Pages that link to "Q41686271"
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The following pages link to Reversible inactivation of the cerebellar interpositus nucleus completely prevents acquisition of the classically conditioned eye-blink response (Q41686271):
Displaying 50 items.
- Modulation, Plasticity and Pathophysiology of the Parallel Fiber-Purkinje Cell Synapse (Q28069538) (← links)
- FoxP2 expression in avian vocal learners and non-learners (Q29618488) (← links)
- Cross-modal savings in the contralateral eyelid conditioned response. (Q30368535) (← links)
- Medial auditory thalamus is necessary for acquisition and retention of eyeblink conditioning to cochlear nucleus stimulation. (Q30384776) (← links)
- Neural circuitry and plasticity mechanisms underlying delay eyeblink conditioning (Q30445647) (← links)
- Ventral lateral geniculate input to the medial pons is necessary for visual eyeblink conditioning in rats (Q30477353) (← links)
- Medial auditory thalamic input to the lateral pontine nuclei is necessary for auditory eyeblink conditioning (Q30477905) (← links)
- Examination of bilateral eyeblink conditioning in rats (Q30478336) (← links)
- Stimulation of the lateral geniculate, superior colliculus, or visual cortex is sufficient for eyeblink conditioning in rats (Q30481744) (← links)
- Cerebellar inactivation impairs cross modal savings of eyeblink conditioning (Q30482262) (← links)
- The role of the cerebellar interpositus nucleus in short and long term memory for trace eyeblink conditioning (Q30483549) (← links)
- Imaging the spread of reversible brain inactivations using fluorescent muscimol (Q30488951) (← links)
- Inactivation of cerebellar output axons impairs acquisition of conditioned eyeblinks. (Q30499687) (← links)
- Developmental changes in eyeblink conditioning and neuronal activity in the pontine nuclei (Q30500871) (← links)
- Pretrial functional connectivity differentiates behavioral outcomes during trace eyeblink conditioning in the rabbit. (Q30727925) (← links)
- Genetic silencing of olivocerebellar synapses causes dystonia-like behaviour in mice (Q30844342) (← links)
- Learning induces a CDC2-related protein kinase, KKIAMRE. (Q31417665) (← links)
- Delay eyeblink classical conditioning is impaired in Fragile X syndrome (Q33623315) (← links)
- Intracerebellar conditioning--Brogden and Gantt revisited (Q33913225) (← links)
- Thalamic bursting in rats during different awake behavioral states. (Q33953173) (← links)
- Cerebellar cortical inhibition and classical eyeblink conditioning (Q34010292) (← links)
- Differential effects of cerebellar inactivation on eyeblink conditioned excitation and inhibition (Q34074848) (← links)
- Mechanisms of neuronal conditioning. (Q34143577) (← links)
- Task-dependent role of the cerebellum in motor learning (Q35599318) (← links)
- Neurogenetic dissection of conditioned behavior: evolution by analogy or homology? (Q35809380) (← links)
- The effects of ethanol on the developing cerebellum and eyeblink classical conditioning. (Q38639348) (← links)
- Assessing the role of inferior olivary sensory signaling in the expression of conditioned eyeblinks using a combined glutamate/GABAA receptor antagonist protocol (Q35951446) (← links)
- Conditioned eyeblink learning is formed and stored without cerebellar granule cell transmission (Q36082124) (← links)
- Characterizing cognitive aging of associative memory in animal models (Q36227694) (← links)
- Anatomical characterization of a rabbit cerebellar eyeblink premotor pathway using pseudorabies and identification of a local modulatory network in anterior interpositus. (Q36330878) (← links)
- Anatomical evidence for the involvement of medial cerebellar output from the interpositus nuclei in cognitive functions (Q36414951) (← links)
- Excitatory Cerebellar Nucleocortical Circuit Provides Internal Amplification during Associative Conditioning (Q36547769) (← links)
- Mechanisms of cerebellar gait ataxia (Q36762167) (← links)
- A trigeminal conditioned stimulus yields fast acquisition of cerebellum-dependent conditioned eyeblinks. (Q36865264) (← links)
- Metabolic mapping of the rat cerebellum during delay and trace eyeblink conditioning (Q36915309) (← links)
- Inactivation of median preoptic nucleus causes c-Fos expression in hypocretin- and serotonin-containing neurons in anesthetized rat (Q36992168) (← links)
- Neurotransmitter release during delay eyeblink classical conditioning: role of norepinephrine in consolidation and effect of age. (Q37365011) (← links)
- Involvement of the ipsilateral and contralateral cerebellum in the acquisition of unilateral classical eyeblink conditioning in guinea pigs (Q37728256) (← links)
- Mechanisms of cerebellar contributions to cognition in humans (Q39649018) (← links)
- Amygdala, deep cerebellar nuclei and red nucleus contribute to delay eyeblink conditioning in C57BL /6 mice (Q42873603) (← links)
- Selective changes in AMPA receptors in rabbit cerebellum following classical conditioning of the eyelid-nictitating membrane response (Q43780063) (← links)
- Transsynaptic tracing of conditioned eyeblink circuits in the mouse cerebellum (Q44249578) (← links)
- GABA Neurotransmission in the Cerebellar Interposed Nuclei: Involvement in Classically Conditioned Eyeblinks and Neuronal Activity (Q44628453) (← links)
- Inhibiting the expression of a classically conditioned behavior prevents its extinction. (Q44662049) (← links)
- Role of cerebellar interpositus nucleus in the genesis and control of reflex and conditioned eyelid responses. (Q45106034) (← links)
- Inhibition of cortisol production by metyrapone enhances trace, but not delay, eyeblink conditioning (Q46597426) (← links)
- Cerebellar dysfunction explains the extinction-like abolition of conditioned eyeblinks after NBQX injections in the inferior olive. (Q46825826) (← links)
- Inhibition of the amygdala central nucleus by stimulation of cerebellar output in rats: a putative mechanism for extinction of the conditioned fear response (Q47820246) (← links)
- Dynamic modulation of activity in cerebellar nuclei neurons during pavlovian eyeblink conditioning in mice. (Q48111685) (← links)
- On-line compensation for perturbations of a reaching movement is cerebellar dependent: support for the task dependency hypothesis (Q48127514) (← links)