Pages that link to "Q40598692"
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The following pages link to Soluble LDL-R are formed by cell surface cleavage in response to phorbol esters (Q40598692):
Displaying 11 items.
- The generation and function of soluble apoE receptors in the CNS (Q21202840) (← links)
- The cellular trafficking of the secretory proprotein convertase PCSK9 and its dependence on the LDLR (Q24303965) (← links)
- Adenoviral-mediated expression of Pcsk9 in mice results in a low-density lipoprotein receptor knockout phenotype (Q24563948) (← links)
- Apolipoprotein E isoform-specific effects on lipoprotein receptor processing (Q33112703) (← links)
- Overexpression of PCSK9 accelerates the degradation of the LDLR in a post-endoplasmic reticulum compartment (Q33767310) (← links)
- Expression and regulation of a low‐density lipoprotein receptor exon 12 splice variant (Q34385583) (← links)
- Rapid temporal dynamics of transcription, protein synthesis, and secretion during macrophage activation (Q37623632) (← links)
- Mutations affecting the transmembrane domain of the LDL receptor: impact of charged residues on the membrane insertion. (Q38882139) (← links)
- Mutation G805R in the transmembrane domain of the LDL receptor gene causes familial hypercholesterolemia by inducing ectodomain cleavage of the LDL receptor in the endoplasmic reticulum (Q40419629) (← links)
- Mutation p.L799R in the LDLR, which affects the transmembrane domain of the LDLR, prevents membrane insertion and causes secretion of the mutant LDLR. (Q50950950) (← links)
- Association between serum soluble low-density lipoprotein receptor levels and metabolic factors in healthy Japanese individuals. (Q53059801) (← links)