Pages that link to "Q44884968"

The following pages link to Partial loss of presenilins causes seborrheic keratosis and autoimmune disease in mice (Q44884968):

Displaying 39 items.

• Presenilin modulates EGFR signaling and cell transformation by regulating the ubiquitin ligase Fbw7 (Q24301886) (← links)
• BACE1 and presenilin: two unusual aspartyl proteases involved in Alzheimer's disease (Q28257726) (← links)
• Partial loss of presenilin impairs age-dependent neuronal survival in the cerebral cortex (Q28588759) (← links)
• Presenilin/gamma-Secretase and Inflammation (Q33918695) (← links)
• Mutant presenilin-1 deregulated peripheral immunity exacerbates Alzheimer-like pathology (Q33935822) (← links)
• Proteases and proteolysis in Alzheimer disease: a multifactorial view on the disease process (Q34110227) (← links)
• Adult epidermal Notch activity induces dermal accumulation of T cells and neural crest derivatives through upregulation of jagged 1 (Q34240735) (← links)
• The γ-secretase complex: from structure to function (Q34677532) (← links)
• An Overview of Notch Signaling in Adult Tissue Renewal and Maintenance (Q35180702) (← links)
• Presenilins and γ-secretase: structure, function, and role in Alzheimer Disease (Q35658184) (← links)
• Familial frontotemporal dementia-associated presenilin-1 c.548G>T mutation causes decreased mRNA expression and reduced presenilin function in knock-in mice (Q35924427) (← links)
• Defective signal transduction in B lymphocytes lacking presenilin proteins (Q36458887) (← links)
• Therapeutic options in Alzheimer's disease (Q36511328) (← links)
• Disease-modifying therapies in Alzheimer's disease: how far have we come? (Q36656437) (← links)
• A two decade contribution of molecular cell biology to the centennial of Alzheimer's disease: are we progressing toward therapy? (Q36674481) (← links)
• γ-Secretase Activity Is Required for Regulated Intramembrane Proteolysis of Tumor Necrosis Factor (TNF) Receptor 1 and TNF-mediated Pro-apoptotic Signaling. (Q36674736) (← links)
• Notch1 and TGFbeta1 cooperatively regulate Foxp3 expression and the maintenance of peripheral regulatory T cells (Q36843721) (← links)
• Novel molecular and cellular therapeutic targets in acute lymphoblastic leukemia and lymphoproliferative disease (Q37248354) (← links)
• gamma-Secretase heterogeneity in the Aph1 subunit: relevance for Alzheimer's disease (Q37339100) (← links)
• Presenilins, Notch dose control the fate of pancreatic endocrine progenitors during a narrow developmental window (Q37362813) (← links)
• The different faces of Notch in T-helper-cell differentiation (Q37376021) (← links)
• BRI2 interacts with BACE1 and regulates its cellular levels by promoting its degradation and reducing its mRNA levels (Q37638750) (← links)
• gamma-secretases: from cell biology to therapeutic strategies (Q37686625) (← links)
• Gamma-secretase and the intramembrane proteolysis of Notch (Q37785197) (← links)
• Notch Signaling in the Regulation of Stem Cell Self-Renewal and Differentiation (Q37785203) (← links)
• Association of skin with the pathogenesis and treatment of neurodegenerative amyloidosis (Q37982715) (← links)
• Consequences of inhibiting amyloid precursor protein processing enzymes on synaptic function and plasticity (Q38026045) (← links)
• Treatment strategies targeting amyloid β-protein (Q38040990) (← links)
• Interleukin-1 receptor type 1 is a substrate for gamma-secretase-dependent regulated intramembrane proteolysis. (Q39919544) (← links)
• Presenilin-dependent gamma-secretase-mediated control of p53-associated cell death in Alzheimer's disease. (Q40269778) (← links)
• Treg-mediated immunosuppression involves activation of the Notch-HES1 axis by membrane-bound TGF-beta (Q42001733) (← links)
• Conditional inactivation of presenilin 1 prevents amyloid accumulation and temporarily rescues contextual and spatial working memory impairments in amyloid precursor protein transgenic mice. (Q46610675) (← links)
• Identification of gamma-secretase inhibitor potency determinants on presenilin (Q46886707) (← links)
• Conditionally targeted deletion of PSEN1 leads to diastolic heart dysfunction. (Q48272508) (← links)
• Deletion of presenilin 1 hydrophilic loop sequence leads to impaired gamma-secretase activity and exacerbated amyloid pathology. (Q48592298) (← links)
• Regulated intramembrane proteolysis: emergent role in cell signalling pathways (Q50100385) (← links)
• Defects of immune regulation in the presenilin-1 mutant knockin mouse. (Q53383098) (← links)
• Amyloid in skin and brain: What′s the link? (Q53415531) (← links)
• Loss of Presenilin 2 Function Is Associated with Defective LPS-Mediated Innate Immune Responsiveness. (Q53466041) (← links)