Pages that link to "Q41786916"
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The following pages link to Adenovirus type 5 early region 1B 156R protein promotes cell transformation independently of repression of p53-stimulated transcription (Q41786916):
Displaying 22 items.
- SUMO modification of E1B-55K oncoprotein regulates isoform-specific binding to the tumour suppressor protein PML (Q24338995) (← links)
- Adenovirus type 5 early region 1B 55-kDa oncoprotein can promote cell transformation by a mechanism independent from blocking p53-activated transcription (Q28572354) (← links)
- Adenovirus type 5 early region 1B 55K oncoprotein-dependent degradation of cellular factor Daxx is required for efficient transformation of primary rodent cells (Q28574947) (← links)
- Regulatory, biosafety and safety challenges for novel cells as substrates for human vaccines (Q30413201) (← links)
- Aggresome formation by the adenoviral protein E1B55K is not conserved among adenovirus species and is not required for efficient degradation of nuclear substrates (Q36759991) (← links)
- In vivo potential effects of adenovirus type 5 E1A and E1B on lung carcinogenesis and lymphoproliferative inflammation. (Q36845679) (← links)
- The adenoviral E1B 55-kilodalton protein controls expression of immune response genes but not p53-dependent transcription (Q37145192) (← links)
- Adenovirus E1B 55-kilodalton protein: multiple roles in viral infection and cell transformation (Q37391112) (← links)
- Adenovirus degradation of cellular proteins. (Q37983134) (← links)
- Human amniocyte-derived cells are a promising cell host for adenoviral vector production under serum-free conditions (Q38878799) (← links)
- Functional cooperation between human adenovirus type 5 early region 4, open reading frame 6 protein, and cellular homeobox protein HoxB7. (Q39354514) (← links)
- The Human Adenovirus Type 5 E1B 55-Kilodalton Protein Is Phosphorylated by Protein Kinase CK2 (Q39424095) (← links)
- Proteasome-dependent degradation of Daxx by the viral E1B-55K protein in human adenovirus-infected cells. (Q39702317) (← links)
- Adenovirus E1B55K region is required to enhance cyclin E expression for efficient viral DNA replication. (Q40018751) (← links)
- Normal human cell proteins that interact with the adenovirus type 5 E1B 55kDa protein (Q40353337) (← links)
- Sp100A is a tumor suppressor that activates p53-dependent transcription and counteracts E1A/E1B-55K-mediated transformation (Q40947307) (← links)
- PML isoforms IV and V contribute to adenovirus-mediated oncogenic transformation by functionally inhibiting the tumor-suppressor p53. (Q41792379) (← links)
- The adenoviral oncogene E1A-13S interacts with a specific isoform of the tumor suppressor PML to enhance viral transcription (Q42022579) (← links)
- A 49-kilodalton isoform of the adenovirus type 5 early region 1B 55-kilodalton protein is sufficient to support virus replication (Q42090834) (← links)
- Intrinsic disorder in the common N-terminus of human adenovirus 5 E1B-55K and its related E1BN proteins indicated by studies on E1B-93R. (Q42613429) (← links)
- Cross-talk between phosphorylation and SUMOylation regulates transforming activities of an adenoviral oncoprotein. (Q53270092) (← links)
- Double-edged Role of PML Nuclear Bodies during Human Adenovirus Infection (Q104607567) (← links)