Pages that link to "Q43059618"

The following pages link to Inhibition of phospholamban phosphorylation by O-GlcNAcylation: implications for diabetic cardiomyopathy (Q43059618):

Displaying 31 items.

• Global mass spectrometry and transcriptomics array based drug profiling provides novel insight into glucosamine induced endoplasmic reticulum stress (Q34634894) (← links)
• Protein O-GlcNAcylation and cardiovascular (patho)physiology (Q34683061) (← links)
• High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylation. (Q35535273) (← links)
• Muscle ring finger-3 protects against diabetic cardiomyopathy induced by a high fat diet. (Q35716907) (← links)
• MuRF2 regulates PPARγ1 activity to protect against diabetic cardiomyopathy and enhance weight gain induced by a high fat diet (Q35920226) (← links)
• The roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease (Q35994096) (← links)
• Chronic ingestion of a Western diet increases O-linked-β-N-acetylglucosamine (O-GlcNAc) protein modification in the rat heart (Q36024572) (← links)
• Excess protein O-GlcNAcylation and the progression of diabetic cardiomyopathy. (Q36312605) (← links)
• Cardiac O-GlcNAcylation blunts autophagic signaling in the diabetic heart (Q36336533) (← links)
• Overview: the maturing of proteomics in cardiovascular research (Q36406428) (← links)
• Post-translational protein modification by O-linked N-acetyl-glucosamine: its role in mediating the adverse effects of diabetes on the heart. (Q36484390) (← links)
• O-GlcNAc and the cardiovascular system. (Q37619691) (← links)
• Role of oxidative stress in diabetes-mediated vascular dysfunction: unifying hypothesis of diabetes revisited (Q38000380) (← links)
• Regulation of sarco(endo)plasmic reticulum Ca2 -ATPase and calsequestrin gene expression in the heart (Q38025471) (← links)
• The role of post-translational modifications in acute and chronic cardiovascular disease (Q38222880) (← links)
• The regulation of sarco(endo)plasmic reticulum calcium-ATPases (SERCA). (Q38366647) (← links)
• Cardioprotection in ischaemia-reperfusion injury: novel mechanisms and clinical translation (Q38546856) (← links)
• The role of CaMKII in diabetic heart dysfunction (Q38551638) (← links)
• Protein O-GlcNAcylation: emerging mechanisms and functions (Q39295247) (← links)
• SERCA control of cell death and survival (Q46330392) (← links)
• The Role of Stress-Induced O-GlcNAc Protein Modification in the Regulation of Membrane Transport (Q49874500) (← links)
• Ready display of antigenic peptides in a protein 'mimogen'. (Q52818612) (← links)
• Chronic O-GlcNAcylation and Diabetic Cardiomyopathy: The Bitterness of Glucose (Q58699161) (← links)
• Regulation of cardiac O-GlcNAcylation: More than just nutrient availability (Q89515695) (← links)
• O-GlcNAc stimulation: A new metabolic approach to treat septic shock (Q91855261) (← links)
• O-GlcNAc Transferase Promotes Compensated Cardiac Function and Protein Kinase A O-GlcNAcylation During Early and Established Pathological Hypertrophy From Pressure Overload (Q92311962) (← links)
• First characterization of glucose flux through the hexosamine biosynthesis pathway (HBP) in ex vivo mouse heart (Q92502878) (← links)
• Augmented O-GlcNAcylation attenuates intermittent hypoxia-induced cardiac remodeling through the suppression of NFAT and NF-κB activities in mice (Q92603358) (← links)
• Contribution of Impaired Insulin Signaling to the Pathogenesis of Diabetic Cardiomyopathy (Q92852811) (← links)
• Very Low-Density Lipoproteins of Metabolic Syndrome Modulates STIM1, Suppresses Store-Operated Calcium Entry, and Deranges Myofilament Proteins in Atrial Myocytes (Q92941380) (← links)
• Enhancement of O-linked N-acetylglucosamine modification promotes metastasis in patients with colorectal cancer and concurrent type 2 diabetes mellitus (Q98159007) (← links)