Pages that link to "Q42825163"
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The following pages link to Oncogenic and transcriptional cooperation with Ha-Ras requires phosphorylation of c-Jun on serines 63 and 73 (Q42825163):
Displaying 50 items.
- The N-terminal transactivation domain of ATF2 is a target for the co-operative activation of the c-jun promoter by p300 and 12S E1A (Q22010019) (← links)
- IkappaB kinases phosphorylate NF-kappaB p65 subunit on serine 536 in the transactivation domain (Q22010650) (← links)
- JNK1, a potential therapeutic target for hepatocellular carcinoma (Q23912822) (← links)
- Characterization of the physical interaction between estrogen receptor alpha and JUN proteins (Q24291491) (← links)
- T cell signal transduction and the role of CD7 in costimulation (Q24291521) (← links)
- Transformation suppressor protein Pdcd4 interferes with JNK-mediated phosphorylation of c-Jun and recruitment of the coactivator p300 by c-Jun (Q24302065) (← links)
- Constitutive c-jun N-terminal kinase activity in acute myeloid leukemia derives from Flt3 and affects survival and proliferation (Q24303657) (← links)
- Mutual regulation of c-Jun and ATF2 by transcriptional activation and subcellular localization (Q24306331) (← links)
- Menin uncouples Elk-1, JunD and c-Jun phosphorylation from MAP kinase activation (Q24306677) (← links)
- Signal transduction by tumor necrosis factor mediated by JNK protein kinases (Q24308037) (← links)
- JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain (Q24308838) (← links)
- Activation of cAMP and mitogen responsive genes relies on a common nuclear factor (Q24313652) (← links)
- Mammalian sterile 20-like kinase 3 (MST3) mediates oxidative-stress-induced cell death by modulating JNK activation (Q24314652) (← links)
- Selective interaction of JNK protein kinase isoforms with transcription factors (Q24317346) (← links)
- Mixed lineage kinase phosphorylates transcription factor E47 and inhibits TrkB expression to link neuronal death and survival pathways (Q24321504) (← links)
- A selective small-molecule inhibitor of c-Jun N-terminal kinase 1 (Q24338446) (← links)
- A new c-Jun N-terminal kinase (JNK)-interacting protein, Sab (SH3BP5), associates with mitochondria (Q24534841) (← links)
- Pin1 is overexpressed in breast cancer and cooperates with Ras signaling in increasing the transcriptional activity of c-Jun towards cyclin D1. (Q24535558) (← links)
- The tumor promoter arsenite stimulates AP-1 activity by inhibiting a JNK phosphatase (Q24561826) (← links)
- A common intermediary factor (p52/54) recognizing "acidic blob"-type domains is required for transcriptional activation by the Jun proteins (Q24600911) (← links)
- Control of cell cycle progression by c-Jun is p53 dependent (Q24607483) (← links)
- c-JUN prevents methylation of p16(INK4a) (and Cdk6): the villain turned bodyguard (Q24617313) (← links)
- The functional contrariety of JNK (Q24633325) (← links)
- A comprehensive pathway map of epidermal growth factor receptor signaling (Q24672490) (← links)
- Feedback regulation of p38 activity via ATF2 is essential for survival of embryonic liver cells (Q24680095) (← links)
- The prolyl isomerase Pin1 in breast development and cancer (Q24795560) (← links)
- Regulation of Stress Responses and Translational Control by Coronavirus (Q26744222) (← links)
- Prolyl isomerase Pin1 as a molecular switch to determine the fate of phosphoproteins (Q26825421) (← links)
- JNK signalling in cancer: in need of new, smarter therapeutic targets (Q27023869) (← links)
- Co-Targeting of JNK and HUNK in Resistant HER2-Positive Breast Cancer (Q27314840) (← links)
- Pathological impact of hepatitis B virus surface proteins on the liver is associated with the host genetic background (Q27317287) (← links)
- Landscape of Pin1 in the cell cycle (Q28081788) (← links)
- Oncogenic transformation by ras and fos is mediated by c-Jun N-terminal phosphorylation (Q28138110) (← links)
- Integrin LFA-1 interacts with the transcriptional co-activator JAB1 to modulate AP-1 activity (Q28141877) (← links)
- Signal transduction by the JNK group of MAP kinases (Q28142592) (← links)
- Transcriptional regulation of Syndecan-1 expression by growth factors (Q28145412) (← links)
- Mutations in protein kinase subdomain X differentially affect MEKK2 and MEKK1 activity (Q28186572) (← links)
- Distinct roles of Jun : Fos and Jun : ATF dimers in oncogenesis (Q28201509) (← links)
- Regulation of two JunD isoforms by Jun N-terminal kinases (Q28203115) (← links)
- AP-1 repressor protein JDP-2: inhibition of UV-mediated apoptosis through p53 down-regulation (Q28210595) (← links)
- Induction of BIM, a proapoptotic BH3-only BCL-2 family member, is critical for neuronal apoptosis (Q28212224) (← links)
- Mood stabilizers, glycogen synthase kinase-3beta and cell survival (Q28216404) (← links)
- Expression of peroxiredoxin 1 and 4 promotes human lung cancer malignancy (Q28248261) (← links)
- c-Jun NH2-terminal kinases target the ubiquitination of their associated transcription factors (Q28257088) (← links)
- Stimulation of hTAFII68 (NTD)-mediated transactivation by v-Src (Q28257266) (← links)
- The oncogene c-Jun impedes somatic cell reprogramming (Q28263863) (← links)
- The AP-1 repressor protein, JDP2, potentiates hepatocellular carcinoma in mice (Q28275462) (← links)
- Ras inhibition boosts galectin-7 at the expense of galectin-1 to sensitize cells to apoptosis (Q28287799) (← links)
- c-Jun can recruit JNK to phosphorylate dimerization partners via specific docking interactions (Q28298557) (← links)
- P21-activated protein kinase (PAK2)-mediated c-Jun phosphorylation at 5 threonine sites promotes cell transformation (Q28301616) (← links)