Pages that link to "Q44332855"
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The following pages link to Opioid inhibition of Ih via adenylyl cyclase (Q44332855):
Displaying 50 items.
- Pharmacology of airway afferent nerve activity (Q24792133) (← links)
- Comparison of the effects of [Phe1psi(CH2-NH)Gly2]nociceptin(1-13)NH2 in rat brain, rat vas deferens and CHO cells expressing recombinant human nociceptin receptors (Q28343380) (← links)
- Attacking pain at its source: new perspectives on opioids (Q30047792) (← links)
- Cardiac opioids (Q33899254) (← links)
- Cellular actions of opioids and other analgesics: implications for synergism in pain relief (Q33955119) (← links)
- Cellular actions of nociceptin: transduction mechanisms. (Q34040153) (← links)
- Cellular and molecular mechanisms of opioid action (Q34094138) (← links)
- High levels of endorphin and related pathologies of veterinary concern. A review (Q34098400) (← links)
- Regulation of firing frequency in nociceptive neurons by pro-inflammatory mediators. (Q34606323) (← links)
- Hyperpolarization-activated channels in trigeminal ganglia innervating healthy and pulp-exposed teeth (Q34655039) (← links)
- Regulation of recombinant and native hyperpolarization-activated cation channels. (Q36012074) (← links)
- A Novel Carbamoyloxy Arylalkanoyl Arylpiperazine Compound (SKL-NP) Inhibits Hyperpolarization-Activated Cyclic Nucleotide-Gated (HCN) Channel Currents in Rat Dorsal Root Ganglion Neurons (Q36166087) (← links)
- Characteristics of HCN channels and their participation in neuropathic pain (Q37156571) (← links)
- Dopamine D4 receptor excitation of lateral habenula neurons via multiple cellular mechanisms. (Q37251919) (← links)
- Peripheral mechanisms of pain and analgesia (Q37314270) (← links)
- Hyperpolarization-activated, cyclic nucleotide-gated (HCN) channels in the regulation of midbrain dopamine systems (Q37727999) (← links)
- Dual Peripheral Actions of Immune Cells in Neuropathic Pain (Q37828478) (← links)
- Modulation of Peripheral Sensory Neurons by the Immune System: Implications for Pain Therapy (Q37942106) (← links)
- Anti-nociceptive action of peripheral mu-opioid receptors by G-beta-gamma protein-mediated inhibition of TRPM3 channels. (Q38615910) (← links)
- Combination of Intrathecal Opioids with Bupivacaine Attenuates Opioid Dose Escalation in Chronic Noncancer Pain Patients (Q39258887) (← links)
- Mobilization of Ca2 from intracellular stores in transfected neuro2a cells by activation of multiple opioid receptor subtypes. (Q39455257) (← links)
- Characterization of prostanoid receptor‐evoked responses in rat sensory neurones (Q41029701) (← links)
- Opioid receptors and the regulation of ion conductances (Q41040009) (← links)
- Endogenous opioid regulation of hippocampal function (Q41191019) (← links)
- Where is the locus in opioid withdrawal? (Q41468959) (← links)
- Peripheral pain mechanisms (Q41587254) (← links)
- Enhancement of synaptic transmission by cyclic AMP modulation of presynaptic Ih channels (Q41716474) (← links)
- Local anaesthetics block hyperpolarization-activated inward current in rat small dorsal root ganglion neurones (Q42182136) (← links)
- Capsaicin Blocks the Hyperpolarization-Activated Inward Currents via TRPV1 in the Rat Dorsal Root Ganglion Neurons (Q42211363) (← links)
- Pacemaker channels in mouse thalamocortical neurones are regulated by distinct pathways of cAMP synthesis (Q42454411) (← links)
- Rat central ORL-1 receptor uncouples from adenylyl cyclase during membrane preparation. (Q42456478) (← links)
- Peripheral delta opioid receptors require priming for functional competence in vivo (Q43188585) (← links)
- The effects of endomorphin-1 and endomorphin-2 in CHO cells expressing recombinant mu-opioid receptors and SH-SY5Y cells (Q43260336) (← links)
- Contribution of the hyperpolarization-activated current (I(h)) to membrane potential and GABA release in hippocampal interneurons. (Q43657500) (← links)
- Prostaglandin E(2) inhibits calcium current in two sub-populations of acutely isolated mouse trigeminal sensory neurons. (Q43908344) (← links)
- Vasoactive intestinal polypeptide and pituitary adenylate cyclase-activating polypeptide activate hyperpolarization-activated cationic current and depolarize thalamocortical neurons in vitro. (Q44070778) (← links)
- Kappa-opioid receptor-mediated enhancement of the hyperpolarization-activated current (I(h)) through mobilization of intracellular calcium in rat nucleus raphe magnus (Q44340893) (← links)
- Coupling of the cloned rat kappa-opioid receptor to adenylyl cyclase is dependent on receptor expression. (Q44346843) (← links)
- μ-Opioid Peptides Inhibit Thalamic Neurons (Q44347905) (← links)
- cAMP-mediated mechanisms for pain sensitization during opioid withdrawal. (Q44348673) (← links)
- Increased Opioid Inhibition of GABA Release in Nucleus Accumbens during Morphine Withdrawal (Q44349727) (← links)
- Opioid inhibition of hippocampal interneurons via modulation of potassium and hyperpolarization-activated cation (Ih) currents. (Q44349901) (← links)
- Opioid-activated postsynaptic, inward rectifying potassium currents in whole cell recordings in substantia gelatinosa neurons (Q44350931) (← links)
- A novel δ opioid receptor‐mediated enhancement of GABAA receptor function induced by stress in ventral tegmental area neurons (Q44351247) (← links)
- Pharmacology of vagal afferent nerve activity in guinea pig airways (Q44376367) (← links)
- Serotonin reduces the hyperpolarization-activated current (Ih) in ventral tegmental area dopamine neurons: involvement of 5-HT2 receptors and protein kinase C. (Q44532530) (← links)
- Hyperpolarization-activated current (Ih) in primary auditory neurons (Q46184350) (← links)
- Role of the hyperpolarization-activated current Ih in somatosensory neurons (Q46308455) (← links)
- cAMP and cGMP contribute to sensory neuron hyperexcitability and hyperalgesia in rats with dorsal root ganglia compression (Q46667765) (← links)
- The role of pacemaker currents in neuropathic pain (Q46893224) (← links)