Pages that link to "Q40250104"
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The following pages link to The role of tissue transglutaminase in 1-methyl-4-phenylpyridinium (MPP )-induced toxicity in differentiated human SH-SY5Y neuroblastoma cells (Q40250104):
Displaying 13 items.
- Presence of tissue transglutaminase in granular endoplasmic reticulum is characteristic of melanized neurons in Parkinson's disease brain (Q28291271) (← links)
- Cholesterol supports the retinoic acid-induced synaptic vesicle formation in differentiating human SH-SY5Y neuroblastoma cells (Q28304530) (← links)
- Type 2 transglutaminase in Huntington’s disease: a double‐edged sword with clinical potential (Q34775357) (← links)
- Tissue transglutaminase expression and activity in normal and glaucomatous human trabecular meshwork cells and tissues (Q37111745) (← links)
- Transglutaminase-2 Is Involved in All-Trans Retinoic Acid-Induced Invasion and Matrix Metalloproteinases Expression of SH-SY5Y Neuroblastoma Cells via NF-κB Pathway (Q37224569) (← links)
- Potential of transglutaminase 2 as a therapeutic target. (Q37776508) (← links)
- Amphotericin B Increases Transglutaminase 2 Expression Associated with Upregulation of Endocytotic Activity in Mouse Microglial Cell Line BV-2. (Q38715410) (← links)
- Sphingosine kinase 1 and sphingosine-1-phosphate in oxidative stress evoked by 1-methyl-4-phenylpyridinium (MPP ) in human dopaminergic neuronal cells (Q39036572) (← links)
- Transglutaminase 2 interaction with small heat shock proteins mediate cell survival upon excitotoxic stress (Q39470547) (← links)
- Blockade of enzyme activity inhibits tissue transglutaminase-mediated transamidation of α-synuclein in a cellular model of Parkinson's disease (Q39570119) (← links)
- Differential Effects of Methyl-4-Phenylpyridinium Ion, Rotenone, and Paraquat on Differentiated SH-SY5Y Cells (Q40715534) (← links)
- New Insights into Transglutaminase 2 and Links to Neurodegenerative Diseases (Q47350887) (← links)
- An extracellular transglutaminase is required for apple pollen tube growth. (Q50554531) (← links)