Pages that link to "Q42456739"
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The following pages link to SOCS2 induces neurite outgrowth by regulation of epidermal growth factor receptor activation (Q42456739):
Displaying 30 items.
- JAK/STAT3 pathway is involved in survival of neurons in response to insulin-like growth factor and negatively regulated by suppressor of cytokine signaling-3 (Q24306803) (← links)
- Thrombospondins as key regulators of synaptogenesis in the central nervous system (Q27024828) (← links)
- The role of suppressors of cytokine signalling in human neoplasms (Q27026783) (← links)
- Structure of the SOCS4-ElonginB/C Complex Reveals a Distinct SOCS Box Interface and the Molecular Basis for SOCS-Dependent EGFR Degradation (Q27649020) (← links)
- Transcriptional activation of the suppressor of cytokine signaling-3 (SOCS-3) gene via STAT3 is increased in F9 REX1 (ZFP-42) knockout teratocarcinoma stem cells relative to wild-type cells (Q30176405) (← links)
- Phenoxodiol protects against Cisplatin induced neurite toxicity in a PC-12 cell model (Q33292921) (← links)
- Suppressor of Cytokine Signalling-6 Promotes Neurite Outgrowth via JAK2/STAT5-Mediated Signalling Pathway, Involving Negative Feedback Inhibition (Q34085697) (← links)
- Gold nanoparticle-based beacon to detect STAT5b mRNA expression in living cells: a case optimized by bioinformatics screen. (Q35576282) (← links)
- Serendipitous SAD Solution for DMSO-Soaked SOCS2-ElonginC-ElonginB Crystals Using Covalently Incorporated Dimethylarsenic: Insights into Substrate Receptor Conformational Flexibility in Cullin RING Ligases. (Q35677393) (← links)
- Suppressor of Cytokine Signaling-2 (SOCS2) Regulates the Microglial Response and Improves Functional Outcome after Traumatic Brain Injury in Mice (Q35987373) (← links)
- Expression and function of erythropoietin receptors in tumors: implications for the use of erythropoiesis-stimulating agents in cancer patients (Q36854821) (← links)
- Deciphering proteins and their functions in the regenerating retina (Q37802881) (← links)
- Cellular functions regulated by phosphorylation of EGFR on Tyr845. (Q38109021) (← links)
- SOCS proteins in development and disease (Q38124037) (← links)
- SOCS proteins in regulation of receptor tyrosine kinase signaling (Q38202542) (← links)
- Regulation of neurotrophin receptor (Trk) signaling: suppressor of cytokine signaling 2 (SOCS2) is a new player. (Q38214593) (← links)
- Potential roles of suppressor of cytokine signaling in wound healing. (Q38733695) (← links)
- Metamorphosis and the regenerative capacity of spinal cord axons in Xenopus laevis (Q39819418) (← links)
- SOCS-3 induces myoblast differentiation. (Q40470317) (← links)
- SOCS2 Binds to and Regulates EphA2 through Multiple Mechanisms. (Q41543057) (← links)
- Is integration and survival of newborn neurons the bottleneck for effective neural repair by endogenous neural precursor cells? (Q41967459) (← links)
- ErbB1 epidermal growth factor receptor is a valid target for reducing the effects of multiple inhibitors of axonal regeneration. (Q43031168) (← links)
- Feedback regulation of RTK signaling in development. (Q46578462) (← links)
- Suppressor of Cytokine Signalling 2 (SOCS2) Regulates Numbers of Mature Newborn Adult Hippocampal Neurons and Their Dendritic Spine Maturation (Q48500310) (← links)
- Expression of SOCS2 mRNA and protein in the ischemic core and penumbra after transient focal cerebral ischemia in rats. (Q52146889) (← links)
- BC-Box Motif-Mediated Neuronal Differentiation of Somatic Stem Cells. (Q52699503) (← links)
- Reduced expression of SOCS2 and SOCS6 in hepatocellular carcinoma correlates with aggressive tumor progression and poor prognosis. (Q54269263) (← links)
- A novel role of suppressor of cytokine signaling-2 in the regulation of TrkA neurotrophin receptor biology. (Q54380745) (← links)
- Endocytic Adaptor Protein HIP1R Controls Intracellular Trafficking of Epidermal Growth Factor Receptor in Neuronal Dendritic Development (Q60046111) (← links)
- Structural insights into substrate recognition by the SOCS2 E3 ubiquitin ligase. (Q64935699) (← links)