Pages that link to "Q44204259"
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The following pages link to Reduced glomerular thromboxane receptor sites and vasoconstrictor responses in diabetic rats (Q44204259):
Displaying 15 items.
- Receptors for atrial natriuretic peptide are decreased in the kidney of rats with streptozotocin-induced diabetes mellitus (Q34222755) (← links)
- Pathophysiology of the diabetic kidney. (Q38111707) (← links)
- Reduced proximal tubule angiotensin II receptor expression in streptozotocin-induced diabetes mellitus (Q38302116) (← links)
- Functional impairment of renal afferent arteriolar voltage-gated calcium channels in rats with diabetes mellitus (Q39774627) (← links)
- Hypertension in diabetic patients: an update of interventional studies to preserve renal function (Q40526153) (← links)
- Eicosanoids in the pathogenesis of the functional and structural alterations of the kidney in diabetes (Q40779242) (← links)
- Renal vascular reactivity to vasopressin in rats with diabetes mellitus (Q43813739) (← links)
- Effects of diabetes on the vascular response to nitric oxide and constrictor prostanoids: gender and regional differences. (Q44283312) (← links)
- Acute and chronic effects of thromboxane A2 inhibition on the renal hemodynamics in streptozotocin-induced diabetic rats (Q51638707) (← links)
- Effect of acute thromboxane A2 inhibition on the renal hemodynamics in a spontaneously non-insulin-dependent diabetic rat, Otsuka Long-Evans Tokushima Fatty rat. (Q54067147) (← links)
- High glucose inhibits cytosolic calcium signaling in cultured rat mesangial cells (Q70616851) (← links)
- Impairment of afferent arteriolar myogenic responsiveness in the galactose-fed rat is prevented by tolrestat (Q71657482) (← links)
- Activation of protein kinase C reduces thromboxane receptors in glomeruli and mesangial cells (Q72889615) (← links)
- Changes in reactivity towards 5-hydroxytryptamine in the renal vasculature of the diabetic spontaneously hypertensive rat (Q73510170) (← links)
- Thromboxane receptors in human kidney tissues (Q77990079) (← links)