Pages that link to "Q41203275"
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The following pages link to Presence of mutations in all three ras genes in human thyroid tumors (Q41203275):
Displaying 50 items.
- Evidence for a telomere-independent "clock" limiting RAS oncogene-driven proliferation of human thyroid epithelial cells (Q24552563) (← links)
- UbcH10 overexpression may represent a marker of anaplastic thyroid carcinomas (Q24651210) (← links)
- Selective use of sorafenib in the treatment of thyroid cancer (Q26752891) (← links)
- RAS mutations in thyroid cancer (Q27026163) (← links)
- Molecular differential diagnosis of follicular thyroid carcinoma and adenoma based on gene expression profiling by using formalin-fixed paraffin-embedded tissues. (Q31141133) (← links)
- Thyroid cancer: current molecular perspectives (Q33760910) (← links)
- Lessons from mouse models of thyroid cancer (Q33819911) (← links)
- Gene p53 mutations are restricted to poorly differentiated and undifferentiated carcinomas of the thyroid gland (Q33896893) (← links)
- Molecular genotyping of papillary thyroid carcinoma follicular variant according to its histological subtypes (encapsulated vs infiltrative) reveals distinct BRAF and RAS mutation patterns (Q34119294) (← links)
- Molecular approaches to thyroid cancer diagnosis (Q34160828) (← links)
- From amplification to gene in thyroid cancer: a high-resolution mapped bacterial-artificial-chromosome resource for cancer chromosome aberrations guides gene discovery after comparative genome hybridization (Q34386622) (← links)
- Expression patterns of cellular growth-controlling genes in non-medullary thyroid cancer: basic aspects (Q34438847) (← links)
- Molecular pathobiology of thyroid neoplasms (Q35094749) (← links)
- Genetic and biological subgroups of low-stage follicular thyroid cancer (Q35749248) (← links)
- High prevalence of mutations of the p53 gene in poorly differentiated human thyroid carcinomas (Q35823973) (← links)
- Synthesis and evaluation of antitumor activity of novel N-acyllavendamycin analogues and quinoline-5,8-diones (Q35857145) (← links)
- Genetic alterations involved in the transition from well-differentiated to poorly differentiated and anaplastic thyroid carcinomas (Q36025032) (← links)
- Thyroid carcinoma-associated genetic mutations also occur in thyroid lymphomas (Q36209975) (← links)
- Absence of RAS and p53 mutations in thyroid carcinomas of children after Chernobyl in contrast to adult thyroid tumours (Q36292212) (← links)
- Modeling thyroid cancer in the mouse (Q36297044) (← links)
- New treatment in advanced thyroid cancer (Q36361115) (← links)
- Thyrotropin-induced mitogenesis is Ras dependent but appears to bypass the Raf-dependent cytoplasmic kinase cascade (Q36549662) (← links)
- Search for NTRK1 proto-oncogene rearrangements in human thyroid tumours originated after therapeutic radiation (Q36621089) (← links)
- Gene rearrangement and Chernobyl related thyroid cancers (Q36621093) (← links)
- Association of RET codon 691 polymorphism in radiation-induced human thyroid tumours with C-cell hyperplasia in peritumoural tissue (Q36644648) (← links)
- Inhibition of thyrotropin-stimulated DNA synthesis by microinjection of inhibitors of cellular Ras and cyclic AMP-dependent protein kinase (Q36693217) (← links)
- De novo HRAS and KRAS mutations in two siblings with short stature and neuro-cardio-facio-cutaneous features (Q37004691) (← links)
- Genomic Profiling of Thyroid Cancer Reveals a Role for Thyroglobulin in Metastasis (Q37005057) (← links)
- Molecular biology of thyroid cancer initiation. (Q37022649) (← links)
- Thyroid carcinoma: molecular pathways and therapeutic targets (Q37147768) (← links)
- Molecular analysis of thyroid tumors (Q37860130) (← links)
- Molecular genetics and diagnosis of thyroid cancer (Q37924088) (← links)
- The quest for diagnostic molecular markers for thyroid nodules with indeterminate or suspicious cytology (Q37996353) (← links)
- Genetics and epigenetics of sporadic thyroid cancer (Q38128097) (← links)
- Deregulation of microRNA expression in thyroid neoplasias (Q38163815) (← links)
- Ras mutations are uncommon in sporadic thyroid cancer in children and young adults (Q38466394) (← links)
- Induction of cell death by stimulation of protein kinase C in human epithelial cells expressing a mutant ras oncogene: a potential therapeutic target (Q39708208) (← links)
- Thyroid carcinoma: clinical, pathologic correlations (Q40492981) (← links)
- Oncogenes and anti-oncogenes in human epithelial thyroid tumors (Q40698212) (← links)
- Ret oncogene activation in human thyroid neoplasms is restricted to the papillary cancer subtype. (Q41019851) (← links)
- Growth inhibition of a colonic adenocarcinoma cell line (HT29) by T cells specific for mutant p21 ras. (Q41488786) (← links)
- Expression of a potential metastasis suppressor gene (nm23) in thyroid neoplasms. (Q41530729) (← links)
- ROS-generating NADPH oxidase NOX4 is a critical mediator in oncogenic H-Ras-induced DNA damage and subsequent senescence (Q41821426) (← links)
- Oncogenic RAS-induced senescence in human primary thyrocytes: molecular effectors and inflammatory secretome involved. (Q42245179) (← links)
- DNA methylation state of the galectin-3 gene represents a potential new marker of thyroid malignancy. (Q42553510) (← links)
- Does autocrine growth factor secretion form part of a mechanism which paradoxically protects against tumour development? (Q42590657) (← links)
- The status of CDKN2A alpha (p16INK4A) and beta (p14ARF) transcripts in thyroid tumour progression (Q43199707) (← links)
- Ki-ras mutational analysis in rat follicular-cell proliferative lesions of the thyroid gland induced by radioactive iodine and potassium perchlorate (Q44821229) (← links)
- Lack of mutational events of RAS genes in sporadic thyroid cancer but high risk associated with HRAS T81C single nucleotide polymorphism (case-control study). (Q45106309) (← links)
- Analysis of RET proto-oncogene abnormalities in patients with MEN 2A, MEN 2B, familial or sporadic medullary thyroid carcinoma (Q47706781) (← links)