Pages that link to "Q45179456"

The following pages link to Expression of toll-like receptors 2, 4 and 9 is increased in gingival tissue from patients with type 2 diabetes and chronic periodontitis (Q45179456):

Displaying 30 items.

• Host response mechanisms in periodontal diseases (Q26801846) (← links)
• Toll-like receptor 2 and type 2 diabetes (Q33634951) (← links)
• mRNA expression of pattern recognition receptors and their signaling mediators in healthy and diseased gingival tissues. (Q33663746) (← links)
• Lipopolysaccharide differentially affects the osteogenic differentiation of periodontal ligament stem cells and bone marrow mesenchymal stem cells through Toll-like receptor 4 mediated nuclear factor κB pathway (Q33826980) (← links)
• Resveratrol ameliorates experimental periodontitis in diabetic mice through negative regulation of TLR4 signaling (Q35080161) (← links)
• High acid phosphatase level in the gingival tissues of periodontitis subjects (Q35169611) (← links)
• Toll-Like Receptor 9-Mediated Inflammation Triggers Alveolar Bone Loss in Experimental Murine Periodontitis (Q35745585) (← links)
• Periodontal CD14 mRNA expression is downregulated in patients with chronic periodontitis and type 2 diabetes (Q35845658) (← links)
• Local complement-targeted intervention in periodontitis: proof-of-concept using a C5a receptor (CD88) antagonist (Q36417215) (← links)
• Microbial Nucleic Acid Sensing in Oral and Systemic Diseases (Q36428624) (← links)
• Toll-like receptor 4 signaling augments transforming growth factor-β responses: a novel mechanism for maintaining and amplifying fibrosis in scleroderma (Q36511520) (← links)
• Analysis of Expression and Localization of TLR-2 by Immunofluorescent Technique in Healthy and Inflammed Oral Tissues (Q37573131) (← links)
• A review of the evidence for pathogenic mechanisms that may link periodontitis and diabetes. (Q38102715) (← links)
• Chemokine function in periodontal disease and oral cavity cancer (Q38497234) (← links)
• Local inflammatory reactions in patients with diabetes and periodontitis (Q38562441) (← links)
• Ginkgolide B Suppresses TLR4-Mediated Inflammatory Response by Inhibiting the Phosphorylation of JAK2/STAT3 and p38 MAPK in High Glucose-Treated HUVECs (Q38635852) (← links)
• MicroRNA-126 Regulates Inflammatory Cytokine Secretion in Human Gingival Fibroblasts Under High Glucose Via Targeting Tumor Necrosis Factor Receptor Associated Factor 6. (Q38734421) (← links)
• The role of Toll-like receptors in periodontitis (Q38753462) (← links)
• Increased nucleic Acid receptor expression in chronic periodontitis (Q39424405) (← links)
• High glucose concentrations alter the biomineralization process in human osteoblastic cells (Q39442579) (← links)
• Necroptosis in the periodontal homeostasis: Signals emanating from dying cells. (Q40111437) (← links)
• High glucose-induced oxidative stress increases IL-8 production in human gingival epithelial cells. (Q40757892) (← links)
• Prioritization of candidate genes for periodontitis using multiple computational tools (Q42240245) (← links)
• A review of the evidence for pathogenic mechanisms that may link periodontitis and diabetes. (Q46100434) (← links)
• The role of Toll-like receptor 2 and 4 in gingival tissues of chronic periodontitis subjects with type 2 diabetes (Q48026434) (← links)
• Immunohistochemical localization of TLR2 and CD14 in gingival tissue of healthy individuals and patients with chronic periodontitis (Q50093319) (← links)
• Effects of periodontal treatment on inflammation and oxidative stress markers in patients with metabolic syndrome (Q51294392) (← links)
• Prediabetes Enhances Periodontal Inflammation Consistent With Activation of Toll-Like Receptor-Mediated Nuclear Factor-κB Pathway in Rats (Q51616056) (← links)
• Toll-like receptor 4-mediated hyper-responsiveness of gingival epithelial cells to lipopolysaccharide in high-glucose environments. (Q54206207) (← links)
• Response of Human Mesenchymal Stromal Cells from Periodontal Tissue to LPS Depends on the Purity but Not on the LPS Source (Q97882706) (← links)