Pages that link to "Q39966013"
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The following pages link to Density-dependent shift of transforming growth factor-beta-1 from inhibition to stimulation of vascular smooth muscle cell growth is based on unconventional regulation of proliferation, apoptosis and contact inhibition. (Q39966013):
Displaying 10 items.
- TGF-β/Smad3 inhibit vascular smooth muscle cell apoptosis through an autocrine signaling mechanism involving VEGF-A. (Q34007727) (← links)
- Monocyte chemoattractant protein-1/CCR2 axis promotes vein graft neointimal hyperplasia through its signaling in graft-extrinsic cell populations (Q36310750) (← links)
- AMP-activated protein kinase inhibits transforming growth factor-β-mediated vascular smooth muscle cell growth: implications for a Smad-3-dependent mechanism (Q36339405) (← links)
- Hepatitis B virus X protein modulates apoptosis in human renal proximal tubular epithelial cells by activating the JAK2/STAT3 signaling pathway (Q36857763) (← links)
- Smooth muscle cell-specific Tgfbr1 deficiency attenuates neointimal hyperplasia but promotes an undesired vascular phenotype for injured arteries (Q37710674) (← links)
- α-2-Macroglobulin: a physiological guardian. (Q38053998) (← links)
- Cyclic Nucleotide-Directed Protein Kinases in Cardiovascular Inflammation and Growth (Q47713533) (← links)
- MicroRNA expression profile and functional analysis reveal their roles in contact inhibition and its disruption switch of rat vascular smooth muscle cells. (Q52561272) (← links)
- Conditional inactivation of TGF-β type II receptor in smooth muscle cells and epicardium causes lethal aortic and cardiac defects. (Q54688073) (← links)
- Anti-malarial Drugs Reduce Vascular Smooth Muscle Cell Proliferation via Activation of AMPK and Inhibition of Smad3 Signaling (Q98613644) (← links)