Pages that link to "Q46640585"
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The following pages link to Gene expression profiles of vascular smooth muscle show differential expression of mitogen-activated protein kinase pathways during captopril therapy of heart failure (Q46640585):
Displaying 9 items.
- Mechanisms of Vascular Smooth Muscle Contraction and the Basis for Pharmacologic Treatment of Smooth Muscle Disorders (Q26753129) (← links)
- Losartan decreases p42/44 MAPK signaling and preserves LZ MYPT1 expression (Q33428533) (← links)
- Regulation of heat shock protein 60 and 72 expression in the failing heart (Q33621507) (← links)
- Increased degradation of MYPT1 contributes to the development of tolerance to nitric oxide in porcine pulmonary artery (Q33994665) (← links)
- Mitochondrial OPA1, apoptosis, and heart failure (Q37341059) (← links)
- The potential role of MLC phosphatase and MAPK signalling in the pathogenesis of vascular dysfunction in heart failure (Q37361567) (← links)
- Degradation of leucine zipper-positive isoform of MYPT1 may contribute to development of nitrate tolerance (Q43235396) (← links)
- A transcriptomic model to predict increase in fibrous cap thickness in response to high-dose statin treatment: Validation by serial intracoronary OCT imaging (Q92280999) (← links)
- The vasculature in HFpEF vs HFrEF: differences in contractile protein expression produce distinct phenotypes (Q92465833) (← links)