Pages that link to "Q41515043"
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The following pages link to Modulation of cell growth, p34cdc2 and cyclin A levels by SV-40 large T antigen. (Q41515043):
Displaying 13 items.
- Multiple mechanisms for the phosphorylation of C-terminal regulatory sites in rabbit muscle glycogen synthase expressed in COS cells (Q24534141) (← links)
- Disappearance of cyclin A correlates with permanent withdrawal of cardiomyocytes from the cell cycle in human and rat hearts (Q24629391) (← links)
- Induction of cyclin A gene expression by homocysteine in vascular smooth muscle cells (Q28378757) (← links)
- Transactivation of murine cyclin A by polyomavirus large and small T antigens. (Q33843350) (← links)
- Induction of Duplication Reversion in Human Fibroblasts, by Wild-Type and Mutated SV40 T Antigen, Covaries With the Ability to Induce Host DNA Synthesis (Q33970521) (← links)
- Simian virus 40 large T antigen and two independent T-antigen segments sensitize cells to apoptosis following genotoxic damage (Q35000664) (← links)
- Activation of cyclin A-dependent protein kinases during apoptosis (Q35195381) (← links)
- Cell killing by the novel imidazoacridinone antineoplastic agent, C-1311, is inhibited at high concentrations coincident with dose-differentiated cell cycle perturbation (Q36136247) (← links)
- Binding of polyomavirus small T antigen to protein phosphatase 2A is required for elimination of p27 and support of S-phase induction in concert with large T antigen (Q39596484) (← links)
- The inhibition of cultured myoblast differentiation by the simian virus 40 large T antigen occurs after myogenin expression and Rb up-regulation and is not exerted by transformation-competent cytoplasmic mutants. (Q39872360) (← links)
- Mechanisms of transcriptional regulation of cellular genes by SV40 large T- and small T-antigens (Q41677060) (← links)
- BK virus as a cofactor in the etiology of prostate cancer in its early stages (Q43064525) (← links)
- Recombination and its roles in DNA repair, cellular immortalization and cancer (Q56902268) (← links)