Pages that link to "Q46492867"
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The following pages link to Amyloid fibril formation and seeding by wild-type human lysozyme and its disease-related mutational variants. (Q46492867):
Displaying 50 items.
- The structural basis of protein folding and its links with human disease (Q24524125) (← links)
- A camelid antibody fragment inhibits the formation of amyloid fibrils by human lysozyme (Q27641839) (← links)
- A Nanobody Binding to Non-Amyloidogenic Regions of the Protein Human Lysozyme Enhances Partial Unfolding but Inhibits Amyloid Fibril Formation (Q27679384) (← links)
- Prediction of sequence-dependent and mutational effects on the aggregation of peptides and proteins (Q28281645) (← links)
- Low levels of asparagine deamidation can have a dramatic effect on aggregation of amyloidogenic peptides: implications for the study of amyloid formation (Q28345108) (← links)
- The roles of turn formation and cross-strand interactions in fibrillization of peptides derived from the OspA single-layer beta-sheet (Q28359810) (← links)
- Ultrastructural organization of amyloid fibrils by atomic force microscopy (Q28386688) (← links)
- Protéine BIGH3 : mutation du codon 124 et amylose cornéenne (Q29029715) (← links)
- Amyloid-like fibril formation in an all beta-barrel protein. Partially structured intermediate state(s) is a precursor for fibril formation. (Q30164898) (← links)
- Prolonged glycation of hen egg white lysozyme generates non amyloidal structures. (Q30353896) (← links)
- The impact of protein disulfide bonds on the amyloid fibril morphology. (Q30360911) (← links)
- Functional amyloids in the mouse sperm acrosome (Q30583694) (← links)
- RNA aptamers generated against oligomeric Abeta40 recognize common amyloid aptatopes with low specificity but high sensitivity (Q30879431) (← links)
- Amyloid protofilaments from the calcium-binding protein equine lysozyme: formation of ring and linear structures depends on pH and metal ion concentration (Q31148826) (← links)
- Observation of sequence specificity in the seeding of protein amyloid fibrils. (Q33204175) (← links)
- Fibrils from designed non-amyloid-related synthetic peptides induce AA-amyloidosis during inflammation in an animal model (Q33479158) (← links)
- Computational study of aggregation mechanism in human lysozyme[D67H] (Q33632612) (← links)
- Spatial Extent of Charge Repulsion Regulates Assembly Pathways for Lysozyme Amyloid Fibrils (Q33869235) (← links)
- α-synuclein reactive antibodies as diagnostic biomarkers in blood sera of Parkinson's disease patients. (Q33889446) (← links)
- Transmissibility of systemic amyloidosis by a prion-like mechanism (Q34068073) (← links)
- Simulations of human lysozyme: probing the conformations triggering amyloidosis (Q34187378) (← links)
- Analysis of the native structure, stability and aggregation of biotinylated human lysozyme (Q34483083) (← links)
- Development of a label-free aptasensor for monitoring the self-association of lysozyme. (Q34716175) (← links)
- A brief overview of amyloids and Alzheimer's disease. (Q34868191) (← links)
- Unfolded proteins studied by Raman optical activity (Q34989888) (← links)
- Carnosine's effect on amyloid fibril formation and induced cytotoxicity of lysozyme (Q35069891) (← links)
- Intrinsic determinants of Aβ(12-24) pH-dependent self-assembly revealed by combined computational and experimental studies. (Q35225705) (← links)
- Disease‐related amyloidogenic variants of human lysozyme trigger the unfolded protein response and disturb eye development inDrosophila melanogaster (Q35648011) (← links)
- Type IV pilus structure and bacterial pathogenicity (Q35752406) (← links)
- Anti-fibrillation propensity of a flavonoid baicalein against the fibrils of hen egg white lysozyme: potential therapeutics for lysozyme amyloidosis (Q35837281) (← links)
- Stabilization of partially folded states in protein folding/misfolding transitions by hydrostatic pressure (Q36219394) (← links)
- Equine lysozyme: the molecular basis of folding, self-assembly and innate amyloid toxicity. (Q36833026) (← links)
- Heat-induced fibrillation of BclXL apoptotic repressor (Q36997143) (← links)
- Guiding protein aggregation with macromolecular crowding (Q37178759) (← links)
- Amyloid formation by the pro-inflammatory S100A8/A9 proteins in the ageing prostate (Q37182479) (← links)
- Pressure-dissociable reversible assembly of intrinsically denatured lysozyme is a precursor for amyloid fibrils (Q37626820) (← links)
- Misfolding of amyloidogenic proteins and their interactions with membranes (Q38223686) (← links)
- Effect of Cinnamomum Verum Extract on the Amyloid Formation of Hen Egg-white Lysozyme and Study of its Possible Role in Alzheimer's Disease (Q38259182) (← links)
- Characterization of the structure and dynamics of amyloidogenic variants of human lysozyme by NMR spectroscopy (Q38272161) (← links)
- Characterization of heat induced spherulites of lysozyme reveals new insight on amyloid initiation. (Q38695657) (← links)
- Molecular Insight into Human Lysozyme and Its Ability to Form Amyloid Fibrils in High Concentrations of Sodium Dodecyl Sulfate: A View from Molecular Dynamics Simulations (Q38808079) (← links)
- ThT 101: a primer on the use of thioflavin T to investigate amyloid formation (Q38844934) (← links)
- Eugenol prevents amyloid formation of proteins and inhibits amyloid-induced hemolysis (Q38987418) (← links)
- S100A6 amyloid fibril formation is calcium-modulated and enhances superoxide dismutase-1 (SOD1) aggregation (Q39257498) (← links)
- Molecular interactions of amyloid nanofibrils with biological aggregation modifiers: implications for cytotoxicity mechanisms and biomaterial design (Q39384630) (← links)
- Thermally induced fibrillar aggregation of hen egg white lysozyme (Q40312596) (← links)
- Does the cytotoxic effect of transient amyloid oligomers from common equine lysozyme in vitro imply innate amyloid toxicity? (Q40485526) (← links)
- Analysis of core region from egg white lysozyme forming amyloid fibrils (Q41855790) (← links)
- Lysozyme Mutants Accumulate in Cells while Associated at their N-terminal Alpha-domain with the Endoplasmic Reticulum Chaperone GRP78/BiP. (Q41884061) (← links)
- A non-natural variant of human lysozyme (I59T) mimics the in vitro behaviour of the I56T variant that is responsible for a form of familial amyloidosis (Q41997964) (← links)