Pages that link to "Q42010327"

The following pages link to Sequence composition of disordered regions fine-tunes protein half-life (Q42010327):

Displaying 42 items.

• Intracellular Dynamics of the Ubiquitin-Proteasome-System (Q26784629) (← links)
• The contribution of intrinsically disordered regions to protein function, cellular complexity, and human disease (Q28080017) (← links)
• An Inducible System for Rapid Degradation of Specific Cellular Proteins Using Proteasome Adaptors (Q28551149) (← links)
• Variation in auxin sensing guides AUX/IAA transcriptional repressor ubiquitylation and destruction. (Q30313118) (← links)
• Pri sORF peptides induce selective proteasome-mediated protein processing (Q34494721) (← links)
• Gates, Channels, and Switches: Elements of the Proteasome Machine (Q35862786) (← links)
• Substrate Ubiquitination Controls the Unfolding Ability of the Proteasome (Q36074198) (← links)
• Site-specific proteasome phosphorylation controls cell proliferation and tumorigenesis (Q36833345) (← links)
• Rapidly Translated Polypeptides Are Preferred Substrates for Cotranslational Protein Degradation. (Q36850107) (← links)
• The Proteasome Ubiquitin Receptor hRpn13 and Its Interacting Deubiquitinating Enzyme Uch37 Are Required for Proper Cell Cycle Progression (Q36884720) (← links)
• Ubiquitin-like domains can target to the proteasome but proteolysis requires a disordered region (Q36946185) (← links)
• Phosphorylation of the C-terminal tail of proteasome subunit α7 is required for binding of the proteasome quality control factor Ecm29. (Q37006187) (← links)
• Regulation of Proteasomal Degradation by Modulating Proteasomal Initiation Regions (Q37040613) (← links)
• Conserved Sequence Preferences Contribute to Substrate Recognition by the Proteasome (Q37078653) (← links)
• Mouse Mammary Tumor Virus Signal Peptide Uses a Novel p97-Dependent and Derlin-Independent Retrotranslocation Mechanism To Escape Proteasomal Degradation (Q37727169) (← links)
• How do disordered regions achieve comparable functions to structured domains? (Q38370771) (← links)
• Structural disorder and its role in proteasomal degradation (Q38557562) (← links)
• Reciprocal proteasome-mediated degradation of PIFs and HFR1 underlies photomorphogenic development in Arabidopsis. (Q38831482) (← links)
• Ubiquitin recognition by the proteasome (Q39077452) (← links)
• AAA-ATPases in Protein Degradation. (Q39414304) (← links)
• An assay for 26S proteasome activity based on fluorescence anisotropy measurements of dye-labeled protein substrates (Q41729079) (← links)
• Altered Co-Translational Processing Plays a Role in Huntington's Pathogenesis-A Hypothesis (Q41851500) (← links)
• Incomplete proteasomal degradation of green fluorescent proteins in the context of tandem fluorescent protein timers (Q42053360) (← links)
• A conserved degron containing an amphipathic helix regulates the cholesterol-mediated turnover of human squalene monooxygenase, a rate-limiting enzyme in cholesterol synthesis. (Q47103155) (← links)
• In Vivo Reporters for Protein Half-Life (Q47763557) (← links)
• Constraints and consequences of the emergence of amino acid repeats in eukaryotic proteins (Q47956508) (← links)
• Structure and Function of the 26S Proteasome. (Q52322457) (← links)
• Predicting proteome dynamics using gene expression data (Q58740967) (← links)
• Structural basis for substrate gripping and translocation by the ClpB AAA disaggregase. (Q64970320) (← links)
• The proteasome gets a grip on protein complexity (Q86861895) (← links)
• Proteins containing ubiquitin-like (Ubl) domains not only bind to 26S proteasomes but also induce their activation (Q89766695) (← links)
• An Extended Conformation for K48 Ubiquitin Chains Revealed by the hRpn2:Rpn13:K48-Diubiquitin Structure (Q90239219) (← links)
• Intrinsically disordered regions regulate both catalytic and non-catalytic activities of the MutLα mismatch repair complex (Q90429771) (← links)
• Proteasome Activation as a New Therapeutic Approach To Target Proteotoxic Disorders (Q92157489) (← links)
• Disordered region of cereblon is required for efficient degradation by proteolysis-targeting chimera (Q92229206) (← links)
• A repetitive acidic region contributes to the extremely rapid degradation of the cell-context essential protein TRIM52 (Q92325136) (← links)
• The Balancing Act of Intrinsically Disordered Proteins: Enabling Functional Diversity while Minimizing Promiscuity (Q92411725) (← links)
• The 26S Proteasome Utilizes a Kinetic Gateway to Prioritize Substrate Degradation (Q92726626) (← links)
• The proteasome 19S cap and its ubiquitin receptors provide a versatile recognition platform for substrates (Q92932075) (← links)
• A masked initiation region in retinoblastoma protein regulates its proteasomal degradation (Q93216460) (← links)
• Flexibility of intrinsically disordered degrons in AUX/IAA proteins reinforces auxin co-receptor assemblies (Q94569962) (← links)
• Design principles of gene evolution for niche adaptation through changes in protein-protein interaction networks (Q99710909) (← links)