Pages that link to "Q39281709"
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The following pages link to Glioblastoma resistance to anti-VEGF therapy is associated with myeloid cell infiltration, stem cell accumulation, and a mesenchymal phenotype (Q39281709):
Displaying 50 items.
- Myeloid cell signatures in tumor microenvironment predicts therapeutic response in cancer (Q26752838) (← links)
- Autophagy in stem cells (Q26823995) (← links)
- Dynamic quantitative intravital imaging of glioblastoma progression reveals a lack of correlation between tumor growth and blood vessel density (Q27308856) (← links)
- Cancer drug resistance: an evolving paradigm (Q29616694) (← links)
- Contribution of the Microenvironmental Niche to Glioblastoma Heterogeneity. (Q33786677) (← links)
- Macrophage migration inhibitory factor downregulation: a novel mechanism of resistance to anti-angiogenic therapy (Q33853991) (← links)
- Integrin inhibitor suppresses bevacizumab-induced glioma invasion (Q33912329) (← links)
- Hypoxia suppresses cylindromatosis (CYLD) expression to promote inflammation in glioblastoma: possible link to acquired resistance to anti-VEGF therapy. (Q34227712) (← links)
- An orthotopic glioblastoma mouse model maintaining brain parenchymal physical constraints and suitable for intravital two-photon microscopy. (Q34241937) (← links)
- The impact of bevacizumab treatment on survival and quality of life in newly diagnosed glioblastoma patients (Q34293163) (← links)
- RAGE expression in tumor-associated macrophages promotes angiogenesis in glioma. (Q34716082) (← links)
- Glial progenitors as targets for transformation in glioma (Q34735661) (← links)
- Bevacizumab treatment induces metabolic adaptation toward anaerobic metabolism in glioblastomas (Q34807753) (← links)
- Survival outcome of early versus delayed bevacizumab treatment in patients with recurrent glioblastoma (Q34979357) (← links)
- Upregulation of hypoxia-inducible factors and autophagy in von Hippel-Lindau-associated retinal hemangioblastoma (Q35173651) (← links)
- Neutrophils promote the malignant glioma phenotype through S100A4. (Q35577475) (← links)
- Characterization of Arginase Expression in Glioma-Associated Microglia and Macrophages (Q36218172) (← links)
- Expression and prognostic impact of matrix metalloproteinase-2 (MMP-2) in astrocytomas (Q36289595) (← links)
- Bone marrow derived myeloid cells orchestrate antiangiogenic resistance in glioblastoma through coordinated molecular networks (Q36390700) (← links)
- Tumor angiogenesis and anti-angiogenic therapy in malignant gliomas revisited (Q36428702) (← links)
- Differential expression of vascular endothelial growth factor A, its receptors VEGFR-1, -2, and -3 and co-receptors neuropilin-1 and -2 does not predict bevacizumab response in human astrocytomas (Q36493852) (← links)
- Arsenic trioxide disrupts glioma stem cells via promoting PML degradation to inhibit tumor growth (Q36545726) (← links)
- Induction of WNT11 by hypoxia and hypoxia-inducible factor-1α regulates cell proliferation, migration and invasion (Q36565126) (← links)
- Glioblastoma: Defining Tumor Niches (Q36796616) (← links)
- The role of microglia and macrophages in glioma maintenance and progression (Q36924298) (← links)
- Cytokine patterns in brain tumour progression (Q36999339) (← links)
- Periostin (POSTN) Regulates Tumor Resistance to Antiangiogenic Therapy in Glioma Models (Q37407395) (← links)
- Six-color intravital two-photon imaging of brain tumors and their dynamic microenvironment (Q37599076) (← links)
- Tissue factor expression provokes escape from tumor dormancy and leads to genomic alterations (Q37628002) (← links)
- Assessment of bevacizumab resistance increased by expression of BCAT1 in IDH1 wild-type glioblastoma: application of DSC perfusion MR imaging (Q37688411) (← links)
- Tumor-associated macrophages induce vasculogenic mimicry of glioblastoma multiforme through cyclooxygenase-2 activation (Q37708918) (← links)
- Current status of antiangiogenic therapies for glioblastomas (Q38169761) (← links)
- Resistance to antiangiogenic therapy (Q38197943) (← links)
- Bevacizumab for the treatment of high-grade glioma: an update after phase III trials. (Q38198121) (← links)
- Reciprocal Supportive Interplay between Glioblastoma and Tumor-Associated Macrophages (Q38199935) (← links)
- CNS macrophages and peripheral myeloid cells in brain tumours (Q38203815) (← links)
- Current evidence of temozolomide and bevacizumab in treatment of gliomas. (Q38230650) (← links)
- Resistance to antiangiogenic therapies (Q38232852) (← links)
- Interferon-regulatory factor-1 (IRF1) regulates bevacizumab induced autophagy (Q38260673) (← links)
- Antiangiogenic therapies for glioblastoma (Q38264310) (← links)
- Bevacizumab in high-grade gliomas: past, present, and future (Q38386873) (← links)
- Autophagy in stem and progenitor cells (Q38616599) (← links)
- Tumor-associated microglia/macrophages predict poor prognosis in high-grade gliomas and correlate with an aggressive tumor subtype. (Q38647050) (← links)
- The contribution of tumor-associated macrophages in glioma neo-angiogenesis and implications for anti-angiogenic strategies (Q38675589) (← links)
- EMT- and MET-related processes in nonepithelial tumors: importance for disease progression, prognosis, and therapeutic opportunities. (Q38677439) (← links)
- The Impact of Hypoxia and Mesenchymal Transition on Glioblastoma Pathogenesis and Cancer Stem Cells Regulation (Q38685226) (← links)
- Epithelial-mesenchymal transition in glioblastoma progression (Q38719842) (← links)
- Limited role for transforming growth factor-β pathway activation-mediated escape from VEGF inhibition in murine glioma models. (Q38765212) (← links)
- Short peptides interfering with signaling pathways as new therapeutic tools for cancer treatment (Q38767147) (← links)
- Chimeric Mouse model to track the migration of bone marrow derived cells in glioblastoma following anti-angiogenic treatments (Q38800593) (← links)