Pages that link to "Q46327841"
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The following pages link to Huntingtin spheroids and protofibrils as precursors in polyglutamine fibrilization (Q46327841):
Displaying 50 items.
- Ubiquitin-proteasome system involvement in Huntington's disease (Q21129327) (← links)
- Misfolding and amyloid aggregation of apomyoglobin (Q26830356) (← links)
- Disease-Associated Polyglutamine Stretches in Monomeric Huntingtin Adopt a Compact Structure (Q27677065) (← links)
- Role of Different Alpha-Synuclein Strains in Synucleinopathies, Similarities with other Neurodegenerative Diseases (Q28082714) (← links)
- Huntingtin aggregation and toxicity in Huntington's disease (Q28204163) (← links)
- Protein aggregation and neurodegenerative disease (Q28273600) (← links)
- Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death (Q28287762) (← links)
- Nuclear localization of a non-caspase truncation product of atrophin-1, with an expanded polyglutamine repeat, increases cellular toxicity (Q28593520) (← links)
- Huntingtin bodies sequester vesicle-associated proteins by a polyproline-dependent interaction. (Q30164317) (← links)
- Differential effects of glycation on protein aggregation and amyloid formation. (Q30374807) (← links)
- An Intein-based Strategy for the Production of Tag-free Huntingtin Exon 1 Proteins Enables New Insights into the Polyglutamine Dependence of Httex1 Aggregation and Fibril Formation (Q30385936) (← links)
- Monoclonal antibodies recognize distinct conformational epitopes formed by polyglutamine in a mutant huntingtin fragment (Q30490726) (← links)
- Mutant huntingtin fragments form oligomers in a polyglutamine length-dependent manner in vitro and in vivo (Q30494325) (← links)
- Detection of Mutant Huntingtin Aggregation Conformers and Modulation of SDS-Soluble Fibrillar Oligomers by Small Molecules (Q30547549) (← links)
- Super-resolution fluorescence of huntingtin reveals growth of globular species into short fibers and coexistence of distinct aggregates (Q30610715) (← links)
- Polyglutamine toxicity in yeast induces metabolic alterations and mitochondrial defects (Q30663550) (← links)
- Delayed emergence of subdiffraction-sized mutant huntingtin fibrils following inclusion body formation (Q30724979) (← links)
- Formation and toxicity of soluble polyglutamine oligomers in living cells (Q30994681) (← links)
- Interaction of huntingtin fragments with brain membranes--clues to early dysfunction in Huntington's disease (Q33231356) (← links)
- Screening for modulators of aggregation with a microplate elongation assay (Q33260630) (← links)
- The interplay between PolyQ and protein context delays aggregation by forming a reservoir of protofibrils (Q33268282) (← links)
- Molecular pathogenesis and cellular pathology of spinocerebellar ataxia type 7 neurodegeneration (Q33328864) (← links)
- Conformational targeting of fibrillar polyglutamine proteins in live cells escalates aggregation and cytotoxicity (Q33459255) (← links)
- Biophysical underpinnings of the repeat length dependence of polyglutamine amyloid formation (Q33675599) (← links)
- The chaperonin TRiC controls polyglutamine aggregation and toxicity through subunit-specific interactions (Q33693921) (← links)
- Differential hydrophobicity drives self-assembly in Huntington's disease (Q33713859) (← links)
- Structure-function relationships of pre-fibrillar protein assemblies in Alzheimer's disease and related disorders (Q33716773) (← links)
- Fibril polymorphism affects immobilized non-amyloid flanking domains of huntingtin exon1 rather than its polyglutamine core (Q33761247) (← links)
- Atomistic simulations of the effects of polyglutamine chain length and solvent quality on conformational equilibria and spontaneous homodimerization (Q33761386) (← links)
- Monomeric, oligomeric and polymeric proteins in huntington disease and other diseases of polyglutamine expansion (Q33788913) (← links)
- Polyglutamine induced misfolding of huntingtin exon1 is modulated by the flanking sequences (Q33818809) (← links)
- Formation of morphologically similar globular aggregates from diverse aggregation-prone proteins in mammalian cells (Q33911387) (← links)
- Suppression of Huntington's disease pathology in Drosophila by human single-chain Fv antibodies. (Q33914222) (← links)
- Investigating the structural impact of the glutamine repeat in huntingtin assembly (Q33922305) (← links)
- In-cell aggregation of a polyglutamine-containing chimera is a multistep process initiated by the flanking sequence (Q33941017) (← links)
- Orthogonal cross-seeding: an approach to explore protein aggregates in living cells (Q33958095) (← links)
- Assessing the contribution of heterogeneous distributions of oligomers to aggregation mechanisms of polyglutamine peptides (Q34181169) (← links)
- Protein aggregation and aggregate toxicity: new insights into protein folding, misfolding diseases and biological evolution (Q34225700) (← links)
- Hsp70 and Hsp40 Functionally Interact with Soluble Mutant Huntingtin Oligomers in a Classic ATP-dependent Reaction Cycle (Q34352145) (← links)
- Polyglutamine amyloid core boundaries and flanking domain dynamics in huntingtin fragment fibrils determined by solid-state nuclear magnetic resonance. (Q34414446) (← links)
- Full motor recovery despite striatal neuron loss and formation of irreversible amyloid-like inclusions in a conditional mouse model of Huntington's disease. (Q34461236) (← links)
- Polyglutamine fibrillogenesis: the pathway unfolds (Q34466014) (← links)
- Simultaneous measurement of amyloid fibril formation by dynamic light scattering and fluorescence reveals complex aggregation kinetics (Q34563267) (← links)
- A compact beta model of huntingtin toxicity (Q34624307) (← links)
- Abnormal mitochondrial dynamics, mitochondrial loss and mutant huntingtin oligomers in Huntington's disease: implications for selective neuronal damage (Q34627072) (← links)
- Type 2 transglutaminase in Huntington’s disease: a double‐edged sword with clinical potential (Q34775357) (← links)
- The Josephin domain determines the morphological and mechanical properties of ataxin-3 fibrils (Q34800791) (← links)
- Structural formation of huntingtin exon 1 aggregates probed by small-angle neutron scattering. (Q34978135) (← links)
- Disulfide scrambling in superoxide dismutase 1 reduces its cytotoxic effect in cultured cells and promotes protein aggregation (Q35021849) (← links)
- Location trumps length: polyglutamine-mediated changes in folding and aggregation of a host protein (Q35051262) (← links)