Pages that link to "Q45861661"

The following pages link to Impaired nuclear functions lead to increased senescence and inefficient differentiation in human myoblasts with a dominant p.R545C mutation in the LMNA gene (Q45861661):

Displaying 14 items.

• Genome-wide analysis links emerin to neuromuscular junction activity in Caenorhabditis elegans. (Q33741805) (← links)
• Altered chromosomal positioning, compaction, and gene expression with a lamin A/C gene mutation (Q33778527) (← links)
• Uncoordinated transcription and compromised muscle function in the lmna-null mouse model of Emery- Emery-Dreyfuss muscular dystrophy. (Q33834124) (← links)
• Nuclear lamins (Q34136884) (← links)
• A change in nuclear pore complex composition regulates cell differentiation (Q34248662) (← links)
• The role of nuclear lamin B1 in cell proliferation and senescence. (Q35642054) (← links)
• A Novel Lamin A Mutant Responsible for Congenital Muscular Dystrophy Causes Distinct Abnormalities of the Cell Nucleus. (Q36260910) (← links)
• Lamins in development, tissue maintenance and stress. (Q36440522) (← links)
• Lamina-associated polypeptide (LAP)2α and nucleoplasmic lamins in adult stem cell regulation and disease (Q38174396) (← links)
• Diverse lamin-dependent mechanisms interact to control chromatin dynamics. Focus on laminopathies (Q38284300) (← links)
• Retinoblastoma-independent regulation of cell proliferation and senescence by the p53-p21 axis in lamin A /C-depleted cells. (Q39550057) (← links)
• Gene expression, chromosome position and lamin A/C mutations (Q42854627) (← links)
• Lamina-associated polypeptide 1 is dispensable for embryonic myogenesis but required for postnatal skeletal muscle growth. (Q50480636) (← links)
• Clinical aspects of Emery-Dreifuss muscular dystrophy. (Q55262202) (← links)