) (← links)
- Nitric oxide regulates cardiac intracellular Na⁺ and Ca²⁺ by modulating Na/K ATPase via PKCε and phospholemman-dependent mechanism (Q30413897) (← links)
- Displacement-encoded and manganese-enhanced cardiac MRI reveal that nNOS, not eNOS, plays a dominant role in modulating contraction and calcium influx in the mammalian heart (Q30423062) (← links)
- Impaired contractile function and calcium handling in hearts of cardiac-specific calcineurin b1-deficient mice (Q30491332) (← links)
- The Subcellular Localisation of Neuronal Nitric Oxide Synthase Determines the Downstream Effects of NO on Myocardial Function (Q30847242) (← links)
- Obesity and insulin resistance induce early development of diastolic dysfunction in young female mice fed a Western diet (Q33584753) (← links)
- Partial restoration of cardiac function with ΔPDZ nNOS in aged mdx model of Duchenne cardiomyopathy (Q33649295) (← links)
- Nitric oxide signalling and neuronal nitric oxide synthase in the heart under stress (Q33777846) (← links)
- Beta 3-adrenoreceptor regulation of nitric oxide in the cardiovascular system (Q33838174) (← links)
- Exercise-induced oxidative stress: cellular mechanisms and impact on muscle force production (Q34016655) (← links)
- Caveolin contributes to the modulation of basal and β-adrenoceptor stimulated function of the adult rat ventricular myocyte by simvastatin: a novel pleiotropic effect (Q34166022) (← links)
- NADPH oxidase-2 inhibition restores contractility and intracellular calcium handling and reduces arrhythmogenicity in dystrophic cardiomyopathy (Q34296737) (← links)
- Diesterified nitrone rescues nitroso-redox levels and increases myocyte contraction via increased SR Ca(2 ) handling (Q34540361) (← links)
- Neuronal nitric oxide synthase-dependent amelioration of diastolic dysfunction in rats with chronic renocardiac syndrome (Q35081519) (← links)
- Mechanical and non-mechanical functions of Dystrophin can prevent cardiac abnormalities in Drosophila (Q35122156) (← links)
- Nitric oxide signaling and the regulation of myocardial function. (Q35765975) (← links)
- Plasma membrane calcium pump (PMCA4)-neuronal nitric-oxide synthase complex regulates cardiac contractility through modulation of a compartmentalized cyclic nucleotide microdomain (Q35842187) (← links)
- Nitric Oxide Synthase 1 Modulates Basal and β-Adrenergic-Stimulated Contractility by Rapid and Reversible Redox-Dependent S-Nitrosylation of the Heart (Q36104908) (← links)
- Effects of increased systolic Ca(2 ) and β-adrenergic stimulation on Ca(2 ) transient decline in NOS1 knockout cardiac myocytes (Q36337502) (← links)
- Modulation of myocardial contraction by peroxynitrite (Q36461910) (← links)
- Regulation of endothelial nitric-oxide synthase (NOS) S-glutathionylation by neuronal NOS: evidence of a functional interaction between myocardial constitutive NOS isoforms (Q36481833) (← links)
- Neuronal nitric oxide synthase is indispensable for the cardiac adaptive effects of exercise. (Q36619416) (← links)
- Hydralazine and organic nitrates restore impaired excitation-contraction coupling by reducing calcium leak associated with nitroso-redox imbalance (Q36647370) (← links)
- ROS and endothelial nitric oxide synthase (eNOS)-dependent trafficking of angiotensin II type 2 receptor begets neuronal NOS in cardiac myocytes. (Q36785528) (← links)
- Protein Inhibitor of NOS1 Plays a Central Role in the Regulation of NOS1 Activity in Human Dilated Hearts. (Q37144494) (← links)
- Alterations in ryanodine receptors and related proteins in heart failure (Q37239340) (← links)
- Localisation of AMPK γ subunits in cardiac and skeletal muscles (Q37368921) (← links)
- Targeting cardiac sympatho-vagal imbalance using gene transfer of nitric oxide synthase (Q37376499) (← links)
- Neuronal nitric oxide synthase signaling within cardiac myocytes targets phospholamban. (Q37408590) (← links)
- Cyclic GMP signaling in cardiovascular pathophysiology and therapeutics (Q37420916) (← links)
- Reactive oxygen species: impact on skeletal muscle (Q37483737) (← links)
- Beta3-adrenergic receptors in cardiac and vascular tissues emerging concepts and therapeutic perspectives. (Q37798966) (← links)
- Evaluation of the Therapeutic Utility of Phosphodiesterase 5A Inhibition in the mdx Mouse Model of Duchenne Muscular Dystrophy (Q37893076) (← links)
- Nitric oxide synthase and cyclic GMP signaling in cardiac myocytes: From contractility to remodeling (Q37917873) (← links)
- Sub-cellular targeting of constitutive NOS in health and disease (Q37938337) (← links)
- Nitric oxide synthase regulation of cardiac excitation-contraction coupling in health and disease (Q38196172) (← links)
- Acute ascorbic acid infusion increases left ventricular diastolic function in postmenopausal women. (Q41652796) (← links)
- Sildenafil and B-Type Natriuretic Peptide Acutely Phosphorylate Titin and Improve Diastolic Distensibility In Vivo (Q42149439) (← links)
- Regulation of myocyte contraction via neuronal nitric oxide synthase: role of ryanodine receptor S-nitrosylation (Q42663223) (← links)
- The role of nitric oxide and reactive oxygen species in the positive inotropic response to mechanical stretch in the mammalian myocardium (Q42956438) (← links)