Pages that link to "Q39676079"
Jump to navigation
Jump to search
The following pages link to c-Jun and hypoxia-inducible factor 1 functionally cooperate in hypoxia-induced gene transcription. (Q39676079):
Displaying 45 items.
- Vaccinia-related kinase 2 modulates the stress response to hypoxia mediated by TAK1 (Q24337845) (← links)
- Hypoxia upregulates PGI-synthase and increases PGI₂ release in human vascular cells exposed to inflammatory stimuli (Q24339277) (← links)
- Molecular responses to hypoxia in tumor cells (Q24797310) (← links)
- STAT3 or USF2 contributes to HIF target gene specificity (Q31132835) (← links)
- The effects of timing of fine needle aspiration biopsies on gene expression profiles in breast cancers (Q33372939) (← links)
- Endothelial and smooth muscle cell ion channels in pulmonary vasoconstriction and vascular remodeling (Q33925149) (← links)
- SSeCKS regulates angiogenesis and tight junction formation in blood-brain barrier (Q34206194) (← links)
- Perlecan Domain V induces VEGf secretion in brain endothelial cells through integrin α5β1 and ERK-dependent signaling pathways (Q34428518) (← links)
- EBV-LMP1 targeted DNAzyme enhances radiosensitivity by inhibiting tumor angiogenesis via the JNKs/HIF-1 pathway in nasopharyngeal carcinoma (Q35742407) (← links)
- HIF-1 at the crossroads of hypoxia, inflammation, and cancer (Q36070195) (← links)
- Activating Injury-Responsive Genes with Hypoxia Enhances Axon Regeneration through Neuronal HIF-1α. (Q36304932) (← links)
- Upstream stimulatory factor 2 and hypoxia-inducible factor 2α (HIF2α) cooperatively activate HIF2 target genes during hypoxia (Q36362933) (← links)
- Pro-life role for c-Jun N-terminal kinase and p38 mitogen-activated protein kinase at rostral ventrolateral medulla in experimental brain stem death (Q36499143) (← links)
- Regulation of angiogenesis by hypoxia and hypoxia-inducible factors (Q36659549) (← links)
- Enhanceosomes as integrators of hypoxia inducible factor (HIF) and other transcription factors in the hypoxic transcriptional response. (Q36980048) (← links)
- The hypoxia-inducible factor 1/NOR-1 axis regulates the survival response of endothelial cells to hypoxia (Q37410754) (← links)
- Regulation of HIF-1α activity by overexpression of thioredoxin is independent of thioredoxin reductase status (Q37462587) (← links)
- Molecular mechanisms regulating macrophage response to hypoxia (Q38008339) (← links)
- Gene expression profiling reveals the profound upregulation of hypoxia-responsive genes in primary human astrocytes (Q38518910) (← links)
- Involvement of c-Jun N-terminal kinase in reversal of multidrug resistance of human leukemia cells in hypoxia by 5-bromotetrandrine (Q39192471) (← links)
- The response of c-jun/AP-1 to chronic hypoxia is hypoxia-inducible factor 1 alpha dependent. (Q39674326) (← links)
- Activating transcription factor 2 increases transactivation and protein stability of hypoxia-inducible factor 1alpha in hepatocytes (Q39807372) (← links)
- VEGF and Bcl-2 interact via MAPKs signaling pathway in the response to hypoxia in neuroblastoma (Q39910127) (← links)
- HIF-1alpha expression in response to lipopolysaccaride mediates induction of hepatic inflammatory cytokine TNFalpha (Q40142488) (← links)
- Critical role for NF-kappaB-induced JunB in VEGF regulation and tumor angiogenesis (Q40178664) (← links)
- Essential role of PI-3K, ERKs and calcium signal pathways in nickel-induced VEGF expression (Q40352133) (← links)
- ERKs activation and calcium signaling are both required for VEGF induction by vanadium in mouse epidermal Cl41 cells (Q40352138) (← links)
- Erythropoietin disrupts hypoxia-inducible factor signaling in ovarian cancer cells (Q40361909) (← links)
- Estrogen receptor inhibits c-Jun-dependent stress-induced cell death by binding and modifying c-Jun activity in human breast cancer cells (Q40613355) (← links)
- The von Hippel Lindau/hypoxia-inducible factor (HIF) pathway regulates the transcription of the HIF-proline hydroxylase genes in response to low oxygen (Q40631949) (← links)
- Glucose utilization is essential for hypoxia-inducible factor 1 alpha-dependent phosphorylation of c-Jun (Q40815592) (← links)
- Differential expression of transcription factors and inflammation-, ROS-, and cell death-related genes in organotypic cultures in the modiolus, the organ of Corti and the stria vascularis of newborn rats (Q42455189) (← links)
- Regulation of Cyr61/CCN1 expression by hypoxia through cooperation of c-Jun/AP-1 and HIF-1α in retinal vascular endothelial cells (Q42844511) (← links)
- Expression of proinflammatory cytokines via HIF-1alpha and NF-kappaB activation on desferrioxamine-stimulated HMC-1 cells (Q43763430) (← links)
- Toxicity of doxorubicin on pig liver after chemoembolization with doxorubicin-loaded microspheres: a pilot DNA-microarrays and histology study. (Q44131322) (← links)
- Evidence for a role of p38 kinase in hypoxia-inducible factor 1-independent induction of vascular endothelial growth factor expression by sodium arsenite (Q44249791) (← links)
- Hypoxia induces differentiation of pulmonary artery adventitial fibroblasts into myofibroblasts (Q44619265) (← links)
- Microarray analysis identifies Autotaxin, a tumour cell motility and angiogenic factor with lysophospholipase D activity, as a specific target of cell transformation by v-Jun. (Q44703503) (← links)
- Mechanism of inhibition of ascites tumor growth in mice by curcumin is mediated by NF-kB and caspase activated DNase (Q46597565) (← links)
- Functional cooperation between HIF-1α and c-Jun in mediating primary and acquired resistance to gefitinib in NSCLC cells with activating mutation of EGFR. (Q54977360) (← links)
- Quiescent Hypervascularity Mediated by Gain of HIF-1 Function (Q56551639) (← links)
- Induction of activating transcription factor 3 by anoxia is independent of p53 and the hypoxic HIF signalling pathway (Q58211826) (← links)
- Hepatocyte growth factor induces apoptosis through the extrinsic pathway in hepatoma cells: favouring role of hypoxia-inducible factor-1 deficiency (Q73563311) (← links)
- Hepatocyte growth factor signaling regulates transactivation of genes belonging to the plasminogen activation system via hypoxia inducible factor-1 (Q79185010) (← links)
- Characterization of Hypoxia Signature to Evaluate the Tumor Immune Microenvironment and Predict Prognosis in Glioma Groups (Q96155624) (← links)