Pages that link to "Q39884340"
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The following pages link to Markedly reduced toxicity of a hydrogen sulphide-releasing derivative of naproxen (ATB-346). (Q39884340):
Displaying 50 items.
- Anti-cancer activity of new designer hydrogen sulfide-donating hybrids (Q26825705) (← links)
- Gaseous mediators nitric oxide and hydrogen sulfide in the mechanism of gastrointestinal integrity, protection and ulcer healing (Q28083755) (← links)
- Hydrogen Sulfide in Physiology and Diseases of the Digestive Tract (Q28087344) (← links)
- Hydrogen sulfide-releasing naproxen suppresses colon cancer cell growth and inhibits NF-κB signaling (Q28387183) (← links)
- Gastrointestinal-sparing effects of novel NSAIDs in rats with compromised mucosal defence (Q28482202) (← links)
- Profound Chemopreventative Effects of a Hydrogen Sulfide-Releasing NSAID in the APCMin/ Mouse Model of Intestinal Tumorigenesis (Q28550361) (← links)
- Synthesis of Amino-ADT Provides Access to Hydrolytically Stable Amide-Coupled Hydrogen Sulfide Releasing Drug Targets (Q28831055) (← links)
- NSAIDs do not require the presence of a carboxylic acid to exert their anti-inflammatory effect - why do we keep using it? (Q30996980) (← links)
- Hydrogen Sulfide: An Endogenous Mediator of Resolution of Inflammation and Injury (Q34226230) (← links)
- Cytoprotective effects of hydrogen sulfide in novel rat models of non-erosive esophagitis. (Q34380226) (← links)
- Hydrogen sulfide cytoprotective signaling is endothelial nitric oxide synthase-nitric oxide dependent (Q34403540) (← links)
- H(2)S-releasing aspirin protects against aspirin-induced gastric injury via reducing oxidative stress (Q34431045) (← links)
- Enhanced synthesis and diminished degradation of hydrogen sulfide in experimental colitis: a site-specific, pro-resolution mechanism (Q34938166) (← links)
- The H2S-releasing naproxen derivative, ATB-346, inhibits alveolar bone loss and inflammation in rats with ligature-induced periodontitis. (Q35158599) (← links)
- Arylthioamides as H2S Donors: l-Cysteine-Activated Releasing Properties and Vascular Effects in Vitro and in Vivo (Q35181925) (← links)
- Hydrogen sulphide protects against NSAID-enteropathy through modulation of bile and the microbiota. (Q35314338) (← links)
- Activation of the farnesoid-X receptor protects against gastrointestinal injury caused by non-steroidal anti-inflammatory drugs in mice (Q35635951) (← links)
- Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury. (Q35647095) (← links)
- Sulphide quinone reductase contributes to hydrogen sulphide metabolism in murine peripheral tissues but not in the CNS (Q36147313) (← links)
- NOSH-Aspirin: A Novel Nitric Oxide-Hydrogen Sulfide-Releasing Hybrid: A New Class of Anti-inflammatory Pharmaceuticals (Q36176425) (← links)
- Nitric oxide, afferent sensory nerves, and antioxidative enzymes in the mechanism of protection mediated by tricarbonyldichlororuthenium(II) dimer and sodium hydrosulfide against aspirin-induced gastric damage. (Q36290877) (← links)
- Panax Notoginseng Saponins Ameliorates Coxsackievirus B3-Induced Myocarditis by Activating the Cystathionine-γ-Lyase/Hydrogen Sulfide Pathway (Q36359696) (← links)
- Long-term release of a thiobenzamide from a backbone functionalized poly(lactic acid) (Q36558154) (← links)
- Potent multitarget FAAH-COX inhibitors: Design and structure-activity relationship studies (Q36576347) (← links)
- Hydrogen sulfide: a gasotransmitter of clinical relevance (Q37511893) (← links)
- Development of hydrogen sulfide-based therapeutics for cardiovascular disease (Q37772430) (← links)
- New Pharmacologic Therapies in Gastrointestinal Disease (Q37800679) (← links)
- Role of melanocortin receptors in the regulation of gouty inflammation. (Q37829674) (← links)
- NSAID gastropathy and enteropathy: distinct pathogenesis likely necessitates distinct prevention strategies (Q37882741) (← links)
- Emerging role of hydrogen sulfide in health and disease: critical appraisal of biomarkers and pharmacological tools (Q37917815) (← links)
- Hydrogen sulfide and the metabolic syndrome (Q37960613) (← links)
- Therapeutic potential of new hydrogen sulfide-releasing hybrids. (Q37960619) (← links)
- Biology and therapeutic potential of hydrogen sulfide and hydrogen sulfide-releasing chimeras (Q38056004) (← links)
- Mechanisms, prevention and clinical implications of nonsteroidal anti-inflammatory drug-enteropathy (Q38097424) (← links)
- Hydrogen sulfide signaling in the gastrointestinal tract (Q38098623) (← links)
- Lead optimization on conventional non-steroidal anti-inflammatory drugs: an approach to reduce gastrointestinal toxicity. (Q38181591) (← links)
- Hydrogen sulfide: physiological properties and therapeutic potential in ischaemia. (Q38236845) (← links)
- Recent developments in chimeric NSAIDs as safer anti-inflammatory agents. (Q38260626) (← links)
- "Gastric cytoprotection" is still relevant (Q38293038) (← links)
- Hydrogen sulfide-based therapeutics: exploiting a unique but ubiquitous gasotransmitter (Q38409371) (← links)
- Tandem mass spectrometry of nitric oxide and hydrogen sulfide releasing aspirins: a hint into activity behavior (Q38532862) (← links)
- Nonsteroidal Anti-Inflammatory Therapy: A Journey Toward Safety (Q38558587) (← links)
- Using hydrogen sulfide to design and develop drugs (Q38643114) (← links)
- Hydrogen sulphide as a signalling molecule regulating physiopathological processes in gastrointestinal motility. (Q38717958) (← links)
- A novel pH-controlled hydrogen sulfide donor protects gastric mucosa from aspirin-induced injury (Q38733697) (← links)
- Synthesis and biological evaluation of novel hydrogen sulfide releasing glycyrrhetic acid derivatives (Q38792152) (← links)
- Clinical importance of nonsteroidal anti-inflammatory drug enteropathy: the relevance of tumor necrosis factor as a promising target. (Q38809387) (← links)
- Investigational drugs targeting the prostaglandin E2 signaling pathway for the treatment of inflammatory pain (Q39007494) (← links)
- Gaseous Mediators in Gastrointestinal Mucosal Defense and Injury (Q39451528) (← links)
- Indomethacin injury to the rat small intestine is dependent upon biliary secretion and is associated with overgrowth of enterococci (Q39710654) (← links)