Pages that link to "Q41082397"

The following pages link to MLL5 Orchestrates a Cancer Self-Renewal State by Repressing the Histone Variant H3.3 and Globally Reorganizing Chromatin (Q41082397):

Displaying 33 items.

• DNA repair mechanisms and their clinical impact in glioblastoma (Q26740256) (← links)
• MLL5 maintains spindle bipolarity by preventing aberrant cytosolic aggregation of PLK1 (Q27310288) (← links)
• PTEN regulates glioblastoma oncogenesis through chromatin-associated complexes of DAXX and histone H3.3. (Q33705644) (← links)
• Epigenome Maintenance in Response to DNA Damage (Q33811644) (← links)
• Regulation of the JMJD3 (KDM6B) histone demethylase in glioblastoma stem cells by STAT3 (Q36338775) (← links)
• The Histone Variant H3.3 Is Enriched at Drosophila Amplicon Origins but Does Not Mark Them for Activation. (Q36958166) (← links)
• Epigenetic regulation of nitric oxide synthase 2, inducible (Nos2) by NLRC4 inflammasomes involves PARP1 cleavage (Q37620549) (← links)
• Advances in the molecular genetics of gliomas - implications for classification and therapy (Q38777058) (← links)
• Potential coordination role between O-GlcNAcylation and epigenetics (Q38794897) (← links)
• Epigenetic modification in chromatin machinery and its deregulation in pediatric brain tumors: Insight into epigenetic therapies (Q39069889) (← links)
• Variants of core histones and their roles in cell fate decisions, development and cancer. (Q39112515) (← links)
• MLL5 (KMT2E): structure, function, and clinical relevance (Q39131004) (← links)
• Therapeutic Targeting of Histone Modifications in Adult and Pediatric High-Grade Glioma (Q39236500) (← links)
• Epigenetics and Cancer Stem Cells: Unleashing, Hijacking, and Restricting Cellular Plasticity. (Q39441283) (← links)
• Epigenomic Consequences of Coding and Noncoding Driver Mutations (Q39456766) (← links)
• Reprogramming metabolism by histone methyltransferase NSD2 drives endocrine resistance via coordinated activation of pentose phosphate pathway enzymes (Q40761448) (← links)
• Global chromatin architecture defines functional cancer hierarchies (Q41453839) (← links)
• Small molecule epigenetic screen identifies novel EZH2 and HDAC inhibitors that target glioblastoma brain tumor-initiating cells. (Q42321107) (← links)
• H3.3K27M Cooperates with Trp53 Loss and PDGFRA Gain in Mouse Embryonic Neural Progenitor Cells to Induce Invasive High-Grade Gliomas. (Q47436402) (← links)
• Fate mapping of human glioblastoma reveals an invariant stem cell hierarchy. (Q48134470) (← links)
• MLL5 suppresses antiviral innate immune response by facilitating STUB1-mediated RIG-I degradation. (Q52624830) (← links)
• Tumorigenic Cell Reprogramming and Cancer Plasticity: Interplay between Signaling, Microenvironment, and Epigenetics. (Q55251323) (← links)
• SET-9 and SET-26 are H3K4me3 readers and play critical roles in germline development and longevity. (Q55437768) (← links)
• Analysis of chromatin accessibility uncovers TEAD1 as a regulator of migration in human glioblastoma (Q57059935) (← links)
• Epigenetic Targeting of Glioblastoma (Q58554073) (← links)
• Molecular Pathogenesis and Emerging Treatment for Glioblastoma (Q90508484) (← links)
• Matrix stiffness induces a tumorigenic phenotype in mammary epithelium through changes in chromatin accessibility (Q91723803) (← links)
• Targeting epigenetic regulators for cancer therapy: mechanisms and advances in clinical trials (Q92221539) (← links)
• Glioblastoma stem cells: lessons from the tumor hierarchy in a lethal cancer (Q92500797) (← links)
• Off-target effect of the BMI1 inhibitor PTC596 drives epithelial-mesenchymal transition in glioblastoma multiforme (Q92622543) (← links)
• Target identification reveals lanosterol synthase as a vulnerability in glioma (Q92686739) (← links)
• High-resolution structural genomics reveals new therapeutic vulnerabilities in glioblastoma (Q93087327) (← links)
• Reciprocal H3.3 gene editing identifies K27M and G34R mechanisms in pediatric glioma including NOTCH signaling (Q97540968) (← links)