Pages that link to "Q41634973"
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The following pages link to Neutral Lipid Storage Disease with Fatty Liver and Cholestasis (Q41634973):
Displaying 42 items.
- Adipose triglyceride lipase-mediated lipolysis of cellular fat stores is activated by CGI-58 and defective in Chanarin-Dorfman Syndrome (Q24329220) (← links)
- Mammalian patatin domain containing proteins: a family with diverse lipolytic activities involved in multiple biological functions (Q24653003) (← links)
- Dissecting adipose tissue lipolysis: molecular regulation and implications for metabolic disease (Q26824349) (← links)
- Phospholipase A2 enzymes: physical structure, biological function, disease implication, chemical inhibition, and therapeutic intervention (Q26997377) (← links)
- Identification of mouse Prp19p as a lipid droplet-associated protein and its possible involvement in the biogenesis of lipid droplets (Q28592348) (← links)
- Neutral Lipid Storage Diseases: clinical/genetic features and natural history in a large cohort of Italian patients (Q33667982) (← links)
- CGI-58/ABHD5 is a coenzyme A-dependent lysophosphatidic acid acyltransferase (Q33741180) (← links)
- Comparative gene identification 58/α/β hydrolase domain 5 lacks lysophosphatidic acid acyltransferase activity (Q33945126) (← links)
- Lipid droplets: size matters (Q34028156) (← links)
- CGI-58 knockdown in mice causes hepatic steatosis but prevents diet-induced obesity and glucose intolerance (Q34188075) (← links)
- Clinical and genetic characterization of Chanarin-Dorfman syndrome patients: first report of large deletions in the ABHD5 gene. (Q34482020) (← links)
- Lipolysis – A highly regulated multi-enzyme complex mediates the catabolism of cellular fat stores (Q34543395) (← links)
- Not just fat: the structure and function of the lipid droplet (Q34578014) (← links)
- State of the art in muscle lipid diseases (Q34580810) (← links)
- Loss of abhd5 promotes colorectal tumor development and progression by inducing aerobic glycolysis and epithelial-mesenchymal transition (Q34717352) (← links)
- CGI-58/ABHD5 is phosphorylated on Ser239 by protein kinase A: control of subcellular localization (Q34757325) (← links)
- Intestinal Cgi-58 deficiency reduces postprandial lipid absorption (Q35117538) (← links)
- Adipose-selective overexpression of ABHD5/CGI-58 does not increase lipolysis or protect against diet-induced obesity (Q35380219) (← links)
- Muscle-specific deletion of comparative gene identification-58 (CGI-58) causes muscle steatosis but improves insulin sensitivity in male mice (Q35436264) (← links)
- ATGL-mediated fat catabolism regulates cardiac mitochondrial function via PPAR-α and PGC-1. (Q35630386) (← links)
- Effects of rosiglitazone and high fat diet on lipase/esterase expression in adipose tissue (Q35914800) (← links)
- Adipose triglyceride lipase acts on neutrophil lipid droplets to regulate substrate availability for lipid mediator synthesis (Q36125641) (← links)
- Distinct roles for alpha-beta hydrolase domain 5 (ABHD5/CGI-58) and adipose triglyceride lipase (ATGL/PNPLA2) in lipid metabolism and signaling (Q36382868) (← links)
- Distinct mechanisms regulate ATGL-mediated adipocyte lipolysis by lipid droplet coat proteins (Q36533395) (← links)
- iPS cell modeling of cardiometabolic diseases (Q36539750) (← links)
- Mammalian alpha beta hydrolase domain (ABHD) proteins: Lipid metabolizing enzymes at the interface of cell signaling and energy metabolism (Q36612260) (← links)
- Biochemistry and pathophysiology of intravascular and intracellular lipolysis (Q36708030) (← links)
- Cardiac-specific overexpression of perilipin 5 provokes severe cardiac steatosis via the formation of a lipolytic barrier (Q36709863) (← links)
- Deficiency of liver Comparative Gene Identification-58 causes steatohepatitis and fibrosis in mice (Q37002642) (← links)
- Neutral lipid storage disease: genetic disorders caused by mutations in adipose triglyceride lipase/PNPLA2 or CGI-58/ABHD5. (Q37463574) (← links)
- Lipid storage myopathy (Q37806661) (← links)
- Fat in the skin: Triacylglycerol metabolism in keratinocytes and its role in the development of neutral lipid storage disease. (Q37892924) (← links)
- Inborn errors of cytoplasmic triglyceride metabolism. (Q38258625) (← links)
- Growth retardation, impaired triacylglycerol catabolism, hepatic steatosis, and lethal skin barrier defect in mice lacking comparative gene identification-58 (CGI-58). (Q39762596) (← links)
- CGI-58 facilitates the mobilization of cytoplasmic triglyceride for lipoprotein secretion in hepatoma cells (Q40100342) (← links)
- ABHD5/CGI-58 facilitates the assembly and secretion of apolipoprotein B lipoproteins by McA RH7777 rat hepatoma cells (Q41397987) (← links)
- Molecular characterization of human ABHD2 as TAG lipase and ester hydrolase (Q42418890) (← links)
- Critical roles for α/β hydrolase domain 5 (ABHD5)/comparative gene identification-58 (CGI-58) at the lipid droplet interface and beyond (Q47925533) (← links)
- Renal involvement as a rare complication of Dorfman-Chanarin syndrome: a case report (Q51953591) (← links)
- Neutral Lipid Storage Diseases as Cellular Model to Study Lipid Droplet Function (Q64118342) (← links)
- Triglyceride deposit cardiomyovasculopathy: a rare cardiovascular disorder. (Q65002567) (← links)
- A cause of fatty liver: neutral lipid storage disease with ichthyosis--electron microscopic findings (Q73208337) (← links)