Pages that link to "Q45927250"
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The following pages link to Sodium channel cleavage is associated with aberrant neuronal activity and cognitive deficits in a mouse model of Alzheimer's disease. (Q45927250):
Displaying 36 items.
- Emerging Link between Alzheimer's Disease and Homeostatic Synaptic Plasticity (Q26750576) (← links)
- Entorhinal cortical defects in Tg2576 mice are present as early as 2-4 months of age. (Q27307826) (← links)
- An update on transcriptional and post-translational regulation of brain voltage-gated sodium channels (Q28087661) (← links)
- p38 MAP kinase-mediated NMDA receptor-dependent suppression of hippocampal hypersynchronicity in a mouse model of Alzheimer's disease (Q34416848) (← links)
- Early onset of hypersynchronous network activity and expression of a marker of chronic seizures in the Tg2576 mouse model of Alzheimer's disease (Q35175125) (← links)
- Connectivity and circuitry in a dish versus in a brain (Q35684598) (← links)
- Significance of transcytosis in Alzheimer's disease: BACE1 takes the scenic route to axons (Q35883077) (← links)
- MicroRNA-9 induces defective trafficking of Nav1.1 and Nav1.2 by targeting Navβ2 protein coding region in rat with chronic brain hypoperfusion (Q35933018) (← links)
- Analyzing and Modeling the Dysfunction of Inhibitory Neurons in Alzheimer's Disease (Q36236517) (← links)
- Seizure-Induced Regulations of Amyloid-β, STEP61, and STEP61 Substrates Involved in Hippocampal Synaptic Plasticity. (Q36809843) (← links)
- Genetic suppression of transgenic APP rescues Hypersynchronous network activity in a mouse model of Alzeimer's disease (Q37633601) (← links)
- Early-Onset Network Hyperexcitability in Presymptomatic Alzheimer's Disease Transgenic Mice Is Suppressed by Passive Immunization with Anti-Human APP/Aβ Antibody and by mGluR5 Blockade. (Q37719047) (← links)
- BACE1 as a therapeutic target in Alzheimer's disease: rationale and current status (Q38120669) (← links)
- Potential Similarities in Temporal Lobe Epilepsy and Alzheimer’s Disease: From Clinic to Pathology (Q38218353) (← links)
- Network abnormalities and interneuron dysfunction in Alzheimer disease (Q38796950) (← links)
- Ion channel regulation by β-secretase BACE1 - enzymatic and non-enzymatic effects beyond Alzheimer's disease. (Q38851332) (← links)
- Epileptic activity in Alzheimer's disease: causes and clinical relevance (Q39192571) (← links)
- Altered intrinsic excitability of hippocampal CA1 pyramidal neurons in aged PDAPP mice (Q40362793) (← links)
- APP mouse models for Alzheimer's disease preclinical studies (Q41582196) (← links)
- The E280A presenilin mutation reduces voltage-gated sodium channel levels in neuronal cells (Q41991328) (← links)
- BACE1-Dependent Neuregulin-1 Signaling: An Implication for Schizophrenia (Q42004797) (← links)
- Corticothalamic network dysfunction and behavioral deficits in a mouse model of Alzheimer's disease (Q42423885) (← links)
- Blood genome-wide transcriptional profiles reflect broad molecular impairments and strong blood-brain links in Alzheimer's disease (Q45867306) (← links)
- Changes of Functional and Directed Resting-State Connectivity Are Associated with Neuronal Oscillations, ApoE Genotype and Amyloid Deposition in Mild Cognitive Impairment (Q47132754) (← links)
- Navβ2 knockdown improves cognition in APP/PS1 mice by partially inhibiting seizures and APP amyloid processing (Q47147189) (← links)
- Disrupted hippocampal sharp-wave ripple-associated spike dynamics in a transgenic mouse model of dementia (Q47737770) (← links)
- ΔFosB Regulates Gene Expression and Cognitive Dysfunction in a Mouse Model of Alzheimer's Disease. (Q48199243) (← links)
- Reduced cooperativity of voltage-gated sodium channels in the hippocampal interneurons of an aged mouse model of Alzheimer's disease (Q50089415) (← links)
- Stimulation of entorhinal cortex-dentate gyrus circuitry is antidepressive. (Q52584553) (← links)
- Nav1.1-Overexpressing Interneuron Transplants Restore Brain Rhythms and Cognition in a Mouse Model of Alzheimer's Disease (Q52689780) (← links)
- Genetic and molecular basis of epilepsy-related cognitive dysfunction (Q89576661) (← links)
- Amyloid β-Induced Upregulation of Nav1.6 Underlies Neuronal Hyperactivity in Tg2576 Alzheimer's Disease Mouse Model (Q90183474) (← links)
- Exercise-Induced Cognitive Improvement Is Associated with Sodium Channel-Mediated Excitability in APP/PS1 Mice (Q91636820) (← links)
- GluN2A NMDA Receptor Enhancement Improves Brain Oscillations, Synchrony, and Cognitive Functions in Dravet Syndrome and Alzheimer's Disease Models (Q92659597) (← links)
- Critical analysis of the use of β-site amyloid precursor protein-cleaving enzyme 1 inhibitors in the treatment of Alzheimer's disease (Q97570180) (← links)
- Behavioral and neural network abnormalities in human APP transgenic mice resemble those of App knock-in mice and are modulated by familial Alzheimer's disease mutations but not by inhibition of BACE1 (Q99402092) (← links)