Pages that link to "Q40161773"
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The following pages link to Carvedilol inhibits mitochondrial complex I and induces resistance to H2O2-mediated oxidative insult in H9C2 myocardial cells (Q40161773):
Displaying 28 items.
- Protective role of deoxyschizandrin and schisantherin A against myocardial ischemia-reperfusion injury in rats (Q21559663) (← links)
- MTO1-deficient mouse model mirrors the human phenotype showing complex I defect and cardiomyopathy (Q27320993) (← links)
- The nuclear genes Mtfr1 and Dufd1 regulate mitochondrial dynamic and cellular respiration (Q28863043) (← links)
- Chlorogenic acid analogues from Gynura nepalensis protect H9c2 cardiomyoblasts against H2O2-induced apoptosis (Q32186803) (← links)
- Sodium tanshinone IIA sulfonate protects rat myocardium against ischemia-reperfusion injury via activation of PI3K/Akt/FOXO3A/Bim pathway (Q33551875) (← links)
- Exome sequencing identifies ACAD9 mutations as a cause of complex I deficiency (Q34148592) (← links)
- Blockade of electron transport during ischemia preserves bcl-2 and inhibits opening of the mitochondrial permeability transition pore (Q34792021) (← links)
- ß-blocker timolol prevents arrhythmogenic Ca²⁺ release and normalizes Ca²⁺ and Zn²⁺ dyshomeostasis in hyperglycemic rat heart (Q34907705) (← links)
- Lymphocyte-specific protein tyrosine kinase (Lck) interacts with CR6-interacting factor 1 (CRIF1) in mitochondria to repress oxidative phosphorylation (Q35713802) (← links)
- Cosegregation of novel mitochondrial 16S rRNA gene mutations with the age-associated T414G variant in human cybrids (Q36935463) (← links)
- Drugs interfering with mitochondrial disorders (Q37617357) (← links)
- Impaired cardiac and skeletal muscle bioenergetics in children, adolescents, and young adults with Barth syndrome. (Q37642420) (← links)
- Mitochondria play a central role in nonischemic cardiomyocyte necrosis: common to acute and chronic stressor states (Q37983499) (← links)
- Mitochondrial toxicity of cardiac drugs and its relevance to mitochondrial disorders. (Q38261496) (← links)
- β-Sitosterol enhances cellular glutathione redox cycling by reactive oxygen species generated from mitochondrial respiration: protection against oxidant injury in H9c2 cells and rat hearts (Q39053718) (← links)
- The extent of neurodegeneration and neuroprotection in two chemical in vitro models related to Parkinson's disease is critically dependent on cell culture conditions (Q39212503) (← links)
- Carvedilol and antioxidant proteins in a type I diabetes animal model (Q39224330) (← links)
- Herba Cistanches stimulates cellular glutathione redox cycling by reactive oxygen species generated from mitochondrial respiration in H9c2 cardiomyocytes (Q39266121) (← links)
- Evidence for ectopic aerobic ATP production on C6 glioma cell plasma membrane. (Q39630943) (← links)
- Low-dose carvedilol protects against acute septic renal injury in rats during the early and late phases (Q41269792) (← links)
- Profound cardioprotection with timolol in a female rat model of aging-related altered left ventricular function. (Q42487326) (← links)
- Selection and Characterization of Palmitic Acid Responsive Patients with an OXPHOS Complex I Defect (Q43106711) (← links)
- Beta-blocker timolol alleviates hyperglycemia-induced cardiac damage via inhibition of endoplasmic reticulum stress. (Q45338593) (← links)
- Functional expression of oxidative phosphorylation proteins in the rod outer segment disc (Q48208419) (← links)
- Novel SLC25A32 mutation in a patient with a severe neuromuscular phenotype (Q50441570) (← links)
- A clinical, genetic, and biochemical characterization of SPG7 mutations in a large cohort of patients with hereditary spastic paraplegia (Q57085813) (← links)
- Cellular rescue-assay aids verification of causative DNA-variants in mitochondrial complex I deficiency (Q57251796) (← links)
- Riboflavin-responsive oxidative phosphorylation complex I deficiency caused by defective ACAD9: new function for an old gene (Q57987777) (← links)