Pages that link to "Q46667580"
Jump to navigation
Jump to search
The following pages link to Direct effects of nucleoside reverse transcriptase inhibitors on rat cardiac mitochondrial bioenergetics (Q46667580):
Displaying 13 items.
- The role of protease inhibitors in the pathogenesis of HIV-associated lipodystrophy: cellular mechanisms and clinical implications (Q24598812) (← links)
- The association of HIV infection with left ventricular mass/hypertrophy (Q33570964) (← links)
- Absence of a universal mechanism of mitochondrial toxicity by nucleoside analogs (Q35879021) (← links)
- HIV-1 antiretrovirals induce oxidant injury and increase intima-media thickness in an atherogenic mouse model (Q37188672) (← links)
- Mitochondrial DNA impairment in nucleoside reverse transcriptase inhibitor-associated cardiomyopathy (Q37239515) (← links)
- BMS-986001, an HIV nucleoside reverse transcriptase inhibitor, does not degrade mitochondrial DNA in long-term primary cultures of cells isolated from human kidney, muscle, and adipose tissue (Q37335637) (← links)
- Increased mtDNA levels without change in mitochondrial enzymes in peripheral blood mononuclear cells of infants born to HIV-infected mothers on antiretroviral therapy (Q37492507) (← links)
- Electrocardiographic findings in a cross-sectional study of human immunodeficiency virus (HIV) patients in Enugu, south-east Nigeria (Q37685608) (← links)
- Adenosine 3',5'-cyclic monophosphate (cAMP)-dependent phosphoregulation of mitochondrial complex I is inhibited by nucleoside reverse transcriptase inhibitors (Q38568978) (← links)
- Isoproterenol cytotoxicity is dependent on the differentiation state of the cardiomyoblast H9c2 cell line (Q39566710) (← links)
- Assessment of cardiac diastolic dysfunction in HIV-infected people without cardiovascular symptoms in China (Q83378479) (← links)
- Intracellular metabolism and potential cardiotoxicity of a β-D-2'-C-methyl-2,6-diaminopurine ribonucleoside phosphoramidate that inhibits hepatitis C virus replication (Q90603791) (← links)
- Antiretroviral drug-induced endothelial dysfunction is improved by dual PPARα/γ stimulation in obesity (Q91711355) (← links)