Pages that link to "Q46128976"
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The following pages link to Complement mRNA in the mammalian brain: responses to Alzheimer's disease and experimental brain lesioning (Q46128976):
Displaying 50 items.
- Human oligodendroglial cells express low levels of C1 inhibitor and membrane cofactor protein mRNAs (Q24791117) (← links)
- Using animal models to determine the significance of complement activation in Alzheimer's disease (Q24796236) (← links)
- Inflammation and Alzheimer's disease (Q28138617) (← links)
- Microarray reveals complement components are regulated in the serum-deprived rat retinal ganglion cell line (Q28565397) (← links)
- The role of complement in Alzheimer's disease pathology (Q33971345) (← links)
- Complement components of the innate immune system in health and disease in the CNS. (Q33974402) (← links)
- Contribution of complement activation pathways to neuropathology differs among mouse models of Alzheimer's disease (Q34550347) (← links)
- Roles of the complement system in human neurodegenerative disorders: pro-inflammatory and tissue remodeling activities. (Q34560179) (← links)
- Microglia and inflammatory mechanisms in the clearance of amyloid beta peptide (Q34963822) (← links)
- Yin and Yang: complement activation and regulation in Alzheimer’s disease (Q35563953) (← links)
- Up-regulated production and activation of the complement system in Alzheimer's disease brain (Q35786312) (← links)
- Spontaneous classical pathway activation and deficiency of membrane regulators render human neurons susceptible to complement lysis (Q35829491) (← links)
- Synaptic pathology and glial responses to neuronal injury precede the formation of senile plaques and amyloid deposits in the aging cerebral cortex (Q35834240) (← links)
- Characterization of rat C5a anaphylatoxin receptor (C5aR): cloning of rat C5aR cDNA and study of C5aR expression by rat astrocytes (Q36886221) (← links)
- What does complement do in Alzheimer's disease? Old molecules with new insights (Q37368120) (← links)
- Molecular profiles of schizophrenia in the CNS at different stages of illness. (Q37439158) (← links)
- The pivotal role of the complement system in aging and age-related macular degeneration: hypothesis re-visited (Q37644606) (← links)
- Recent rodent models for Alzheimer's disease: clinical implications and basic research (Q37956513) (← links)
- Complement pathway biomarkers and age-related macular degeneration. (Q38614012) (← links)
- History of innate immunity in neurodegenerative disorders (Q39542789) (← links)
- Association Between Microglia, Inflammatory Factors, and Complement with Loss of Hippocampal Mossy Fiber Synapses Induced by Trimethyltin (Q39985747) (← links)
- Complement biosynthesis in the central nervous system (Q40406085) (← links)
- The immunopathology of Alzheimer's disease and some related disorders (Q40807293) (← links)
- Characterization of the murine homolog of C1qR(P): identical cellular expression pattern, chromosomal location and functional activity of the human and murine C1qR(P). (Q40842986) (← links)
- Sulfated glycoprotein-2: an emerging molecular marker for neurodegeneration (Q40917158) (← links)
- Complement regulatory protein expression by a human oligodendrocyte cell line: cytokine regulation and comparison with astrocytes. (Q41153197) (← links)
- C1qB and clusterin mRNA increase in association with neurodegeneration in sporadic amyotrophic lateral sclerosis (Q41688205) (← links)
- Human postmortem brain-derived cerebrovascular smooth muscle cells express all genes of the classical complement pathway: a potential mechanism for vascular damage in cerebral amyloid angiopathy and Alzheimer's disease (Q42044152) (← links)
- Microglia, Alzheimer's disease, and complement (Q42322507) (← links)
- Acute inflammatory reaction following experimental intracerebral hemorrhage in rat. (Q42490148) (← links)
- C1q, the recognition subcomponent of the classical pathway of complement, drives microglial activation. (Q43187968) (← links)
- Complement C3 and C4 expression in C1q sufficient and deficient mouse models of Alzheimer's disease (Q43249892) (← links)
- Induction of complement C9 messenger RNAs in human neuronal cells by inflammatory stimuli: relevance to neurodegenerative disorders (Q43638529) (← links)
- Constitutive expression of proinflammatory complement components by subsets of neurons in the central nervous system (Q43906657) (← links)
- Absence of C1q leads to less neuropathology in transgenic mouse models of Alzheimer's disease. (Q44241464) (← links)
- Complement component C1q inhibits beta-amyloid- and serum amyloid P-induced neurotoxicity via caspase- and calpain-independent mechanisms. (Q48639462) (← links)
- Complement in the pathogenesis of Alzheimer's disease. (Q47709505) (← links)
- Complement regulators C1 inhibitor and CD59 do not significantly inhibit complement activation in Alzheimer disease (Q48182145) (← links)
- Transgenic mouse models of Alzheimer's disease and amyotrophic lateral sclerosis (Q48357472) (← links)
- Expression of complement C4 and C9 genes by human astrocytes (Q48364009) (← links)
- Importance of immunological and inflammatory processes in the pathogenesis and therapy of Alzheimer's disease (Q48373983) (← links)
- Experimental diffuse axonal injury induces enhanced neuronal C5a receptor mRNA expression in rats. (Q48580972) (← links)
- Neuronal expression of mRNAs for complement proteins of the classical pathway in Alzheimer brain (Q48592162) (← links)
- Elevated levels of the complement components C3 and factor B in ventricular cerebrospinal fluid of patients with traumatic brain injury (Q48791340) (← links)
- The presence of the complement cascade does not lead to neuronal cell death in primary hippocampal cultures (Q48822124) (← links)
- Complement activation by neurofibrillary tangles in Alzheimer's disease (Q48847179) (← links)
- Chronic complement C3 gene expression in the CNS of transgenic mice with astrocyte-targeted interleukin-6 expression (Q48901153) (← links)
- Do nonsteroidal anti-inflammatory drugs have a protective effect against dementia? (Q53213527) (← links)
- Detection of complement alternative pathway mRNA and proteins in the Alzheimer's disease brain. (Q53237812) (← links)
- Complement activation in amyloid plaques in Alzheimer's disease brains does not proceed further than C3 (Q53312031) (← links)