Pages that link to "Q44656426"

The following pages link to Catechol-o-methyltransferase (COMT) and proline dehydrogenase (PRODH) mRNAs in the dorsolateral prefrontal cortex in schizophrenia, bipolar disorder, and major depression. (Q44656426):

Displaying 37 items.

• The quantification of COMT mRNA in post mortem cerebellum tissue: diagnosis, genotype, methylation and expression (Q21261456) (← links)
• Functional analysis of genetic variation in catechol-O-methyltransferase (COMT): effects on mRNA, protein, and enzyme activity in postmortem human brain (Q24534089) (← links)
• Hypomethylation of MB-COMT promoter is a major risk factor for schizophrenia and bipolar disorder (Q24652360) (← links)
• COMT genetic variation confers risk for psychotic and affective disorders: a case control study (Q24816526) (← links)
• Schizophrenia genes, gene expression, and neuropathology: on the matter of their convergence (Q29618819) (← links)
• Differential Genetic and Epigenetic Regulation of catechol-O-methyltransferase is Associated with Impaired Fear Inhibition in Posttraumatic Stress Disorder. (Q30429540) (← links)
• Schizophrenia from a neural circuitry perspective: advancing toward rational pharmacological therapies (Q30484459) (← links)
• COMT Val158Met genotype selectively alters prefrontal [18F]fallypride displacement and subjective feelings of stress in response to a psychosocial stress challenge (Q31120316) (← links)
• The effects of gender and catechol O-methyltransferase (COMT) Val108/158Met polymorphism on emotion regulation in velo-cardio-facial syndrome (22q11.2 deletion syndrome): An fMRI study (Q34005465) (← links)
• Haplotypic association spanning the 22q11.21 genes COMT and ARVCF with schizophrenia (Q34344833) (← links)
• Pre-gestational stress alters stress-response of pubertal offspring rat in sexually dimorphic and hemispherically asymmetric manner (Q34800208) (← links)
• A second look: no effect of the COMT Val158Met polymorphism on conflict adaptation in youth with chromosome 22q11.2 deletion syndrome. (Q35090959) (← links)
• Genetic neuropathology of schizophrenia: new approaches to an old question and new uses for postmortem human brains (Q35153945) (← links)
• Genetics in schizophrenia: where are we and what next? (Q35237268) (← links)
• COMT Val(158)Met genotype determines the direction of cognitive effects produced by catechol-O-methyltransferase inhibition (Q35859549) (← links)
• Selective overexpression of Comt in prefrontal cortex rescues schizophrenia-like phenotypes in a mouse model of 22q11 deletion syndrome. (Q36203393) (← links)
• Genetic polymorphisms of the dopamine and serotonin systems modulate the neurophysiological response to feedback and risk taking in healthy humans (Q36420906) (← links)
• Effects of genetically altered brain glucocorticoid receptor action on behavior and adrenal axis regulation in mice (Q36524739) (← links)
• Metabolomic Profiling of Post-Mortem Brain Reveals Changes in Amino Acid and Glucose Metabolism in Mental Illness Compared with Controls (Q36743314) (← links)
• COMT val158met polymorphism and molecular alterations in the human dorsolateral prefrontal cortex: Differences in controls and in schizophrenia (Q36813596) (← links)
• Neuroplasticity of neocortical circuits in schizophrenia. (Q36931624) (← links)
• Catechol-O-methyltransferase (COMT): a gene contributing to sex differences in brain function, and to sexual dimorphism in the predisposition to psychiatric disorders (Q36931628) (← links)
• Catechol-O-methyltransferase val(158)met Polymorphism Interacts with Sex to Affect Face Recognition Ability. (Q37039056) (← links)
• No association of COMT Val158Met polymorphism with suicidal behavior or CSF monoamine metabolites in mood disorders (Q37173463) (← links)
• Candidate genes and the behavioral phenotype in 22q11.2 deletion syndrome. (Q37219660) (← links)
• Putative cortical dopamine levels affect cortical recruitment during planning. (Q37254819) (← links)
• Epistasis between the DAT 3' UTR VNTR and the COMT Val158Met SNP on cortical function in healthy subjects and patients with schizophrenia (Q37304041) (← links)
• Mood disorders after TBI. (Q37700806) (← links)
• COMT haplotypes suggest P2 promoter region relevance for schizophrenia. (Q40516110) (← links)
• Influence of naturally occurring variations in maternal care on prepulse inhibition of acoustic startle and the medial prefrontal cortical dopamine response to stress in adult rats. (Q45286312) (← links)
• A novel protein isoform of catechol O-methyltransferase (COMT): brain expression analysis in schizophrenia and bipolar disorder and effect of Val158Met genotype (Q46772745) (← links)
• Genetics and neuroimaging of attention and hypnotizability may elucidate placebo (Q46866885) (← links)
• Associations between neurodevelopmental genes, neuroanatomy, and ultra high risk symptoms of psychosis in 22q11.2 deletion syndrome. (Q48332128) (← links)
• Prefrontal dopamine levels determine the balance between cognitive stability and flexibility (Q48650321) (← links)
• No significant association between Catechol-O-methyl transferase (COMT) -287A/G gene polymorphism and Tourette's syndrome in family-based association study in Chinese Han population. (Q53090203) (← links)
• Catechol-O-methyltransferase haplotypes are associated with psychosis in Alzheimer disease. (Q53372506) (← links)
• COMT, 5-HTR2A, and SLC6A4 mRNA Expressions in First-Episode Antipsychotic-Naïve Schizophrenia and Association With Treatment Outcomes (Q59329370) (← links)