Pages that link to "Q40804151"
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The following pages link to Activation of BRCA1/BRCA2-associated helicase BACH1 is required for timely progression through S phase (Q40804151):
Displaying 50 items.
- Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome (Q24337192) (← links)
- How the fanconi anemia pathway guards the genome (Q24634556) (← links)
- The Fanconi anemia pathway and DNA interstrand cross-link repair (Q26851561) (← links)
- Mutations in BRIP1 confer high risk of ovarian cancer (Q28249514) (← links)
- Cellular and molecular consequences of defective Fanconi anemia proteins in replication-coupled DNA repair: mechanistic insights (Q28252640) (← links)
- Mitotic homologous recombination maintains genomic stability and suppresses tumorigenesis (Q28274000) (← links)
- FancJ (Brip1) loss-of-function allele results in spermatogonial cell depletion during embryogenesis and altered processing of crossover sites during meiotic prophase I in mice (Q28513158) (← links)
- FANCJ helicase uniquely senses oxidative base damage in either strand of duplex DNA and is stimulated by replication protein A to unwind the damaged DNA substrate in a strand-specific manner (Q30863349) (← links)
- Overexpression of RAD51 suppresses recombination defects: a possible mechanism to reverse genomic instability (Q30922231) (← links)
- BACH1/FANCJ acts with TopBP1 and participates early in DNA replication checkpoint control (Q33531604) (← links)
- Mechanistic and biological aspects of helicase action on damaged DNA. (Q33614603) (← links)
- Insight into the roles of helicase motif Ia by characterizing Fanconi anemia group J protein (FANCJ) patient mutations (Q33675765) (← links)
- Mutations in the BRCT binding site of BRCA1 result in hyper-recombination (Q33930358) (← links)
- Novel function of the Fanconi anemia group J or RECQ1 helicase to disrupt protein-DNA complexes in a replication protein A-stimulated manner (Q33931112) (← links)
- G‐quadruplex nucleic acids and human disease (Q34073316) (← links)
- BRCA1 and BRCA2: breast/ovarian cancer susceptibility gene products and participants in DNA double-strand break repair (Q34110729) (← links)
- MicroRNA profiling of follicular lymphoma identifies microRNAs related to cell proliferation and tumor response (Q34234677) (← links)
- BRCA1-directed, enhanced and aberrant homologous recombination: mechanism and potential treatment strategies (Q34252163) (← links)
- Poly (ADP-ribose) polymerase as a novel therapeutic target in cancer (Q34308770) (← links)
- Fanconi anemia group J mutation abolishes its DNA repair function by uncoupling DNA translocation from helicase activity or disruption of protein-DNA complexes. (Q34310865) (← links)
- Genome-wide analysis reveals a role for BRCA1 and PALB2 in transcriptional co-activation (Q34324578) (← links)
- Molecular and cellular functions of the FANCJ DNA helicase defective in cancer and in Fanconi anemia (Q34377302) (← links)
- FANCB is essential in the male germline and regulates H3K9 methylation on the sex chromosomes during meiosis (Q34482867) (← links)
- Mutation of the BRCA1 SQ-cluster results in aberrant mitosis, reduced homologous recombination, and a compensatory increase in non-homologous end joining (Q34491545) (← links)
- Helicase-inactivating mutations as a basis for dominant negative phenotypes (Q34659857) (← links)
- Hereditary Breast Cancer and the BRCA1-Associated FANCJ/BACH1/BRIP1 (Q35029638) (← links)
- A distinct triplex DNA unwinding activity of ChlR1 helicase (Q35104037) (← links)
- Oncogenic RAS regulates BRIP1 expression to induce dissociation of BRCA1 from chromatin, inhibit DNA repair, and promote senescence. (Q35621531) (← links)
- BRCA1 tumor suppressor network: focusing on its tail (Q35861690) (← links)
- DNA helicases involved in DNA repair and their roles in cancer (Q35961508) (← links)
- DNA helicase and helicase-nuclease enzymes with a conserved iron-sulfur cluster (Q36044809) (← links)
- The Q motif of Fanconi anemia group J protein (FANCJ) DNA helicase regulates its dimerization, DNA binding, and DNA repair function (Q36052367) (← links)
- BRCA1 regulation of epidermal growth factor receptor (EGFR) expression in human breast cancer cells involves microRNA-146a and is critical for its tumor suppressor function (Q36539643) (← links)
- FANCJ helicase defective in Fanconia anemia and breast cancer unwinds G-quadruplex DNA to defend genomic stability (Q36710804) (← links)
- FANCJ couples replication past natural fork barriers with maintenance of chromatin structure (Q36732772) (← links)
- BRCA1 in the DNA damage response and at telomeres (Q36945848) (← links)
- FANCJ at the FORK. (Q36952290) (← links)
- Fanconi anemia proteins, DNA interstrand crosslink repair pathways, and cancer therapy (Q37069628) (← links)
- FANCJ uses its motor ATPase to destabilize protein-DNA complexes, unwind triplexes, and inhibit RAD51 strand exchange (Q37134364) (← links)
- Welcome the family of FANCJ-like helicases to the block of genome stability maintenance proteins (Q37144651) (← links)
- Specialization among iron-sulfur cluster helicases to resolve G-quadruplex DNA structures that threaten genomic stability (Q37201282) (← links)
- FANCM-FAAP24 and FANCJ: FA proteins that metabolize DNA. (Q37271577) (← links)
- PALB2 links BRCA1 and BRCA2 in the DNA-damage response (Q37361223) (← links)
- Modification of ovarian cancer risk by BRCA1/2-interacting genes in a multicenter cohort of BRCA1/2 mutation carriers (Q37362211) (← links)
- FANCJ helicase operates in the Fanconi Anemia DNA repair pathway and the response to replicational stress (Q37391123) (← links)
- Mammalian Rif1 contributes to replication stress survival and homology-directed repair (Q37425607) (← links)
- Absence of truncating BRIP1 mutations in chromosome 17q-linked hereditary prostate cancer families (Q37481918) (← links)
- BRCA1 and its toolbox for the maintenance of genome integrity (Q37507563) (← links)
- Assessing the link between BACH1/FANCJ and MLH1 in DNA crosslink repair (Q37775585) (← links)
- Recognition, signaling, and repair of DNA double-strand breaks produced by ionizing radiation in mammalian cells: The molecular choreography (Q38022419) (← links)