Pages that link to "Q42819869"

The following pages link to Reciprocal antagonism coordinates C-type natriuretic peptide and mitogen-signaling pathways in fibroblasts (Q42819869):

Displaying 41 items.

• Identification of a potent serum factor that causes desensitization of the receptor for C-Type natriuretic peptide (Q24802362) (← links)
• Calcium-dependent dephosphorylation mediates the hyperosmotic and lysophosphatidic acid-dependent inhibition of natriuretic peptide receptor-B/guanylyl cyclase-B (Q28282496) (← links)
• CNP gene expression is activated by Wnt signaling and correlates with Wnt4 expression during renal injury (Q28504731) (← links)
• Regulation of the Guanylyl Cyclase-B Receptor by Alternative Splicing (Q28508538) (← links)
• Natriuretic peptide receptor-B in adult rat ventricle is predominantly confined to the nonmyocyte population (Q28571905) (← links)
• Angiotensin II increases phosphodiesterase 5A expression in vascular smooth muscle cells: a mechanism by which angiotensin II antagonizes cGMP signaling (Q28575995) (← links)
• Guanylyl cyclase-linked natriuretic peptide receptors: structure and regulation (Q31816687) (← links)
• Hypoxia-induced left ventricular dysfunction in myoglobin-deficient mice (Q33967356) (← links)
• Guanylyl cyclase/natriuretic peptide receptor-A signaling antagonizes phosphoinositide hydrolysis, Ca(2 ) release, and activation of protein kinase C (Q34076973) (← links)
• Dephosphorylation and inactivation of NPR2 guanylyl cyclase in granulosa cells contributes to the LH-induced decrease in cGMP that causes resumption of meiosis in rat oocytes (Q34341855) (← links)
• A genetic model provides evidence that the receptor for atrial natriuretic peptide (guanylyl cyclase-A) inhibits cardiac ventricular myocyte hypertrophy (Q34486162) (← links)
• The functional genomics of guanylyl cyclase/natriuretic peptide receptor‐A: Perspectives and paradigms (Q34990330) (← links)
• Genetic inactivation of ERK1 and ERK2 in chondrocytes promotes bone growth and enlarges the spinal canal (Q35097630) (← links)
• Urinary C-type natriuretic peptide excretion: a potential novel biomarker for renal fibrosis during aging (Q35543215) (← links)
• Structure, Regulation, and Function of Mammalian Membrane Guanylyl Cyclase Receptors, With a Focus on Guanylyl Cyclase-A (Q35561813) (← links)
• Guanylyl cyclase / atrial natriuretic peptide receptor-A: role in the pathophysiology of cardiovascular regulation (Q35936915) (← links)
• Guanylyl cyclase/natriuretic peptide receptor-A signaling antagonizes the vascular endothelial growth factor-stimulated MAPKs and downstream effectors AP-1 and CREB in mouse mesangial cells (Q36369123) (← links)
• Membrane guanylyl cyclase receptors: an update (Q36525043) (← links)
• C-type natriuretic peptide in combination with sildenafil attenuates proliferation of rhabdomyosarcoma cells (Q36895049) (← links)
• Mutations in Tyr808 reveal a potential auto-inhibitory mechanism of guanylate cyclase-B regulation (Q36904037) (← links)
• Critical roles of the guanylyl cyclase B receptor in endochondral ossification and development of female reproductive organs (Q37693870) (← links)
• Role of sphingosine 1-phosphate in anti-atherogenic actions of high-density lipoprotein (Q37871354) (← links)
• Natriuretic peptides: molecular biology, pathophysiology and clinical implications for the cardiologist. (Q38120091) (← links)
• cGMP-dependent protein kinase inhibits serum-response element-dependent transcription by inhibiting rho activation and functions (Q38364180) (← links)
• Atrial natriuretic peptide type C induces a cell-cycle switch from proliferation to differentiation in brain-derived neurotrophic factor- or nerve growth factor-primed olfactory receptor neurons. (Q38522445) (← links)
• Advances in treatment of achondroplasia and osteoarthritis (Q38601075) (← links)
• Therapeutic effect of CNP on renal osteodystrophy by antagonizing the FGF-23/MAPK pathway (Q38605457) (← links)
• Molecular Physiology of Membrane Guanylyl Cyclase Receptors (Q38794203) (← links)
• The ras-GTPase activity of neurofibromin restrains ERK-dependent FGFR signaling during endochondral bone formation. (Q38961342) (← links)
• Csk mediates G-protein-coupled lysophosphatidic acid receptor-induced inhibition of membrane-bound guanylyl cyclase activity (Q40308269) (← links)
• Activation of protein kinase C stimulates the dephosphorylation of natriuretic peptide receptor-B at a single serine residue: a possible mechanism of heterologous desensitization (Q40865565) (← links)
• Natriuretic peptides inhibit G protein activation. Mediation through cross-talk between cyclic GMP-dependent protein kinase and regulators of G protein-signaling proteins (Q41723060) (← links)
• Overexpression of protein kinase G using adenovirus inhibits cyclin E transcription and mesangial cell cycle. (Q43568354) (← links)
• Expression of atrial natriuretic peptide receptor-A antagonizes the mitogen-activated protein kinases (Erk2 and P38MAPK) in cultured human vascular smooth muscle cells (Q44040653) (← links)
• Physiological concentration of atrial natriuretic peptide induces endothelial regeneration in vitro (Q44265336) (← links)
• Constitutive activation and uncoupling of the atrial natriuretic peptide receptor by mutations at the dimer interface. Role of the dimer structure in signalling (Q44643908) (← links)
• Structure of the atrial natriuretic peptide receptor extracellular domain in the unbound and hormone-bound states by single-particle electron microscopy (Q46136567) (← links)
• Molecular therapeutic strategies for FGFR3 gene-related skeletal dysplasia (Q47612372) (← links)
• Dephosphorylation of the NPR2 guanylyl cyclase contributes to inhibition of bone growth by fibroblast growth factor (Q48188760) (← links)
• Overexpression of CNP in chondrocytes rescues achondroplasia through a MAPK-dependent pathway (Q75217684) (← links)
• Dephosphorylation is the mechanism of fibroblast growth factor inhibition of guanylyl cyclase-B (Q89112416) (← links)