Pages that link to "Q46071152"
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The following pages link to Moderate heart dysfunction in mice with inducible cardiomyocyte-specific excision of the Serca2 gene (Q46071152):
Displaying 50 items.
- A network-oriented perspective on cardiac calcium signaling (Q27023389) (← links)
- Extreme sarcoplasmic reticulum volume loss and compensatory T-tubule remodeling after Serca2 knockout (Q28512641) (← links)
- Myocardial Structural and Biological Anomalies Induced by High Fat Diet in Psammomys obesus Gerbils (Q28553085) (← links)
- CaMKIIδB Mediates Aberrant NCX1 Expression and the Imbalance of NCX1/SERCA in Transverse Aortic Constriction-Induced Failing Heart (Q28740422) (← links)
- Genotype-Phenotype Map Characteristics of an In silico Heart Cell (Q28740827) (← links)
- Small-molecule activation of SERCA2a SUMOylation for the treatment of heart failure (Q30654383) (← links)
- Integrating multi-scale data to create a virtual physiological mouse heart (Q30734214) (← links)
- Toll-Like Receptor 9 Promotes Survival in SERCA2a KO Heart Failure Mice (Q33604761) (← links)
- There goes the neighborhood: pathological alterations in T-tubule morphology and consequences for cardiomyocyte Ca2 handling (Q33781282) (← links)
- Inhibition of miR-25 improves cardiac contractility in the failing heart. (Q34040266) (← links)
- Noncanonical EF-hand motif strategically delays Ca2 buffering to enhance cardiac performance (Q34327056) (← links)
- Calcium dynamics in the ventricular myocytes of SERCA2 knockout mice: A modeling study (Q34494396) (← links)
- Transcriptional effects of E3 ligase atrogin-1/MAFbx on apoptosis, hypertrophy and inflammation in neonatal rat cardiomyocytes (Q34555764) (← links)
- Disruption of sarcoendoplasmic reticulum calcium ATPase function in Drosophila leads to cardiac dysfunction (Q35009788) (← links)
- Phospholamban interactome in cardiac contractility and survival: A new vision of an old friend (Q35032742) (← links)
- Perturbed atrial calcium handling in an ovine model of heart failure: potential roles for reductions in the L-type calcium current (Q35033114) (← links)
- Mice carrying a conditional Serca2(flox) allele for the generation of Ca(2 ) handling-deficient mouse models (Q35037110) (← links)
- Prominent heart organ-level performance deficits in a genetic model of targeted severe and progressive SERCA2 deficiency (Q35041411) (← links)
- Identification of several mutations in ATP2C1 in Lebanese families: insight into the pathogenesis of Hailey-Hailey disease (Q35058332) (← links)
- Systolic dysfunction in cardiac-specific ligand-inducible MerCreMer transgenic mice (Q35087050) (← links)
- Endoplasmic Reticulum Ca2 Handling in Excitable Cells in Health and Disease (Q35122788) (← links)
- Targeting cardiomyocyte Ca2 homeostasis in heart failure (Q35763705) (← links)
- Sustained Toll-Like Receptor 9 Activation Promotes Systemic and Cardiac Inflammation, and Aggravates Diastolic Heart Failure in SERCA2a KO Mice (Q35804806) (← links)
- The angiotensin receptor-associated protein Atrap is a stimulator of the cardiac Ca2 -ATPase SERCA2a. (Q35970623) (← links)
- Synchrony of cardiomyocyte Ca(2 ) release is controlled by T-tubule organization, SR Ca(2 ) content, and ryanodine receptor Ca(2 ) sensitivity (Q36770437) (← links)
- The cardiomyocyte molecular clock, regulation of Scn5a, and arrhythmia susceptibility (Q36836267) (← links)
- Ca(2 )-Clock-Dependent Pacemaking in the Sinus Node Is Impaired in Mice with a Cardiac Specific Reduction in SERCA2 Abundance (Q36958008) (← links)
- Cardiac fibrosis in mice expressing an inducible myocardial-specific Cre driver. (Q37287472) (← links)
- SERCA2 activity is involved in the CNP-mediated functional responses in failing rat myocardium (Q37327400) (← links)
- SERCA2a gene therapy in heart failure: an anti-arrhythmic positive inotrope. (Q37418216) (← links)
- Divide and conquer: the application of organelle proteomics to heart failure (Q37605842) (← links)
- Multiple and diverse coexpression, location, and regulation of additional SERCA2 and SERCA3 isoforms in nonfailing and failing human heart. (Q37644983) (← links)
- Transcriptional mechanisms regulating Ca(2 ) homeostasis (Q37809267) (← links)
- The Ca2 pumps of the endoplasmic reticulum and Golgi apparatus. (Q37858339) (← links)
- Calcium signaling in cardiac myocytes. (Q37923638) (← links)
- No rest for the weary: diastolic calcium homeostasis in the normal and failing myocardium (Q38048564) (← links)
- Calcium flux balance in the heart (Q38065638) (← links)
- Cardiac sodium transport and excitation-contraction coupling (Q38115058) (← links)
- Biphasic decay of the Ca transient results from increased sarcoplasmic reticulum Ca leak (Q38895836) (← links)
- K201 (JTV-519) alters the spatiotemporal properties of diastolic Ca(2 ) release and the associated diastolic contraction during β-adrenergic stimulation in rat ventricular cardiomyocytes (Q39542247) (← links)
- Improving cardiac Ca2 transport into the sarcoplasmic reticulum in heart failure: lessons from the ubiquitous SERCA2b Ca2 pump (Q39748095) (← links)
- Beta-adrenergic stimulation maintains cardiac function in Serca2 knockout mice (Q41841957) (← links)
- Sodium accumulation in SERCA knockout-induced heart failure (Q41954642) (← links)
- Extracellular signal-regulated kinase activation during cardiac hypertrophy reduces sarcoplasmic/endoplasmic reticulum calcium ATPase 2 (SERCA2) transcription (Q42055700) (← links)
- Modeling Ca2 dynamics of mouse cardiac cells points to a critical role of SERCA's affinity for Ca2 . (Q42141286) (← links)
- Exercise training before cardiac-specific Serca2 disruption attenuates the decline in cardiac function in mice (Q42877943) (← links)
- Cre-loxP DNA recombination is possible with only minimal unspecific transcriptional changes and without cardiomyopathy in Tg(alphaMHC-MerCreMer) mice (Q42923082) (← links)
- Reduced SERCA2 abundance decreases the propensity for Ca2 wave development in ventricular myocytes (Q43216744) (← links)
- Tamoxifen administration routes and dosage for inducible Cre-mediated gene disruption in mouse hearts (Q43247329) (← links)
- β-Adrenergic Inhibition Prevents Action Potential and Calcium Handling Changes during Regional Myocardial Ischemia (Q45182678) (← links)