Pages that link to "Q42597128"
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The following pages link to Effects of III-IV linker mutations on human heart Na channel inactivation gating (Q42597128):
Displaying 50 items.
- A novel nonsense variant in Nav1.5 cofactor MOG1 eliminates its sodium current increasing effect and may increase the risk of arrhythmias (Q24304891) (← links)
- Mutations in sodium channel β-subunit SCN3B are associated with early-onset lone atrial fibrillation (Q24305505) (← links)
- Cell membrane expression of cardiac sodium channel Na(v)1.5 is modulated by alpha-actinin-2 interaction (Q24322255) (← links)
- The role of late I Na in development of cardiac arrhythmias (Q26823149) (← links)
- Cardiac sodium channel Na(v)1.5 and interacting proteins: Physiology and pathophysiology (Q28258096) (← links)
- A single residue differentiates between human cardiac and skeletal muscle Na channel slow inactivation (Q28363700) (← links)
- A common human SCN5A polymorphism modifies expression of an arrhythmia causing mutation (Q31119456) (← links)
- The Na channel voltage sensor associated with inactivation is localized to the external charged residues of domain IV, S4. (Q33869265) (← links)
- Role of the C-terminal domain in inactivation of brain and cardiac sodium channels (Q33953193) (← links)
- Differential effects of sulfhydryl reagents on saxitoxin and tetrodotoxin block of voltage-dependent Na channels (Q34019030) (← links)
- Single-channel analysis of inactivation-defective rat skeletal muscle sodium channels containing the F1304Q mutation (Q34040527) (← links)
- Nonequilibrium Response Spectroscopy of Voltage-Sensitive Ion Channel Gating (Q34167076) (← links)
- Slow inactivation in human cardiac sodium channels (Q34168088) (← links)
- Effects of channel cytoplasmic regions on the activation mechanisms of cardiac versus skeletal muscle Na( ) channels (Q34171895) (← links)
- The outermost lysine in the S4 of domain III contributes little to the gating charge in sodium channels (Q34178080) (← links)
- Lidocaine partially depolarizes the S4 segment in domain IV of the sodium channel (Q34789375) (← links)
- A heterozygous deletion mutation in the cardiac sodium channel gene SCN5A with loss- and gain-of-function characteristics manifests as isolated conduction disease, without signs of Brugada or long QT syndrome (Q34808264) (← links)
- Computational biology in the study of cardiac ion channels and cell electrophysiology. (Q36015209) (← links)
- Sodium channel inactivation: molecular determinants and modulation (Q36267779) (← links)
- Important Role of Asparagines in Coupling the Pore and Votage-Sensor Domain in Voltage-Gated Sodium Channels (Q36363379) (← links)
- Molecular analysis of the putative inactivation particle in the inactivation gate of brain type IIA Na channels (Q36411866) (← links)
- Glutamine Substitution at Alanine1649 in the S4–S5 Cytoplasmic Loop of Domain 4 Removes the Voltage Sensitivity of Fast Inactivation in the Human Heart Sodium Channel (Q36412050) (← links)
- The role of the putative inactivation lid in sodium channel gating current immobilization (Q36412221) (← links)
- Molecular action of lidocaine on the voltage sensors of sodium channels (Q36412378) (← links)
- Interaction between the pore and a fast gate of the cardiac sodium channel (Q36421265) (← links)
- Anomalous Effect of Permeant Ion Concentration on Peak Open Probability of Cardiac Na Channels (Q36435845) (← links)
- Effect of Alkali Metal Cations on Slow Inactivation of Cardiac Na Channels (Q36435851) (← links)
- Molecular identity of the late sodium current in adult dog cardiomyocytes identified by Nav1.5 antisense inhibition (Q36844541) (← links)
- Pathophysiology of the cardiac late Na current and its potential as a drug target (Q37276416) (← links)
- Novel isoforms of the sodium channels Nav1.8 and Nav1.5 are produced by a conserved mechanism in mouse and rat. (Q37304434) (← links)
- Using lidocaine and benzocaine to link sodium channel molecular conformations to state-dependent antiarrhythmic drug affinity (Q37326589) (← links)
- Local anesthetics as effectors of allosteric gating. Lidocaine effects on inactivation-deficient rat skeletal muscle Na channels (Q37362215) (← links)
- The outer vestibule of the Na channel-toxin receptor and modulator of permeation as well as gating (Q37755990) (← links)
- Biology of cardiac sodium channel Nav1.5 expression (Q37936683) (← links)
- Development of recombinant cell line co-expressing mutated Nav1.5, Kir2.1, and hERG for the safety assay of drug candidates (Q39365157) (← links)
- Assessing use-dependent inhibition of the cardiac Na( /-) current (I(Na)) in the PatchXpress automated patch clamp (Q39684518) (← links)
- Outward stabilization of the S4 segments in domains III and IV enhances lidocaine block of sodium channels (Q40131479) (← links)
- Charge at the lidocaine binding site residue Phe-1759 affects permeation in human cardiac voltage-gated sodium channels (Q40158538) (← links)
- Block of wild-type and inactivation-deficient cardiac sodium channels IFM/QQQ stably expressed in mammalian cells (Q40175070) (← links)
- Partial expression defect for the SCN5A missense mutation G1406R depends on splice variant background Q1077 and rescue by mexiletine (Q40288640) (← links)
- Flecainide sensitivity of a Na channel long QT mutation shows an open-channel blocking mechanism for use-dependent block (Q40311694) (← links)
- Potent block of inactivation-deficient Na channels by n-3 polyunsaturated fatty acids (Q40365076) (← links)
- Tetrodotoxin-resistant Na channels in human neuroblastoma cells are encoded by new variants of Nav1.5/SCN5A. (Q40382553) (← links)
- Charge immobilization of the voltage sensor in domain IV is independent of sodium current inactivation (Q40485452) (← links)
- Enhancement of closed-state inactivation in long QT syndrome sodium channel mutation DeltaKPQ. (Q40710544) (← links)
- Modification of the cell based assay for brevetoxins using human cardiac voltage dependent sodium channels expressed in HEK-293 cells (Q40781916) (← links)
- Accelerated inactivation in a mutant Na( ) channel associated with idiopathic ventricular fibrillation (Q40836494) (← links)
- The [beta]2a subunit is a molecular groom for the Ca2 channel inactivation gate. (Q40906963) (← links)
- Recent advances in understanding the molecular mechanisms of the long QT syndrome (Q40962899) (← links)
- Gating of skeletal and cardiac muscle sodium channels in mammalian cells (Q40988455) (← links)