Pages that link to "Q48088995"
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The following pages link to Amyloid β-peptide oligomers stimulate RyR-mediated Ca2 release inducing mitochondrial fragmentation in hippocampal neurons and prevent RyR-mediated dendritic spine remodeling produced by BDNF. (Q48088995):
Displaying 50 items.
- Alzheimer's disease and insulin resistance: translating basic science into clinical applications (Q26996487) (← links)
- Elevated risk of type 2 diabetes for development of Alzheimer disease: a key role for oxidative stress in brain (Q27003253) (← links)
- Amyloid-β oligomers induce tau-independent disruption of BDNF axonal transport via calcineurin activation in cultured hippocampal neurons (Q30542949) (← links)
- Dendritic and axonal mechanisms of Ca2 elevation impair BDNF transport in Aβ oligomer-treated hippocampal neurons (Q30627148) (← links)
- RyR2-Mediated Ca2 Release and Mitochondrial ROS Generation Partake in the Synaptic Dysfunction Caused by Amyloid β Peptide Oligomers (Q33600132) (← links)
- Aluminium induced endoplasmic reticulum stress mediated cell death in SH-SY5Y neuroblastoma cell line is independent of p53. (Q33690089) (← links)
- Endoplasmic reticulum stress and inflammation in the central nervous system (Q33730922) (← links)
- Amyloid β production is regulated by β2-adrenergic signaling-mediated post-translational modifications of the ryanodine receptor. (Q33806103) (← links)
- Calcium ions promote formation of amyloid β-peptide (1-40) oligomers causally implicated in neuronal toxicity of Alzheimer's disease (Q33862507) (← links)
- Alzheimer's disease: relevant molecular and physiopathological events affecting amyloid-β brain balance and the putative role of PPARs (Q33959032) (← links)
- CDK5 is essential for soluble amyloid β-induced degradation of GKAP and remodeling of the synaptic actin cytoskeleton (Q33988231) (← links)
- Involvement of ryanodine receptors in neurotrophin-induced hippocampal synaptic plasticity and spatial memory formation (Q34582715) (← links)
- Peroxisome proliferator-activated receptor (PPAR) γ and PPARα agonists modulate mitochondrial fusion-fission dynamics: relevance to reactive oxygen species (ROS)-related neurodegenerative disorders? (Q34723198) (← links)
- The Aβ oligomer hypothesis for synapse failure and memory loss in Alzheimer's disease (Q35327219) (← links)
- Impaired mitochondrial biogenesis, defective axonal transport of mitochondria, abnormal mitochondrial dynamics and synaptic degeneration in a mouse model of Alzheimer's disease (Q35532902) (← links)
- How the Wnt signaling pathway protects from neurodegeneration: the mitochondrial scenario (Q35569964) (← links)
- Soluble amyloid-β oligomers as synaptotoxins leading to cognitive impairment in Alzheimer's disease (Q35643397) (← links)
- Glucose-Dependent Insulin Secretion in Pancreatic β-Cell Islets from Male Rats Requires Ca2 Release via ROS-Stimulated Ryanodine Receptors (Q35653939) (← links)
- Amyloid-β oligomers induce differential gene expression in adult human brain slices (Q35802078) (← links)
- Inhibition of choline acetyltransferase as a mechanism for cholinergic dysfunction induced by amyloid-β peptide oligomers. (Q36003808) (← links)
- Astaxanthin Protects Primary Hippocampal Neurons against Noxious Effects of Aβ-Oligomers. (Q36686173) (← links)
- The unfolded protein response in Alzheimer's disease (Q38100980) (← links)
- Deregulation of excitatory neurotransmission underlying synapse failure in Alzheimer's disease (Q38106483) (← links)
- Reciprocal disruption of neuronal signaling and Aβ production mediated by extrasynaptic NMDA receptors: a downward spiral. (Q38185273) (← links)
- Brain metabolite clearance: impact on Alzheimer's disease (Q38198972) (← links)
- Ryanodine receptors: physiological function and deregulation in Alzheimer disease. (Q38217975) (← links)
- Endoplasmic reticulum calcium stores in dendritic spines (Q38234887) (← links)
- Roles of Calcium Stores and Store-Operated Channels in Plasticity of Dendritic Spines. (Q38618700) (← links)
- Post-translational remodeling of ryanodine receptor induces calcium leak leading to Alzheimer's disease-like pathologies and cognitive deficits (Q38718079) (← links)
- Role of Endoplasmic Reticulum Stress and Unfolded Protein Responses in Health and Diseases (Q38805953) (← links)
- Loss of mitofusin 2 links beta-amyloid-mediated mitochondrial fragmentation and Cdk5-induced oxidative stress in neuron cells (Q38943638) (← links)
- PPARs in the central nervous system: roles in neurodegeneration and neuroinflammation. (Q39145017) (← links)
- Reversible epigenetic histone modifications and Bdnf expression in neurons with aging and from a mouse model of Alzheimer's disease (Q40114950) (← links)
- Amyloid β-peptide oligomers, ryanodine receptor-mediated Ca(2 ) release, and Wnt-5a/Ca(2 ) signaling: opposing roles in neuronal mitochondrial dynamics? (Q41770757) (← links)
- Ryanodine receptor-mediated Ca(2 ) release underlies iron-induced mitochondrial fission and stimulates mitochondrial Ca(2 ) uptake in primary hippocampal neurons (Q41776723) (← links)
- Canonical Wnt signaling protects hippocampal neurons from Aβ oligomers: role of non-canonical Wnt-5a/Ca(2 ) in mitochondrial dynamics (Q43031899) (← links)
- Long-term Aβ exposure augments mCa2 -independent mROS-mediated depletion of cardiolipin for the shift of a lethal transient mitochondrial permeability transition to its permanent mode in NARP cybrids: a protective targeting of melatonin (Q43421896) (← links)
- The prion protein ligand, stress-inducible phosphoprotein 1, regulates amyloid-β oligomer toxicity. (Q44331418) (← links)
- Memantine rescues transient cognitive impairment caused by high-molecular-weight aβ oligomers but not the persistent impairment induced by low-molecular-weight oligomers. (Q46518657) (← links)
- Development of an iron-selective antioxidant probe with protective effects on neuronal function. (Q47130659) (← links)
- Calcium Dysregulation in Alzheimer's Disease: A Target for New Drug Development (Q47928995) (← links)
- Shedding light on mitophagy in neurons: what is the evidence for PINK1/Parkin mitophagy in vivo? (Q47994216) (← links)
- Amyloid-β oligomers transiently inhibit AMP-activated kinase and cause metabolic defects in hippocampal neurons (Q48259811) (← links)
- Sub-lethal levels of amyloid β-peptide oligomers decrease non-transferrin-bound iron uptake and do not potentiate iron toxicity in primary hippocampal neurons. (Q48553659) (← links)
- The antioxidant N-acetylcysteine prevents the mitochondrial fragmentation induced by soluble amyloid-β peptide oligomers. (Q48686519) (← links)
- Modeling the neuron as a nanocommunication system to identify spatiotemporal molecular events in neurodegenerative disease. (Q55001034) (← links)
- Dysregulation of Intracellular Calcium Signaling in Alzheimer's Disease (Q57131389) (← links)
- A mathematical model for the effects of amyloid beta on intracellular calcium (Q58722978) (← links)
- Triclosan Impairs Hippocampal Synaptic Plasticity and Spatial Memory in Male Rats (Q59808712) (← links)
- N-Acetylcysteine Prevents the Spatial Memory Deficits and the Redox-Dependent RyR2 Decrease Displayed by an Alzheimer’s Disease Rat Model (Q60046048) (← links)