Pages that link to "Q41830652"
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The following pages link to Tau in Alzheimer neurofibrillary tangles. N- and C-terminal regions are differentially associated with paired helical filaments and the location of a putative abnormal phosphorylation site (Q41830652):
Displaying 50 items.
- Tau Proteins and Neurofibrillary Degeneration (Q22242304) (← links)
- Tau cleavage and dephosphorylation in cerebellar granule neurons undergoing apoptosis (Q24312948) (← links)
- Clathrin adaptor CALM/PICALM is associated with neurofibrillary tangles and is cleaved in Alzheimer's brains (Q24338543) (← links)
- An Ultra-specific Avian Antibody to Phosphorylated Tau Protein Reveals a Unique Mechanism for Phosphoepitope Recognition (Q27675104) (← links)
- Viewing molecular mechanisms of ageing through a lens (Q28193216) (← links)
- Deletion of Irs2 reduces amyloid deposition and rescues behavioural deficits in APP transgenic mice (Q28505715) (← links)
- Propagation of tau pathology: hypotheses, discoveries, and yet unresolved questions from experimental and human brain studies (Q31024674) (← links)
- Tau pathology generated by overexpression of tau. (Q33793293) (← links)
- The switch of tau protein to an Alzheimer-like state includes the phosphorylation of two serine-proline motifs upstream of the microtubule binding region (Q33937669) (← links)
- Inside Alzheimer brain with CLARITY: senile plaques, neurofibrillary tangles and axons in 3-D. (Q34038201) (← links)
- A brief overview of amyloids and Alzheimer's disease. (Q34868191) (← links)
- The Department of Neuroscience at the Institute of Psychiatry (Q35227657) (← links)
- In vitro cultured neurons for molecular studies correlating apoptosis with events related to Alzheimer disease (Q35667785) (← links)
- Transgenic expression of the shortest human tau affects its compartmentalization and its phosphorylation as in the pretangle stage of Alzheimer's disease (Q35754598) (← links)
- Detection of a Cdc2-related kinase associated with Alzheimer paired helical filaments. (Q35797860) (← links)
- Immune electron microscopic characterization of monoclonal antibodies to Alzheimer neurofibrillary tangles (Q35831902) (← links)
- Heterogeneity of tau proteins in Alzheimer's disease. Evidence for increased expression of an isoform and preferential distribution of a phosphorylated isoform in neurites (Q35832261) (← links)
- Biochemical and anatomical redistribution of tau protein in Alzheimer's disease (Q35833178) (← links)
- Passive immunization targeting pathological phospho-tau protein in a mouse model reduces functional decline and clears tau aggregates from the brain (Q36005623) (← links)
- Biochemical classification of tauopathies by immunoblot, protein sequence and mass spectrometric analyses of sarkosyl-insoluble and trypsin-resistant tau (Q36465091) (← links)
- Alzheimer neurofibrillary tangles contain 2.1 nm filaments structurally identical to the microtubule-associated protein τ: a high-resolution transmission electron microscope study of tangles and senile plaque core amyloid (Q36695318) (← links)
- Ubiquitination and abnormal phosphorylation of paired helical filaments in Alzheimer's disease (Q36723776) (← links)
- Alzheimer disease hyperphosphorylated tau aggregates hydrophobically (Q36885874) (← links)
- Neurofibrillary tangles and β-amyloid deposits in Alzheimer's disease (Q37064063) (← links)
- Understanding the molecular basis of Alzheimer's disease using a Caenorhabditis elegans model system (Q37515518) (← links)
- Levels of kinesin light chain and dynein intermediate chain are reduced in the frontal cortex in Alzheimer's disease: implications for axoplasmic transport. (Q39441258) (← links)
- Molecular characterization of the minimal protease resistant tau unit of the Alzheimer's disease paired helical filament (Q40871850) (← links)
- Identification of 3- and 4-repeat tau isoforms within the PHF in Alzheimer's disease (Q41082322) (← links)
- Tau in Alzheimer's disease and Down's syndrome is insoluble and abnormally phosphorylated (Q41868884) (← links)
- A68 proteins in Alzheimer's disease are composed of several tau isoforms in a phosphorylated state which affects their electrophoretic mobilities (Q41879896) (← links)
- Accelerated human mutant tau aggregation by knocking out murine tau in a transgenic mouse model (Q41966030) (← links)
- Fibers of tau fragments, but not full length tau, exhibit a cross beta-structure: implications for the formation of paired helical filaments (Q42065690) (← links)
- Monoclonal antibodies with selective specificity for Alzheimer Tau are directed against phosphatase-sensitive epitopes (Q42141099) (← links)
- Prominent axonopathy in the brain and spinal cord of transgenic mice overexpressing four-repeat human tau protein (Q42795195) (← links)
- Immunocytochemistry of neurofibrillary tangles with antibodies to subregions of tau protein: identification of hidden and cleaved tau epitopes and a new phosphorylation site (Q44176630) (← links)
- How Does Hyperphopsphorylation Promote Tau Aggregation and Modulate Filament Structure and Stability? (Q46599058) (← links)
- Abnormal phosphorylation of tau proteins associated with bovine brain microtubules: activation by excess ATP and tyrosine dephosphorylation (Q48139616) (← links)
- Comparison of the phosphorylation of microtubule-associated protein tau by non-proline dependent protein kinases (Q48172695) (← links)
- Overexpression of alpha-internexin causes abnormal neurofilamentous accumulations and motor coordination deficits in transgenic mice (Q48246180) (← links)
- Implication of brain cdc2 and MAP2 kinases in the phosphorylation of tau protein in Alzheimer's disease (Q48444661) (← links)
- The complex relationship between soluble and insoluble tau in tauopathies revealed by efficient dephosphorylation and specific antibodies (Q48445377) (← links)
- Specific monoclonal antibodies against normal microtubule-associated protein-2 (MAP2) epitopes present in Alzheimer pathological structures do not recognize paired helical filaments (Q48590331) (← links)
- Phosphorylation of α-crystallin B in Alexander's disease brain (Q48612224) (← links)
- Protein kinase C and calcium/calmodulin-dependent protein kinase II phosphorylate three-repeat and four-repeat tau isoforms at different rates (Q48790449) (← links)
- Immunological characterization of epitopes on tau of Alzheimer's type and chemically modified tau. (Q48790501) (← links)
- Lithium chloride increases the production of amyloid-beta peptide independently from its inhibition of glycogen synthase kinase 3. (Q48805596) (← links)
- Generation of a monoclonal antibody to the carboxy-terminal domain of tau by immunization with the amino-terminal domain of the amyloid precursor protein (Q48829713) (← links)
- Non-proline-dependent protein kinases phosphorylate several sites found in tau from Alzheimer disease brain (Q49160674) (← links)
- Potentiation of GSK-3-catalyzed Alzheimer-like phosphorylation of human tau by cdk5. (Q52196473) (← links)
- Dephosphorylation of Alzheimer paired helical filaments by protein phosphatase-2A and -2B. (Q52209534) (← links)